|
1
|
Moran AE, Forouzanfar MH, Roth GA, Mensah
GA, Ezzati M, Murray CJ and Naghavi M: Temporal trends in ischemic
heart disease mortality in 21 world regions, 1980 to 2010: The
Global Burden of Disease 2010 study. Circulation. 129:1483–1492.
2014. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Bainey KR and Armstrong PW: Clinical
perspectives on reperfusion injury in acute myocardial infarction.
Am Heart J. 167:637–645. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Xu Y, Wu L, Chen A, Xu C and Feng Q:
Protective effects of olive leaf extract on acrolein-exacerbated
myocardial infarction via an endoplasmic reticulum stress pathway.
Int J Mol Sci. 19(pii): E4932018. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Bulluck H, Yellon DM and Hausenloy DJ:
Reducing myocardial infarct size: Challenges and future
opportunities. Heart. 102:341–348. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Deng X, Xing X, Sun G, Xu X, Wu H, Li G
and Sun X: Guanxin danshen formulation protects against myocardial
ischemia reperfusion injury-induced left ventricular remodeling by
upregulating estrogen receptor β. Front Pharmacol. 8:7772017.
View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Li Y, Xiang Y, Zhang S, Wang Y, Yang J,
Liu W and Xue F: Intramyocardial injection of thioredoxin
2-expressing lentivirus alleviates myocardial ischemia-reperfusion
injury in rats. Am J Transl Res. 9:4428–4439. 2017.PubMed/NCBI
|
|
7
|
Liu H, Cala PM and Anderson SE: Na/H
exchange inhibition protects newborn heart from
ischemia/reperfusion injury by limiting Na+-dependent Ca2+
overload. J Cardiovasc Pharmacol. 55:227–233. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Cadenas S: ROS and redox signaling in
myocardial ischemia-reperfusion injury and cardioprotection. Free
Radic Biol Med. 117:76–89. 2018. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Griffiths EJ and Halestrap AP:
Mitochondrial non-specific pores remain closed during cardiac
ischaemia, but open upon reperfusion. Biochem J. 307:93–98. 1995.
View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Javadov S, Jang S, Parodi-Rullán R,
Khuchua Z and Kuznetsov AV: Mitochondrial permeability transition
in cardiac ischemia-reperfusion: Whether cyclophilin D is a viable
target for cardioprotection? Cell Mol Life Sci. 74:2795–2813. 2017.
View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Duan X, Ji B, Yu K, Liu J, Hei F and Long
C: Pharmacological postconditioning protects isolated rat hearts
against ischemia-reperfusion injury: The role of mitochondrial
permeability transition pore. ASAIO J. 57:197–202. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Li X, Chen Z, Wu Y, Yan Y, Sun X and Yuan
Q: Establishing an artificial pathway for efficient biosynthesis of
hydroxytyrosol. ACS Synth Biol. 7:647–654. 2018. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Poudyal H, Lemonakis N, Efentakis P, Gikas
E, Halabalaki M, Andreadou I, Skaltsounis L and Brown L:
Hydroxytyrosol ameliorates metabolic, cardiovascular and liver
changes in a rat model of diet-induced metabolic syndrome:
Pharmacological and metabolism-based investigation. Pharmacol Res.
117:32–45. 2017. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Sun Y, Zhou D and Shahidi F: Antioxidant
properties of tyrosol and hydroxytyrosol saturated fatty acid
esters. Food Chem. 245:1262–1268. 2018. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
González-Correa JA, Rodríguez-Pérez MD,
Márquez-Estrada L, López-Villodres JA, Reyes JJ,
Rodriguez-Gutierrez G, Fernández-Bolaños J and De La Cruz JP:
Neuroprotective effect of hydroxytyrosol in experimental diabetic
retinopathy: Relationship with cardiovascular biomarkers. J Agric
Food Chem. 66:637–644. 2018. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Zhi LQ, Yao SX, Liu HL, Li M, Duan N and
Ma JB: Hydroxytyrosol inhibits the inflammatory response of
osteoarthritis chondrocytes via SIRT6-mediated autophagy. Mol Med
Rep. 17:4035–4042. 2018.PubMed/NCBI
|
|
17
|
Zubair H, Bhardwaj A, Ahmad A, Srivastava
SK, Khan MA, Patel GK, Singh S and Singh AP: Hydroxytyrosol induces
apoptosis and cell cycle arrest and suppresses multiple oncogenic
signaling pathways in prostate cancer cells. Nutr Cancer.
69:932–942. 2017. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Pan S, Liu L, Pan H, Ma Y, Wang D, Kang K,
Wang J, Sun B, Sun X and Jiang H: Protective effects of
hydroxytyrosol on liver ischemia/reperfusion injury in mice. Mol
Nutr Food Res. 57:1218–1227. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Soto-Alarcon SA, Valenzuela R, Valenzuela
A and Videla LA: Liver protective effects of extra virgin olive
oil: Interaction between its chemical composition and the
cell-signaling pathways involved in protection. Endocr Metab Immune
Disord Drug Targets. 18:75–84. 2018.PubMed/NCBI
|
|
20
|
Pei YH, Chen J, Xie L, Cai XM, Yang RH,
Wang X and Gong JB: Hydroxytyrosol protects against myocardial
ischemia/reperfusion injury through a PI3K/Akt-dependent mechanism.
Mediators Inflamm. 2016:12321032016. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Kastenmayer RJ, Moore RM, Bright AL,
Torres-Cruz R and Elkins WR: Select agent and toxin regulations:
Beyond the eighth edition of the guide for the care and use of
laboratory animals. J Am Assoc Lab Anim Sci. 51:333–338.
2012.PubMed/NCBI
|
|
22
|
Wu N, Zhang X, Guan Y, Shu W, Jia P and
Jia D: Hyper-cholesterolemia abrogates the cardioprotection of
ischemic postconditioning in isolated rat heart: Roles of glycogen
synthase kinase-3β and the mitochondrial permeability transition
pore. Cell Biochem Biophys. 69:123–130. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Wu N, Li W, Shu W and Jia D: Protective
effect of picroside II on myocardial ischemia reperfusion injury in
rats. Drug Des Devel Ther. 8:545–554. 2014.PubMed/NCBI
|
|
24
|
Endlicher R, Kriváková P, Lotkova H,
Milerová M, Drahota Z and Cervinková Z: Tissue specific sensitivity
of mitochondrial permeability transition pore to Ca2+ ions. Acta
Medica (Hradec Kralove). 52:69–72. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
He F, Wu Q, Xu B, Wang X, Wu J, Huang L
and Cheng J: Suppression of Stim1 reduced intracellular calcium
concentration and attenuated hypoxia/reoxygenation induced
apoptosis in H9C2 cells. Biosci Rep. 37(pii): BSR201712492017.
View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Hurst S, Hoek J and Sheu SS: Mitochondrial
Ca2+ and regulation of the permeability transition pore.
J Bioenerg Biomembr. 49:27–47. 2017. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Petit PX, Susin SA, Zamzami N, Mignotte B
and Kroemer G: Mitochondria and programmed cell death: Back to the
future. FEBS Lett. 396:7–13. 1996. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Tong Z, Xie Y, He M, Ma W, Zhou Y, Lai S,
Meng Y and Liao Z: VDAC1 deacetylation is involved in the
protective effects of resveratrol against mitochondria-mediated
apoptosis in cardiomyocytes subjected to anoxia/reoxygenation
injury. Biomed Pharmacother. 95:77–83. 2017. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Penna C, Perrelli MG and Pagliaro P:
Mitochondrial pathways, permeability transition pore, and redox
signaling in cardioprotection: Therapeutic implications. Antioxid
Redox Signal. 18:556–599. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Siemen D and Ziemer M: What is the nature
of the mitochondrial permeability transition pore and what is it
not? IUBMB Life. 65:255–262. 2013. View
Article : Google Scholar : PubMed/NCBI
|
|
31
|
Kwong JQ and Molkentin JD: Physiological
and pathological roles of the mitochondrial permeability transition
pore in the heart. Cell Metab. 21:206–214. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Carraro M and Bernardi P: Calcium and
reactive oxygen species in regulation of the mitochondrial
permeability transition and of programmed cell death in yeast. Cell
Calcium. 60:102–107. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Bernardi P, Rasola A, Forte M and Lippe G:
The mitochondrial permeability transition pore: Channel formation
by F-ATP synthase, integration in signal transduction, and role in
pathophysiology. Physiol Rev. 95:1111–1155. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Morciano G, Bonora M, Campo G, Aquila G,
Rizzo P, Giorgi C, Wieckowski MR and Pinton P: Mechanistic role of
mPTP in ischemia-reperfusion injury. Adv Exp Med Biol. 982:169–189.
2017. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Bopassa JC, Michel P, Gateau-Roesch O,
Ovize M and Ferrera R: Low-pressure reperfusion alters
mitochondrial permeability transition. Am J Physiol Heart Circ
Physiol. 288:H2750–H2755. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Fang R, Zhang LL, Zhang LZ, Li W, Li M and
Wen K: Sphingosine 1-phosphate postconditioning protects against
myocardial ischemia/reperfusion injury in rats via mitochondrial
signaling and Akt-Gsk3β phosphorylation. Arch Med Res. 48:147–155.
2017. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Wang H, Zhao YT, Zhang S, Dubielecka PM,
Du J, Yano N, Chin YE, Zhuang S, Qin G and Zhao TC: Irisin plays a
pivotal role to protect the heart against ischemia and reperfusion
injury. J Cell Physiol. 232:3775–3785. 2017. View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Zhou H, Zhang Y, Hu S, Shi C, Zhu P, Ma Q,
Jin Q, Cao F, Tian F and Chen Y: Melatonin protects cardiac
microvasculature against ischemia/reperfusion injury via
suppression of mitochondrial fission-VDAC1-HK2-mPTP-mitophagy axis.
J Pineal Res. 63:2017. View Article : Google Scholar
|
|
39
|
Liu P and Dong J: Protective effects of
carnosic acid against mitochondria-mediated injury in H9c2
cardiomyocytes induced by hypoxia/reoxygenation. Exp Ther Med.
14:5629–5634. 2017.PubMed/NCBI
|
|
40
|
Marchi S and Pinton P: The mitochondrial
calcium uniporter complex: Molecular components, structure and
physiopathological implications. J Physiol. 592:829–839. 2014.
View Article : Google Scholar : PubMed/NCBI
|
|
41
|
Soni M, Prakash C, Dabur R and Kumar V:
Protective effect of hydroxytyrosol against oxidative stress
mediated by arsenic-induced neurotoxicity in rats. Appl Biochem
Biotechnol. 186:27–39. 2018. View Article : Google Scholar : PubMed/NCBI
|
|
42
|
Siddiqui WA, Ahad A and Ahsan H: The
mystery of BCL2 family: Bcl-2 proteins and apoptosis: An update.
Arch Toxicol. 89:289–317. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
43
|
Czabotar PE, Lessene G, Strasser A and
Adams JM: Control of apoptosis by the BCL-2 protein family:
Implications for physiology and therapy. Nat Rev Mol Cell Biol.
15:49–63. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
44
|
Narita M, Shimizu S, Ito T, Chittenden T,
Lutz RJ, Matsuda H and Tsujimoto Y: Bax interacts with the
permeability transition pore to induce permeability transition and
cytochrome c release in isolated mitochondria. Proc Natl Acad Sci
USA. 95:14681–14686. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
45
|
Ashkenazi A, Fairbrother WJ, Leverson JD
and Souers AJ: From basic apoptosis discoveries to advanced
selective BCL-2 family inhibitors. Nat Rev Drug Discov. 16:273–284.
2017. View Article : Google Scholar : PubMed/NCBI
|
|
46
|
Li H, Sun JJ, Chen GY, Wang WW, Xie ZT,
Tang GF and Wei SD: Carnosic acid nanoparticles suppress liver
ischemia/reperfusion injury by inhibition of ROS, Caspases and
NF-κB signaling pathway in mice. Biomed Pharmacother. 82:237–246.
2016. View Article : Google Scholar : PubMed/NCBI
|
|
47
|
Pastorino JG, Tafani M, Rothman RJ,
Marcinkeviciute A, Hoek JB and Farber JL: Functional consequences
of the sustained or transient activation by Bax of the
mitochondrial permeability transition pore. J Biol Chem.
274:31734–31739. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
48
|
Liao P, Sun G, Zhang C, Wang M, Sun Y,
Zhou Y, Sun X and Jian J: Bauhinia championii flavone attenuates
hypoxia-reoxygenation induced apoptosis in H9c2 cardiomyocytes by
improving mitochondrial dysfunction. Molecules. 21(pii): E14692016.
View Article : Google Scholar : PubMed/NCBI
|
