Identifying potential therapeutic targets for ischemic stroke through immune infiltration analysis and construction of a programmed cell death‑related ceRNA network
Affiliations: Department of Rehabilitation Medicine and Physiotherapy, Qingdao University of Medicine, Qingdao, Shandong 266071, P.R. China, Department of Rehabilitation Medicine, Affiliated Hospital of Qingdao University, Qingdao, Shandong 266003, P.R. China, Department of Physical Therapy Program, Washington University of Medicine, St. Louis, MO 63110, USA, Department of Rehabilitation and Health Care, Jinan Vocational College of Nursing, Jinan, Shandong 250102, P.R. China
- Published online on: September 21, 2022 https://doi.org/10.3892/etm.2022.11616
- Article Number: 680
Copyright: © Cui et al. This is an open access article distributed under the terms of Creative Commons Attribution License.
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zinc finger protein 354B and two for ferroptosis (lncRNA DLEU2L/miR‑4500/ribo-nucleotide reductase regulatory subunit M2 and lncRNA DLEU2L/miR‑4500/BACH1). Based on the aforementioned results, the present study provided potential approaches for bridging programmed cell death, immune infiltration and ceRNA regulatory networks in IS. The present study may provide novel insights into the clinical diagnosis and treatment of IS, and may improve the knowledge of the regulation of pathophysiological processes for IS.