|
1
|
Salomão R, Ferreira BL, Salomão MC, Santos
SS, Azevedo LCP and Brunialti MKC: Sepsis: Evolving concepts and
challenges. Braz J Med Biol Res. 52(e8595)2019.PubMed/NCBI View Article : Google Scholar
|
|
2
|
Candel FJ, Borges Sá M, Belda S, Bou G,
Del Pozo JL, Estrada O, Ferrer R, González Del Castillo J,
Julián-Jiménez A, Martín-Loeches I, et al: Current aspects in
sepsis approach. Turning things around. Rev Esp Quimioter.
31:298–315. 2018.PubMed/NCBI
|
|
3
|
Huang M, Cai S and Su J: The pathogenesis
of sepsis and potential therapeutic targets. Int J Mol Sci.
20(5376)2019.PubMed/NCBI View Article : Google Scholar
|
|
4
|
van der Poll T, van de Veerdonk FL,
Scicluna BP and Netea MG: The immunopathology of sepsis and
potential therapeutic targets. Nat Rev Immunol. 17:407–420.
2017.PubMed/NCBI View Article : Google Scholar
|
|
5
|
Gotts JE and Matthay MA: Sepsis:
Pathophysiology and clinical management. BMJ.
353(i1585)2016.PubMed/NCBI View Article : Google Scholar
|
|
6
|
Fattahi F and Ward PA: Complement and
sepsis-induced heart dysfunction. Mol Immunol. 84:57–64.
2017.PubMed/NCBI View Article : Google Scholar
|
|
7
|
Shi X, Liu Y, Zhang D and Xiao D: Valproic
acid attenuates sepsis-induced myocardial dysfunction in rats by
accelerating autophagy through the PTEN/AKT/mTOR pathway. Life Sci.
232(116613)2019.PubMed/NCBI View Article : Google Scholar
|
|
8
|
Li F, Lang F, Zhang H, Xu L, Wang Y, Zhai
C and Hao E: Apigenin alleviates endotoxin-induced myocardial
toxicity by modulating inflammation, oxidative stress, and
autophagy. Oxid Med Cell Longev. 2017(2302896)2017.PubMed/NCBI View Article : Google Scholar
|
|
9
|
Lado-Abeal J, Martinez-Sánchez N, Cocho
JA, Martín-Pastor M, Castro-Piedras I, Couce-Pico ML, Saha AK and
López M: Lipopolysaccharide (LPS)-induced septic shock causes
profound changes in myocardial energy metabolites in pigs.
Metabolomics. 14(131)2018.PubMed/NCBI View Article : Google Scholar
|
|
10
|
Hsu WT, Galm BP, Schrank G, Hsu TC, Lee
SH, Park JY and Lee CC: Effect of renin-angiotensin-aldosterone
system inhibitors on short-term mortality after sepsis: A
population-based cohort study. Hypertension. 75:483–491.
2020.PubMed/NCBI View Article : Google Scholar
|
|
11
|
Wang SM, Liu GQ, Xian HB, Si JL, Qi SX and
Yu YP: LncRNA NEAT1 alleviates sepsis-induced myocardial injury by
regulating the TLR2/NF-κB signaling pathway. Eur Rev Med Pharmacol
Sci. 23:4898–4907. 2019.PubMed/NCBI View Article : Google Scholar
|
|
12
|
Wu H, Liu J, Li W, Liu G and Li Z:
LncRNA-HOTAIR promotes TNF-α production in cardiomyocytes of
LPS-induced sepsis mice by activating NF-κB pathway. Biochem
Biophys Res Commun. 471:240–246. 2016.PubMed/NCBI View Article : Google Scholar
|
|
13
|
Sun LJ, Qiao W, Xiao YJ, Cui L, Wang X and
Ren WD: Naringin mitigates myocardial strain and the inflammatory
response in sepsis-induced myocardial dysfunction through
regulation of PI3K/AKT/NF-κB pathway. Int Immunopharmacol.
75(105782)2019.PubMed/NCBI View Article : Google Scholar
|
|
14
|
Han D, Li X, Li S, Su T, Fan L, Fan WS,
Qiao HY, Chen JW, Fan MM, Li XJ, et al: Reduced silent information
regulator 1 signaling exacerbates sepsis-induced myocardial injury
and mitigates the protective effect of a liver X receptor agonist.
Free Radic Biol Med. 113:291–303. 2017.PubMed/NCBI View Article : Google Scholar
|
|
15
|
Essex MN, Xu H, Parsons B, Xie L and Li C:
Parecoxib relieves pain and has an opioid-sparing effect following
major gastrointestinal surgery. Int J Gen Med. 10:319–327.
2017.PubMed/NCBI View Article : Google Scholar
|
|
16
|
Ling Y, Chen J, Tao M, Chu X and Zhang X:
A pilot study of nimotuzumab combined with cisplatin and 5-FU in
patients with advanced esophageal squamous cell carcinoma. J Thorac
Dis. 4:58–62. 2012.PubMed/NCBI View Article : Google Scholar
|
|
17
|
Tan JH, Zhou L, Kan HP and Zhang GW:
Parecoxib improves the outcomes of acute mild and moderate
pancreatitis: A 3-year matched cohort study based on a prospective
database. Pancreas. 48:1148–1154. 2019.PubMed/NCBI View Article : Google Scholar
|
|
18
|
Eberstål S, Badn W, Fritzell S,
Esbjörnsson M, Darabi A, Visse E and Siesjö P: Inhibition of
cyclooxygenase-2 enhances immunotherapy against experimental brain
tumors. Cancer Immunol Immunother. 61:1191–1199. 2012.PubMed/NCBI View Article : Google Scholar
|
|
19
|
Chen J, Cong X, Zhan X, Zhou Z and Zheng
W: Effects of parecoxib on pain threshold and inflammatory factors
IL-1β, IL-6 and TNF-α in spinal cord of rats with bone cancer pain.
J Coll Physicians Surg Pak. 29:528–531. 2019.PubMed/NCBI View Article : Google Scholar
|
|
20
|
Zhang J, Wang L, Xie W, Hu S, Zhou H, Zhu
P and Zhu H: Melatonin attenuates ER stress and mitochondrial
damage in septic cardiomyopathy: A new mechanism involving BAP31
upregulation and MAPK-ERK pathway. J Cell Physiol. 235:2847–2856.
2020.PubMed/NCBI View Article : Google Scholar
|
|
21
|
Chen Z, Cao Z, Gui F, Zhang M, Wu X, Peng
H, Yu B, Li W, Ai F and Zhang J: TMEM43 protects against
sepsis-induced cardiac injury via inhibiting ferroptosis in mice.
Cells. 11(2992)2022.PubMed/NCBI View Article : Google Scholar
|
|
22
|
Huang Y, Zhang N, Xie C, You Y, Guo L, Ye
F, Xie X and Wang J: Lipocalin-2 in neutrophils induces ferroptosis
in septic cardiac dysfunction via increasing labile iron pool of
cardiomyocytes. Front Cardiovasc Med. 9(922534)2022.PubMed/NCBI View Article : Google Scholar
|
|
23
|
Lei S, Zhang Y, Su W, Zhou L, Xu J and Xia
ZY: Remifentanil attenuates lipopolysaccharide-induced oxidative
injury by downregulating PKCβ2 activation and inhibiting autophagy
in H9C2 cardiomyocytes. Life Sci. 213:109–115. 2018.PubMed/NCBI View Article : Google Scholar
|
|
24
|
Zhang WX, He BM, Wu Y, Qiao JF and Peng
ZY: Melatonin protects against sepsis-induced cardiac dysfunction
by regulating apoptosis and autophagy via activation of SIRT1 in
mice. Life Sci. 217:8–15. 2019.PubMed/NCBI View Article : Google Scholar
|
|
25
|
Alvarez S, Vico T and Vanasco V: Cardiac
dysfunction, mitochondrial architecture, energy production, and
inflammatory pathways: Interrelated aspects in endotoxemia and
sepsis. Int J Biochem Cell Biol. 81:307–314. 2016.PubMed/NCBI View Article : Google Scholar
|
|
26
|
Chen H, Wang X, Yan X, Cheng X, He X and
Zheng W: RETRACTED: LncRNA MALAT1 regulates sepsis-induced cardiac
inflammation and dysfunction via interaction with miR-125b and p38
MAPK/NFκB. Int Immunopharmacol. 55:69–76. 2018.PubMed/NCBI View Article : Google Scholar
|
|
27
|
Wang Z, Bu L, Yang P, Feng S and Xu F:
Alleviation of sepsis-induced cardiac dysfunction by overexpression
of Sestrin2 is associated with inhibition of p-S6K and activation
of the p-AMPK pathway. Mol Med Rep. 20:2511–2518. 2019.PubMed/NCBI View Article : Google Scholar
|
|
28
|
Yücel G, Zhao Z, El-Battrawy I, Lan H,
Lang S, Li X, Buljubasic F, Zimmermann WH, Cyganek L, Utikal J, et
al: Lipopolysaccharides induced inflammatory responses and
electrophysiological dysfunctions in human-induced pluripotent stem
cell derived cardiomyocytes. Sci Rep. 7(2935)2017.PubMed/NCBI View Article : Google Scholar
|
|
29
|
Muller-Werdan U, Buerke M, Ebelt H,
Heinroth KM, Herklotz A, Loppnow H, Ruß M, Schlegel F, Schlitt A,
Schmidt HB, et al: Septic cardiomyopathy-a not yet discovered
cardiomyopathy? Exp Clin Cardiol. 11:226–236. 2006.PubMed/NCBI
|
|
30
|
Zhou N, Zeng MN, Li K, Yang YY, Bai ZY,
Zheng XK and Feng WS: An integrated metabolomic strategy for the
characterization of the effects of Chinese yam and its three active
components on septic cardiomyopathy. Food Funct. 9:4989–4997.
2018.PubMed/NCBI View Article : Google Scholar
|
|
31
|
Shang X, Lin K, Yu R, Zhu P, Zhang Y, Wang
L, Xu J and Chen K: Resveratrol protects the myocardium in sepsis
by activating the phosphatidylinositol 3-kinases
(PI3K)/AKT/mammalian target of rapamycin (mTOR) pathway and
inhibiting the nuclear factor-κB (NF-κB) signaling pathway. Med Sci
Monit. 25:9290–9298. 2019.PubMed/NCBI View Article : Google Scholar
|
|
32
|
Buerke U, Carter JM, Schlitt A, Russ M,
Schmidt H, Sibelius U, Grandel U, Grimminger F, Seeger W,
Mueller-Werdan U, et al: Apoptosis contributes to septic
cardiomyopathy and is improved by simvastatin therapy. Shock.
29:497–503. 2008.PubMed/NCBI View Article : Google Scholar
|
|
33
|
Lancel S, Joulin O, Favory R, Goossens JF,
Kluza J, Chopin C, Formstecher P, Marchetti P and Neviere R:
Ventricular myocyte caspases are directly responsible for
endotoxin-induced cardiac dysfunction. Circulation. 111:2596–2604.
2005.PubMed/NCBI View Article : Google Scholar
|
|
34
|
Zhou R, Yang X, Li X, Qu Y, Huang Q, Sun X
and Mu D: Recombinant CC16 inhibits NLRP3/caspase-1-induced
pyroptosis through p38 MAPK and ERK signaling pathways in the brain
of a neonatal rat model with sepsis. J Neuroinflammation.
16(239)2019.PubMed/NCBI View Article : Google Scholar
|
|
35
|
Zhang FL, Zhou BW, Yan ZZ, Zhao J, Zhao
BC, Liu WF, Li C and Liu KX: 6-Gingerol attenuates macrophages
pyroptosis via the inhibition of MAPK signaling pathways and
predicts a good prognosis in sepsis. Cytokine.
125(154854)2020.PubMed/NCBI View Article : Google Scholar
|
|
36
|
Koch L, Frommhold D, Buschmann K, Kuss N,
Poeschl J and Ruef P: LPS- and LTA-induced expression of IL-6 and
TNF-α in neonatal and adult blood: Role of MAPKs and NF-κB.
Mediators Inflamm. 2014(283126)2014.PubMed/NCBI View Article : Google Scholar
|
|
37
|
Yego ECK and Dillman JF III: Cytokine
regulation by MAPK activated kinase 2 in keratinocytes exposed to
sulfur mustard. Toxicol In Vitro. 27:2067–2075. 2013.PubMed/NCBI View Article : Google Scholar
|
|
38
|
Luo Y, Che W and Zhao M: Ulinastatin
post-treatment attenuates lipopolysaccharide-induced acute lung
injury in rats and human alveolar epithelial cells. Int J Mol Med.
39:297–306. 2017.PubMed/NCBI View Article : Google Scholar
|
|
39
|
Tien YC, Lin JY, Lai CH, Kuo CH, Lin WY,
Tsai CH, Tsai FJ, Cheng YC, Peng WH and Huang CY: 9Carthamus
tinctorius L. prevents LPS-induced TNFalpha signaling
activation and cell apoptosis through JNK1/2-NFkappaB pathway
inhibition in H9c2 cardiomyoblast cells. J Ethnopharmacol.
130:505–513. 2010.PubMed/NCBI View Article : Google Scholar
|
|
40
|
Li M, Ye J, Zhao G, Hong G, Hu X, Cao K,
Wu Y and Lu Z: Gas6 attenuates lipopolysaccharide-induced TNF-α
expression and apoptosis in H9C2 cells through NF-κB and MAPK
inhibition via the Axl/PI3K/Akt pathway. Int J Mol Med. 44:982–994.
2019.PubMed/NCBI View Article : Google Scholar
|
