[Corrigendum] Endogenous Nampt upregulation is associated with diabetic nephropathy inflammatory‑fibrosis through the NF‑κB p65 and Sirt1 pathway; NMN alleviates diabetic nephropathy inflammatory‑fibrosis by inhibiting endogenous Nampt

Chen,Ye; Liang,Yuzhen; Hu,Tingting; Wei,Riming; Cai,Congjie; Wang,Ping; Wang,Lingyu; Qiao,Wei; Feng,Leping

doi:10.3892/etm.2026.13238

September-2026 Volume 32 Issue 3

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Correction Open Access

[Corrigendum] Endogenous Nampt upregulation is associated with diabetic nephropathy inflammatory‑fibrosis through the NF‑κB p65 and Sirt1 pathway; NMN alleviates diabetic nephropathy inflammatory‑fibrosis by inhibiting endogenous Nampt

Authors:
- Ye Chen
- Yuzhen Liang
- Tingting Hu
- Riming Wei
- Congjie Cai
- Ping Wang
- Lingyu Wang
- Wei Qiao
- Leping Feng
View Affiliations / Copyright

Affiliations: Department of Nutrition and Health, Public Health School, Guilin Medical University, Guilin, Guangxi 541004, P.R. China, Endocrinology Department, Diabetic Metabolic Center, First Affiliated Clinical Hospital, Guangxi Medical University, Nanning, Guangxi 530021, P.R. China, Scientific Experiment Center, Biotechnology School, Guilin Medical University, Guilin, Guangxi 541004, P.R. China

Copyright: © Chen et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
Article Number: 243
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Published online on: July 8, 2026

https://doi.org/10.3892/etm.2026.13238
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Exp Ther Med 14:[Related article:] 4181–4193, 2017; DOI: 10.3892/etm.2017.5098

Following the publication of this paper, it was drawn to the Editor’s attention by a concerned reader that the Sirt1 western blot data shown in Fig. 4A on p. 4187 were strikingly similar to the Sirt1 western blot data shown in Fig. 6A on p. 4188, even though the experimental conditions reported for these figure parts were apparently different.

Figure 6

(A) Time course of endogenous Nampt, NF-κB p65, Sirt1, FoxO1, and Bax protein expression in HBZY-1 cells exposed to a high-glucose milieu for 24, 48, 72, or 96 h. Quantification of western blot analysis indicated the protein expression levels of (B) Nampt, (C) NF-κB p65, (D) Sirt1, (E) FoxO1 and (F) Bax, and β-actin were indicated. Data are presented as means ± standard deviations of three experiments. #P<0.05, ##P<0.01 vs. 24 h (control). Nampt, nicotinamide phosphoribosyltransferase; NF, nuclear factor; Sirt1, sirtuin 1; FoxO1, forkhead box protein O1; Bax, B-cell lymphoma 2-like protein 4.

Upon examining the original data in their paper, the authors have realized that the Sirt1 data correctly featured in Fig. 4A had inadvertently been re-used in Fig. 6A. The authors have repeated the western blots for Sirt1 protein in Fig. 6A, and the results obtained were broadly similar to those obtained originally. The revised version of Fig. 6, featuring the new data for Sirt1 in Fig. 6A, is shown below. All the authors agree to the publication of this Corrigendum, and are grateful to the Editor of Experimental and Therapeutic Medicine for granting them the opportunity to publish this; moreover, they apologize to the readership for any inconvenience caused.

Journals

International Journal of Molecular Medicine

International Journal of Oncology

Molecular Medicine Reports

Oncology Reports

Experimental and Therapeutic Medicine

Oncology Letters

Biomedical Reports

Molecular and Clinical Oncology

World Academy of Sciences Journal

International Journal of Functional Nutrition

International Journal of Epigenetics

Medicine International

[Corrigendum] Endogenous Nampt upregulation is associated with diabetic nephropathy inflammatory‑fibrosis through the NF‑κB p65 and Sirt1 pathway; NMN alleviates diabetic nephropathy inflammatory‑fibrosis by inhibiting endogenous Nampt

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