1
|
Moolenaar WH: Bioactive lysophospholipids
and their G protein-coupled receptors. Exp Cell Res. 253:230–238.
1999. View Article : Google Scholar : PubMed/NCBI
|
2
|
Van Meeteren LA, Frederiks F, Giepmans BN,
et al: Spider and bacterial sphingomyelinases D target cellular
lysophosphatidic acid receptors by hydrolyzing
lysophosphatidylcholine. J Biol Chem. 279:10833–10836.
2004.PubMed/NCBI
|
3
|
Tokumura A: Metabolic pathways and
physiological and pathological significances of lysolipid phosphate
mediators. J Cell Biochem. 92:869–881. 2004. View Article : Google Scholar : PubMed/NCBI
|
4
|
Sorensen SD, Nicole O, Peavy RD, et al:
Common signaling pathways link activation of murine PAR-1, LPA, and
S1P receptors to proliferation of astrocytes. Mol Pharmacol.
64:1199–1209. 2003. View Article : Google Scholar : PubMed/NCBI
|
5
|
Tigyi G and Parrill AL: Molecular
mechanisms of lysophosphatidic acid action. Prog Lipid Res.
42:498–526. 2003. View Article : Google Scholar : PubMed/NCBI
|
6
|
Inoue M, Xie W, Matsushita Y, Chun J, Aoki
J and Ueda H: Lysophosphatidylcholine induces neuropathic pain
through an action of autotaxin to generate lysophosphatidic acid.
Neuroscience. 152:296–298. 2008. View Article : Google Scholar : PubMed/NCBI
|
7
|
Mauco G, Chap H, Simon MF and Douste-Blazy
L: Phosphatidic and lysophosphatidic acid production in
phospholipase C-and thrombin-treated platelets. Possible
involvement of a platelet lipase. Biochimie. 60:653–661. 1978.
View Article : Google Scholar : PubMed/NCBI
|
8
|
Tigyi G, Hong L, Yakubu M, Parfenova H,
Shibata M and Leffler CW: Lysophosphatidic acid alters
cerebrovascular reactivity in piglets. Am J Physiol.
268:H2048–H2055. 1995.PubMed/NCBI
|
9
|
Sun GY, Lu FL, Lin SE and Ko MR:
Decapitation ischemia-induced release of free fatty acids in mouse
brain. Relationship with diacylglycerols and lysophospholipids. Mol
Chem Neuropathol. 17:39–50. 1992. View Article : Google Scholar : PubMed/NCBI
|
10
|
Chen KM, Liu JY, Lai SC, Hsu LS and Lee
HH: Association of plasminogen activators and matrix
metalloproteinase-9 proteolytic cascade with blood-CNS barrier
damage of angiostrongyliasis. Int J Exp Pathol. 87:113–119. 2006.
View Article : Google Scholar : PubMed/NCBI
|
11
|
Schulze C, Smales C, Rubin LL and Staddon
JM: Lysophosphatidic acid increases tight junction permeability in
cultured brain endothelial cells. J Neurochem. 68:991–1000. 1997.
View Article : Google Scholar : PubMed/NCBI
|
12
|
Fujii M, Duris K, Altay O, Soejima Y,
Sherchan P and Zhang JH: Inhibition of Rho kinase by hydroxyfasudil
attenuates brain edema after subarachnoid hemorrhage in rats.
Neurochem Int. 60:327–333. 2012. View Article : Google Scholar : PubMed/NCBI
|
13
|
Liu K, Li Z, Wu T and Ding S: Role of rho
kinase in microvascular damage following cerebral ischemia
reperfusion in rats. Int J Mol Sci. 12:1222–1231. 2011. View Article : Google Scholar : PubMed/NCBI
|
14
|
Ishiguro M, Kawasaki K, Suzuki Y, et al: A
Rho kinase (ROCK) inhibitor, fasudil, prevents matrix
metalloproteinase-9-related hemorrhagic transformation in mice
treated with tissue plasminogen activator. Neuroscience.
220:302–312. 2012. View Article : Google Scholar
|
15
|
Jeong KJ, Park SY, Cho KH, et al: The
Rho/ROCK pathway for lysophosphatidic acid-induced proteolytic
enzyme expression and ovarian cancer cell invasion. Oncogene.
31:4279–4289. 2012. View Article : Google Scholar : PubMed/NCBI
|
16
|
Baskaya MK, Dogan A, Rao AM and Dempsey
RJ: Neuroprotective effects of citicoline on brain edema and
blood-brain barrier breakdown after traumatic brain injury. J
Neurosurg. 92:448–452. 2000. View Article : Google Scholar : PubMed/NCBI
|
17
|
Tawara S and Shimokawa H: Progress of the
study of rho-kinase and future perspective of the inhibitor.
Yakugaku Zasshi. 127:501–514. 2007. View Article : Google Scholar : PubMed/NCBI
|
18
|
Huang XN, Fu J and Wang WZ: The effects of
fasudil on the permeability of the rat blood-brain barrier and
blood-spinal cord barrier following experimental autoimmune
encephalomyelitis. J Neuroimmunol. 239:61–67. 2011. View Article : Google Scholar : PubMed/NCBI
|
19
|
Rosenberg GA: Matrix metalloproteinases
and their multiple roles in neurodegenerative diseases. Lancet
Neurol. 8:205–216. 2009. View Article : Google Scholar : PubMed/NCBI
|
20
|
Asahi M, Asahi K, Jung JC, del Zoppo GJ,
Fini ME and Lo EH: Role for matrix metalloproteinase 9 after focal
cerebral ischemia: effects of gene knockout and enzyme inhibition
with BB-94. J Cereb Blood Flow Metab. 20:1681–1689. 2000.
View Article : Google Scholar : PubMed/NCBI
|
21
|
Asahi M, Wang X, Mori T, et al: Effects of
matrix metalloproteinase-9 gene knock-out on the proteolysis of
blood-brain barrier and white matter components after cerebral
ischemia. J Neurosci. 21:7724–7732. 2001.PubMed/NCBI
|
22
|
Barr TL, Latour LL, Lee KY, et al:
Blood-brain barrier disruption in humans is independently
associated with increased matrix metalloproteinase-9. Stroke.
41:e123–e128. 2010. View Article : Google Scholar : PubMed/NCBI
|
23
|
Rosell A, Cuadrado E, Ortega-Aznar A,
Hernandez-Guillamon M, Lo EH and Montaner J: MMP-9-positive
neutrophil infiltration is associated to blood-brain barrier
breakdown and basal lamina type IV collagen degradation during
hemorrhagic transformation after human ischemic stroke. Stroke.
39:1121–1126. 2008. View Article : Google Scholar
|
24
|
Romanic AM, White RF, Arleth AJ, Ohlstein
EH and Barone FC: Matrix metalloproteinase expression increases
after cerebral focal ischemia in rats: inhibition of matrix
metalloproteinase-9 reduces infarct size. Stroke. 29:1020–1030.
1998. View Article : Google Scholar
|
25
|
Gasche Y, Fujimura M, Morita-Fujimura Y,
Copin JC, Kawase M, Massengale J and Chan PH: Early appearance of
activated matrix metalloproteinase-9 after focal cerebral ischemia
in mice: a possible role in blood-brain barrier dysfunction. J
Cereb Blood Flow Metab. 19:1020–1028. 1999. View Article : Google Scholar : PubMed/NCBI
|
26
|
Aoki T, Sumii T, Mori T, Wang X and Lo EH:
Blood-brain barrier disruption and matrix metalloproteinase-9
expression during reperfusion injury: mechanical versus embolic
focal ischemia in spontaneously hypertensive rats. Stroke.
33:2711–2717. 2002. View Article : Google Scholar
|
27
|
Wang J and Tsirka SE: Neuroprotection by
inhibition of matrix metalloproteinases in a mouse model of
intracerebral haemorrhage. Brain. 128:1622–1633. 2005. View Article : Google Scholar : PubMed/NCBI
|
28
|
Wu H, Zhang Z, Li Y, et al: Time course of
upregulation of inflammatory mediators in the hemorrhagic brain in
rats: correlation with brain edema. Neurochem Int. 57:248–253.
2010. View Article : Google Scholar : PubMed/NCBI
|
29
|
Dietzmann K, von Bossanyi P, Krause D,
Wittig H, Mawrin C and Kirches E: Expression of the plasminogen
activator system and the inhibitors PAI-1 and PAI-2 in
posttraumatic lesions of the CNS and brain injuries following
dramatic circulatory arrests: an immunohistochemical study. Pathol
Res Pract. 196:15–21. 2000. View Article : Google Scholar
|
30
|
Ito T, Takenaka K, Sakai H, Yoshimura S,
Hayashi K, Noda S and Sakai N: Elevation of mRNA levels of
tissue-type plasminogen activator and urokinase-type plasminogen
activator in hippocampus and cerebral cortex following middle
cerebral artery occlusion in rats. Neurol Res. 22:413–419.
2000.PubMed/NCBI
|
31
|
Armstead WM, Cines DB, Bdeir K,
Kulikovskaya I, Stein SC and Higazi AA: uPA impairs
cerebrovasodilation after hypoxia/ischemia through LRP and ERK
MAPK. Brain Res. 1231:121–131. 2008. View Article : Google Scholar : PubMed/NCBI
|
32
|
Patel TH, Sprague S, Lai Q, Jimenez DF,
Barone CM and Ding Y: Blood brain barrier (BBB) dysfunction
associated with increased expression of tissue and urokinase
plasminogen activators following peripheral thermal injury.
Neurosci Lett. 444:222–226. 2008. View Article : Google Scholar
|
33
|
Park SY, Jeong KJ, Panupinthu N, et al:
Lysophosphatidic acid augments human hepatocellular carcinoma cell
invasion through LPA1 receptor and MMP-9 expression. Oncogene.
30:1351–1359. 2011. View Article : Google Scholar : PubMed/NCBI
|
34
|
Sawada K, Morishige K, Tahara M, Ikebuchi
Y, Kawagishi R, Tasaka K and Murata Y: Lysophosphatidic acid
induces focal adhesion assembly through Rho/Rho-associated kinase
pathway in human ovarian cancer cells. Gynecol Oncol. 87:252–259.
2002. View Article : Google Scholar : PubMed/NCBI
|