|
1
|
Weinert T: DNA damage and checkpoint
pathways: molecular anatomy and interactions with repair. Cell.
94:555–558. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Megyesi J, Andrade L, Vieira JM Jr,
Safirstein RL and Price PM: Coordination of the cell cycle is an
important determinant of the syndrome of acute renal failure. Am J
Physiol Renal Physiol. 283:F810–F816. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Zhou H, Kato A, Yasuda H, et al: The
induction of cell cycle regulatory and DNA repair proteins in
cisplatin-induced acute renal failure. Toxicol Appl Pharmacol.
200:111–120. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Price PM, Safirstein RL and Megyesi J:
Protection of renal cells from cisplatin toxicity by cell cycle
inhibitors. Am J Physiol Renal Physiol. 286:F378–F384. 2004.
View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Megyesi J, Safirstein RL and Price PM:
Induction of p21WAF1/CIP1/SDI1 in kidney tubule cells affects the
course of cisplatin-induced acute renal failure. J Clin Invest.
101:777–782. 1998. View
Article : Google Scholar : PubMed/NCBI
|
|
6
|
Zhou H, Fujigaki Y, Kato A, et al:
Inhibition of p21 modifies the response of cortical proximal
tubules to cisplatin in rats. Am J Physiol Renal Physiol.
291:F225–F235. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Nowak G, Price PM and Schnellmann RG: Lack
of a functional p21WAF1/CIP1 gene accelerates caspase-independent
apoptosis induced by cisplatin in renal cells. Am J Physiol Renal
Physiol. 285:F440–F450. 2003.PubMed/NCBI
|
|
8
|
Yu F, Megyesi J, Safirstein RL and Price
PM: Identification of the functional domain of p21(WAF1/CIP1) that
protects cells from cisplatin cytotoxicity. Am J Physiol Renal
Physiol. 289:F514–F520. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Miyaji T, Kato A, Yasuda H, Fujigaki Y and
Hishida A: Role of the increase in p21 in cisplatin-induced acute
renal failure in rats. J Am Soc Nephrol. 12:900–908.
2001.PubMed/NCBI
|
|
10
|
Nath KA: Provenance of the protective
property of p21. Am J Physiol Renal Physiol. 289:F512–F513. 2005.
View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Hengst L and Reed SI: Inhibitors of the
Cip/Kip family. Curr Top Microbiol Immunol. 227:25–41. 1998.
|
|
12
|
Sherr CJ and Roberts JM: Inhibitors of
mammalian G1 cyclin-dependent kinases. Genes Dev. 9:1149–1163.
1995. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Al-Mohanna MA, Manogaran PS, Al-Mukhalafi
ZK, Al-Hussein AK and Aboussekhra A: The tumor suppressor
p16(INK4a) gene is a regulator of apoptosis induced by ultraviolet
light and cisplatin. Oncogene. 23:201–212. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Le HV, Minn AJ and Massagué J:
Cyclin-dependent kinase inhibitors uncouple cell cycle progression
from mitochondrial apoptotic functions in DNA-damaged cancer cells.
J Biol Chem. 280:32018–32025. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Scassa ME, Marazita MC, Ceruti JM, et al:
Cell cycle inhibitor, p19INK4d, promotes cell survival and
decreases chromosomal aberrations after genotoxic insult due to
enhanced DNA repair. DNA Repair (Amst). 6:626–638. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Tavera-Mendoza LE, Wang TT and White JH:
p19INK4D and cell death. Cell Cycle. 5:596–598. 2006. View Article : Google Scholar
|
|
17
|
Yuan Y, Shen H, Franklin DS, Scadden DT
and Cheng T: In vivo self-renewing divisions of haematopoietic stem
cells are increased in the absence of the early G1-phase inhibitor,
p18INK4C. Nat Cell Biol. 6:436–442. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Franklin DS, Godfrey VL, Lee H, et al: CDK
inhibitors p18(INK4c) and p27(Kip1) mediate two separate pathways
to collaboratively suppress pituitary tumorigenesis. Genes Dev.
12:2899–2911. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Wang D and Lippard SJ: Cellular processing
of platinum anticancer drugs. Nat Rev Drug Discov. 4:307–320. 2005.
View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Siddik ZH: Cisplatin: mode of cytotoxic
action and molecular basis of resistance. Oncogene. 22:7265–7279.
2003. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Pabla N and Dong Z: Cisplatin
nephrotoxicity: mechanisms and renoprotective strategies. Kidney
Int. 73:994–1007. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Heyman SN, Lieberthal W, Rogiers R and
Bonventre JV: Animal models of acute tubular necrosis. Curr Opin
Crit Care. 8:526–534. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Lieberthal W, Triaca V and Levine J:
Mechanisms of death induced by cisplatin in proximal tubular
epithelial cells: apoptosis vs. necrosis Am J Physiol.
270:F700–F708. 1996.PubMed/NCBI
|
|
24
|
Razzaque MS, Koji T, Kumatori A and
Taguchi T: Cisplatin-induced apoptosis in human proximal tubular
epithelial cells is associated with the activation of the Fas/Fas
ligand system. Histochem Cell Biol. 111:359–365. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Seth R, Yang C, Kaushal V, Shah SV and
Kaushal GP: p53-dependent caspase-2 activiation in mitochondrial
release of apoptosis-inducing factor and its role in renal tubular
epithelial cell injury. J Biol Chem. 280:31230–31239. 2005.
View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Park MS, De Leon M and Devarajan P:
Cisplatin induces apoptosis in LLC-PK1 cells via activation of
mitochondrial pathways. J Am Soc Nephrol. 13:858–865.
2002.PubMed/NCBI
|
|
27
|
Muruganandan S and Cribb AE:
Calpain-induced endoplasmic reticulum stress and cell death
following cytotoxic damage to renal cells. Toxicol Sci. 94:118–128.
2006. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Cribb AE, Peyrou M, Muruganandan S and
Schneider L: The endoplasmic reticulum in xenobiotic toxicity. Drug
Metab Rev. 37:405–442. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Peyrou M, Hanna PE and Cribb AE:
Cisplatin, gentamicin, and p-aminophenol induce markers of
endoplasmic reticulum stress in the rat kidneys. Toxicol Sci.
99:346–353. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Liu H and Baliga R: Endoplasmic reticulum
stress-associated caspase 12 mediates cisplatin-induced LLC-PK1
cell apoptosis. J Am Soc Nephrol. 16:1985–1992. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Bernales S, Papa FR and Walter P:
Intracellular signaling by the unfolded protein response. Annu Rev
Cell Dev Biol. 22:487–508. 2006. View Article : Google Scholar
|
|
32
|
Malhotra JD and Kaufman RJ: The
endoplasmic reticulum and the unfolded protein response. Semin Cell
Dev Biol. 18:716–731. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Mori K: Tripartite management of unfolded
proteins in the endoplasmic reticulum. Cell. 101:451–454. 2000.
View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Kleizen B and Braakman L: Protein folding
and quality control in the endoplasmic reticulum. Curr Opin Cell
Biol. 16:343–349. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Szegezdi E, Logue SE, Gorman AM and Samali
A: Mediators of endoplasmic reticulum stress-induced apoptosis.
EMBO Rep. 7:880–885. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Price PM, Yu F, Kaldis P, et al:
Dependence of cisplatin-induced cell death in vitro and in vivo on
cyclin-dependent kinase 2. J Am Soc Nephrol. 17:2434–2442. 2006.
View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Yu F, Megyesi J and Price PM: Cytoplasmic
initiation of cisplatin cytotoxicity. Am J Physiol Renal Physiol.
295:F44–F52. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Hirai H, Roussel MF, Kato JY, Ashmun RA
and Sherr CJ: Novel INK4 proteins, p19 and p18, are specific
inhibitors of the cyclin D-dependent kinases CDK4 and CDK6. Mol
Cell Biol. 15:2672–2681. 1995.PubMed/NCBI
|
|
39
|
Cordon-Cardo C: Mutation of cell cycle
regulators. Biological and clinical implaications for human
neoplasia. Am J Pathol. 147:545–560. 1995.PubMed/NCBI
|
|
40
|
Shankland SJ and Wolf G: Cell cycle
regulatory proteins in renal disease: role in hypertrophy,
proliferation, and apoptosis. Am J Physiol Renal Physiol.
278:F515–F529. 2000.PubMed/NCBI
|
