1
|
Carmeliet P: Angiogenesis in life, disease
and medicine. Nature. 438:932–936. 2005. View Article : Google Scholar : PubMed/NCBI
|
2
|
Augustin HG: Tubes, branches, and pillars:
The many ways of forming a new vasculature. Circ Res. 89:645–647.
2001. View Article : Google Scholar : PubMed/NCBI
|
3
|
Larson DM and Haudenschild CC: Junctional
transfer in wounded cultures of bovine aortic endothelial cells.
Lab Invest. 59:373–379. 1988.PubMed/NCBI
|
4
|
Homkajorn B, Sims NR and Muyderman H:
Connexin 43 regulates astrocytic migration and proliferation in
response to injury. Neurosci Lett. 486:197–201. 2010. View Article : Google Scholar : PubMed/NCBI
|
5
|
Walker DL, Vacha SJ, Kirby ML and Lo CW:
Connexin43 deficiency causes dysregulation of coronary
vasculogenesis. Dev Biol. 284:479–498. 2005. View Article : Google Scholar : PubMed/NCBI
|
6
|
Vinken M, Decrock E, De Vuyst E, Ponsaerts
R, D'hondt C, Bultynck G, Ceelen L, Vanhaecke T, Leybaert L and
Rogiers V: Connexins: Sensors and regulators of cell cycling.
Biochim Biophys Acta. 1815:13–25. 2011.
|
7
|
Stains JP and Civitelli R: Gap junctions
regulate extracellular signal-regulated kinase signaling to affect
gene transcription. Mol Biol Cell. 16:64–72. 2005. View Article : Google Scholar :
|
8
|
Gomes P, Srinivas SP, Vereecke J and
Himpens B: ATP-dependent paracrine intercellular communication in
cultured bovine corneal endothelial cells. Invest Ophthalmol Vis
Sci. 46:104–113. 2005. View Article : Google Scholar
|
9
|
Faigle M, Seessle J, Zug S, El Kasmi KC
and Eltzschig HK: ATP release from vascular endothelia occurs
across Cx43 hemichannels and is attenuated during hypoxia. PloS
One. 3:e28012008. View Article : Google Scholar : PubMed/NCBI
|
10
|
Brisset AC, Isakson BE and Kwak BR:
Connexins in vascular physiology and pathology. Antioxid Redox
Signal. 11:267–282. 2009. View Article : Google Scholar
|
11
|
Goodenough DA, Goliger JA and Paul DL:
Connexins, connexons, and intercellular communication. Annu Rev
Biochem. 65:475–502. 1996. View Article : Google Scholar : PubMed/NCBI
|
12
|
Söhl G and Willecke K: Gap junctions and
the connexin protein family. Cardiovasc Res. 62:228–232. 2004.
View Article : Google Scholar : PubMed/NCBI
|
13
|
Hervé JC and Derangeon M:
Gap-junction-mediated cell-to-cell communication. Cell Tissue Res.
352:21–31. 2013. View Article : Google Scholar
|
14
|
Bruzzone R, White TW and Paul DL:
Connections with connexins: The molecular basis of direct
intercellular signaling. Eur J Biochem. 238:1–27. 1996. View Article : Google Scholar : PubMed/NCBI
|
15
|
Maeda S and Tsukihara T: Structure of the
gap junction channel and its implications for its biological
functions. Cell Mol Life Sci. 68:1115–1129. 2011. View Article : Google Scholar
|
16
|
Haefliger JA, Nicod P and Meda P:
Contribution of connexins to the function of the vascular wall.
Cardiovasc Res. 62:345–356. 2004. View Article : Google Scholar : PubMed/NCBI
|
17
|
Van Rijen H, van Kempen MJ, Analbers LJ,
Rook MB, van Ginneken AC, Gros D and Jongsma HJ: Gap junctions in
human umbilical cord endothelial cells contain multiple connexins.
Am J Physiol. 272:C117–C130. 1997. View Article : Google Scholar : PubMed/NCBI
|
18
|
Wang HH, Kung CI, Tseng YY, Lin YC, Chen
CH, Tsai CH and Yeh HI: Activation of endothelial cells to
pathological status by down-regulation of connexin43. Cardiovasc
Res. 79:509–518. 2008. View Article : Google Scholar : PubMed/NCBI
|
19
|
Dhein S, Gaertner C, Georgieff C, Salameh
A, Schlegel F and Mohr FW: Effects of isoprenaline on endothelial
connexins and angiogenesis in a human endothelial cell culture
system. Naunyn Schmiedebergs Arch Pharmacol. 388:101–108. 2015.
View Article : Google Scholar
|
20
|
Wang HH, Su CH, Wu YJ, Li JY, Tseng YM,
Lin YC, Hsieh CL, Tsai CH and Yeh HI: Reduction of connexin43 in
human endothelial progenitor cells impairs the angiogenic
potential. Angiogenesis. 16:553–560. 2013. View Article : Google Scholar : PubMed/NCBI
|
21
|
Xu X, Francis R, Wei CJ, Linask KL and Lo
CW: Connexin 43-mediated modulation of polarized cell movement and
the directional migration of cardiac neural crest cells.
Development. 133:3629–3639. 2006. View Article : Google Scholar : PubMed/NCBI
|
22
|
Kardami E, Stoski RM, Doble BW, Yamamoto
T, Hertzberg EL and Nagy JI: Biochemical and ultrastructural
evidence for the association of basic fibroblast growth factor with
cardiac gap junctions. J Biol Chem. 266:19551–19557.
1991.PubMed/NCBI
|
23
|
Gartner C, Ziegelhöffer B, Kostelka M,
Stepan H, Mohr FW and Dhein S: Knock-down of endothelial connexins
impairs angio-genesis. Pharmacol Res. 65:347–357. 2012. View Article : Google Scholar
|
24
|
Polontchouk L, Ebelt B, Jackels M and
Dhein S: Chronic effects of endothelin 1 and angiotensin II on gap
junctions and intercellular communication in cardiac cells. FASEB
J. 16:87–89. 2002. View Article : Google Scholar
|
25
|
Schulz R, Görge PM, Gorbe A, Ferdinandy P,
Lampe PD and Leybaert L: Connexin 43 is an emerging therapeutic
target in ischemia/reperfusion injury, cardioprotection and
neuropro-tection. Pharmacol Ther. 153:90–106. 2015. View Article : Google Scholar : PubMed/NCBI
|
26
|
Bian B, Yu X, Wang Q, Teng T and Nie J:
Atorvastatin protects myocardium against ischemia-reperfusion
arrhythmia by increasing Connexin 43 expression: A rat model. Eur J
Pharmacol. 768:13–20. 2015. View Article : Google Scholar : PubMed/NCBI
|
27
|
Strale PO, Clarhaut J, Lamiche C, Cronier
L, Mesnil M and Defamie N: Down-regulation of Connexin43 expression
reveals the involvement of caveolin-1 containing lipid rafts in
human U251 glioblastoma cell invasion. Mol Carcinog. 51:845–860.
2012. View
Article : Google Scholar
|
28
|
Wang WK, Chen MC, Leong HF, Kuo YL, Kuo CY
and Lee CH: Connexin 43 suppresses tumor angiogenesis by
down-regulation of vascular endothelial growth factor via
hypoxic-induced factor-1α. Int J Mol Sci. 16:439–451. 2014.
View Article : Google Scholar : PubMed/NCBI
|
29
|
Kwak BR, Mulhaupt F, Veillard N, Gros DB
and Mach F: Altered pattern of vascular connexin expression in
atherosclerotic plaques. Arterioscler Thromb Vasc Biol. 22:225–230.
2002. View Article : Google Scholar : PubMed/NCBI
|
30
|
Chadjichristos CE, Derouette JP and Kwak
BR: Connexins in atherosclerosis. Adv Cardiol. 42:255–267. 2006.
View Article : Google Scholar : PubMed/NCBI
|
31
|
Li H, He J, Yu H, Green CR and Chang J:
Bioglass promotes wound healing by affecting gap junction connexin
43 mediated endothelial cell behavior. Biomaterials. 84:64–75.
2016. View Article : Google Scholar : PubMed/NCBI
|
32
|
Tarzemany R, Jiang G, Jiang JX,
Gallant-Behm C, Wiebe C, Hart DA, Larjava H and Häkkinen L:
Connexin 43 regulates the expression of wound healing-related genes
in human gingival and skin fibroblasts. Exp Cell Res. 367:150–161.
2018. View Article : Google Scholar : PubMed/NCBI
|
33
|
Fang K, Fu W, Beardsley AR, Sun X, Lisanti
MP and Liu J: Overexpression of caveolin-1 inhibits endothelial
cell proliferation by arresting the cell cycle at
G0/G1 phase. Cell Cycle. 6:199–204. 2007.
View Article : Google Scholar : PubMed/NCBI
|
34
|
Lin MI, Yu J, Murata T and Sessa WC:
Caveolin-1-deficient mice have increased tumor microvascular
permeability, angiogenesis, and growth. Cancer Res. 67:2849–2856.
2007. View Article : Google Scholar : PubMed/NCBI
|
35
|
Sotkis A, Wang XG, Yasumura T, Peracchia
LL, Persechini A, Rash JE and Peracchia C: Calmodulin colocalizes
with connexins and plays a direct role in gap junction channel
gating. Cell Commun Adhes. 8:277–281. 2001. View Article : Google Scholar
|
36
|
Giepmans BN, Verlaan I, Hengeveld T,
Janssen H, Calafat J, Falk MM and Moolenaar WH: Gap junction
protein connexin-43 interacts directly with microtubules. Curr
Biol. 11:1364–1368. 2001. View Article : Google Scholar : PubMed/NCBI
|
37
|
Schubert AL, Schubert W, Spray DC and
Lisanti MP: Connexin family members target to lipid raft domains
and interact with caveolin-1. Biochemistry. 41:5754–5764. 2002.
View Article : Google Scholar : PubMed/NCBI
|
38
|
Langlois S, Cowan KN, Shao Q, Cowan BJ and
Laird DW: Caveolin-1 and -2 interact with connexin43 and regulate
gap junctional intercellular communication in keratinocytes. Mol
Biol Cell. 19:912–928. 2008. View Article : Google Scholar :
|
39
|
Razani B, Schlegel A, Liu J and Lisanti
MP: Caveolin-1, a putative tumour suppressor gene. Biochem Soc
Trans. 29:494–499. 2001. View Article : Google Scholar : PubMed/NCBI
|
40
|
Galbiati F, Volonté D, Liu J, Capozza F,
Frank PG, Zhu L, Pestell RG and Lisanti MP: Caveolin-1 expression
negatively regulates cell cycle progression by inducing G(0)/G(1)
arrest via a p53/p21(WAF1/Cip1)-dependent mechanism. Mol Biol Cell.
12:2229–2244. 2001. View Article : Google Scholar : PubMed/NCBI
|
41
|
Hartel FV, Holl M, Arshad M, Aslam M,
Gündüz D, Weyand M, Micoogullari M, Abdallah Y, Piper HM and Noll
T: Transient hypoxia induces ERK-dependent anti-apoptotic cell
survival in endothelial cells. Am J Physiol Cell Physiol.
298:C1501–C1509. 2010. View Article : Google Scholar : PubMed/NCBI
|
42
|
World Medical Association Declaration of
Helsinki: Recommendations guiding physicians in biomedical research
involving human subjects. Cardiovasc Res. 35:2–3. 1997.
|
43
|
Zhao Y, Yu L, Xu S, Qiu F, Fan Y and Fu G:
Down-regulation of connexin43 gap junction by serum deprivation in
human endo-thelial cells was improved by (-)-Epigallocatechin
gallate via ERK MAP kinase pathway. Biochem Biophys Res Commun.
404:217–222. 2011. View Article : Google Scholar
|
44
|
Li B, Jia S, Yue T, Yang L, Huang C,
Verkhratsky A and Peng L: Biphasic regulation of Caveolin-1 gene
expression by fluoxetine in astrocytes: Opposite effects of
PI3K/AKT and MAPK/ERK signaling pathways on c-fos. Front Cell
Neurosci. 11:3352017. View Article : Google Scholar : PubMed/NCBI
|
45
|
Braet K, Vandamme W, Martin PE, Evans WH
and Leybaert L: Photoliberating inositol-1,4,5-trisphosphate
triggers ATP release that is blocked by the connexin mimetic
peptide gap 26. Cell Calcium. 33:37–48. 2003. View Article : Google Scholar : PubMed/NCBI
|
46
|
Leybaert L, Braet K, Vandamme W, Cabooter
L, Martin PE and Evans WH: Connexin channels, connexin mimetic
peptides and ATP release. Cell Commun Adhes. 10:251–257. 2003.
View Article : Google Scholar : PubMed/NCBI
|
47
|
Nandigama R, Padmasekar M, Wartenberg M
and Sauer H: Feed forward cycle of hypotonic stress-induced ATP
release, puri-nergic receptor activation, and growth stimulation of
prostate cancer cells. J Biol Chem. 281:5686–5693. 2006. View Article : Google Scholar
|
48
|
Galbiati F, Volonte D, Engelman JA,
Watanabe G, Burk R, Pestell RG and Lisanti MP: Targeted
downregulation of caveolin-1 is sufficient to drive cell
transformation and hyperac-tivate the p42/44 MAP kinase cascade.
EMBO J. 17:6633–6648. 1998. View Article : Google Scholar : PubMed/NCBI
|
49
|
Park H, Go YM, St John PL, Maland MC,
Lisanti MP, Abrahamson DR and Jo H: Plasma membrane cholesterol is
a key molecule in shear stress-dependent activation of
extracellular signal-regulated kinase. J Biol Chem.
273:32304–32311. 1998. View Article : Google Scholar : PubMed/NCBI
|
50
|
Engelman JA, Chu C, Lin A, Jo H, Ikezu T,
Okamoto T, Kohtz DS and Lisanti MP: Caveolin-mediated regulation of
signaling along the p42/44 MAP kinase cascade in vivo. A role for
the caveolin-scaffolding domain. FEBS Lett. 428:205–211. 1998.
View Article : Google Scholar : PubMed/NCBI
|
51
|
Chambard JC, Lefloch R, Pouysségur J and
Lenormand P: ERK implication in cell cycle regulation. Biochim
Biophys Acta. 1773:1299–1310. 2007. View Article : Google Scholar
|