1
|
Tektonidou MG: Antiphospholipid syndrome
nephropathy: From pathogenesis to treatment. Front Immunol.
9:11812018. View Article : Google Scholar : PubMed/NCBI
|
2
|
Giannakopoulos B, Passam F, Rahgozar S and
Krilis SA: Current concepts on the pathogenesis of the
antiphospholipid syndrome. Blood. 109:422–430. 2007. View Article : Google Scholar
|
3
|
Pierangeli SS, Chen PP, Raschi E, Scurati
S, Grossi C, Borghi MO, Palomo I, Harris EN and Meroni PL:
Antiphospholipid antibodies and the antiphospholipid syndrome:
Pathogenic mechanisms. Semin Thromb Hemost. 34:236–250. 2008.
View Article : Google Scholar : PubMed/NCBI
|
4
|
McDonnell T, Wincup C, Buchholz I,
Pericleous C, Giles I, Ripoll V, Cohen H, Delcea M and Rahman A:
The role of beta-2-glycoprotein I in health and disease associating
structure with function: More than just APS. Blood Rev.
39:1006102020. View Article : Google Scholar :
|
5
|
Martínez-Flores JA, Serrano M, Pérez D,
Cámara A G, Lora D, Morillas L, Ayala R, Paz-Artal E, Morales JM
and Serrano A: Circulating immune complexes of IgA bound to beta 2
glycoprotein are strongly associated with the occurrence of acute
thrombotic events. J Atheroscler Thromb. 23:1242–1253. 2016.
View Article : Google Scholar : PubMed/NCBI
|
6
|
Zhang W, Gao F, Lu D, Sun N, Yin X, Jin M
and Liu Y: Anti-β2 glycoprotein I antibodies in complex with β2
glycoprotein I induce platelet activation via two receptors:
Apolipoprotein E receptor 2' and glycoprotein I bα. Front Med.
10:76–84. 2016. View Article : Google Scholar
|
7
|
Zha C, Zhang W, Gao F, Xu J, Jia R, Cai J
and Liu Y: Anti-β2GPI/β2GPI induces
neutrophil extracellular traps formation to promote thrombogenesis
via the TLR4/MyD88/MAPKs axis activation. Neuropharmacology.
138:140–150. 2018. View Article : Google Scholar : PubMed/NCBI
|
8
|
Segel GB, Halterman MW and Lichtman MA:
The paradox of the neutrophil's role in tissue injury. J Leukoc
Biol. 89:359–372. 2011. View Article : Google Scholar
|
9
|
Zychlinsky A, Prevost MC and Sansonetti
PJ: Shigella flexneri induces apoptosis in infected macrophages.
Nature. 358:167–169. 1992. View Article : Google Scholar : PubMed/NCBI
|
10
|
Cookson BT and Brennan MA:
Pro-inflammatory programmed cell death. Trends Microbiol.
9:113–114. 2011. View Article : Google Scholar
|
11
|
Miao EA, Rajan JV and Aderem A:
Caspase-1-induced pyroptotic cell death. Immunol Rev. 243:206–214.
2011. View Article : Google Scholar : PubMed/NCBI
|
12
|
Liu X, Zhang Z, Ruan J, Pan Y, Magupalli
VG, Wu H and Lieberman J: Inflammasome-activated gasdermin D causes
pyroptosis by forming membrane pores. Nature. 535:153–158. 2016.
View Article : Google Scholar : PubMed/NCBI
|
13
|
Mezzaroma E, Toldo S, Farkas D, Seropian
IM, Van Tassell BW, Salloum FN, Kannan HR, Menna AC, Voelkel NF and
Abbate A: The inflammasome promotes adverse cardiac remodeling
following acute myocardial infarction in the mouse. Proc Natl Acad
Sci USA. 108:19725–19730. 2011. View Article : Google Scholar : PubMed/NCBI
|
14
|
Inoue Y, Shirasuna K, Kimura H, Usui F,
Kawashima A, Karasawa T, Tago K, Dezaki K, Nishimura S, Sagara J,
et al: NLRP3 regulates neutrophil functions and contributes to
hepatic ischemia-reperfusion injury independently of inflammasomes.
J Immunol. 192:4342–4351. 2014. View Article : Google Scholar
|
15
|
Lu B, Nakamura T, Inouye K, Li J, Tang Y,
Lundbäck P, Valdes-Ferrer SI, Olofsson PS, Kalb T, Roth J, et al:
Novel role of PKR in inflammasome activation and HMGB1 release.
Nature. 488:670–674. 2012. View Article : Google Scholar : PubMed/NCBI
|
16
|
Yim HC, Wang D, Yu L, White CL, Faber PW,
Williams BR and Sadler AJ: The kinase activity of PKR represses
inflammasome activity. Cell Res. 26:367–379. 2016. View Article : Google Scholar : PubMed/NCBI
|
17
|
Goh KC, deVeer MJ and Williams BR: The
protein kinase PKR is required for p38 MAPK activation and the
innate immune response to bacterial endotoxin. EMBO J.
19:4292–4297. 2000. View Article : Google Scholar : PubMed/NCBI
|
18
|
Ma CH, Wu CH, Jou IM, Tu YK, Hung CH, Chou
WC, Chang YC, Hsieh PL and Tsai KL: PKR promotes oxidative stress
and apoptosis of human articular chondrocytes by causing
mitochondrial dysfunction through p38 MAPK activation-PKR
activation causes apoptosis in human chondrocytes. Antioxidants
(Basel). 8:3702019. View Article : Google Scholar
|
19
|
Kang R, Chen R, Zhang Q, Hou W, Wu S, Cao
L, Huang J, Yu Y, Fan XG, Yan Z, et al: HMGB1 in health and
disease. Mol Aspects Med. 40:1–116. 2014. View Article : Google Scholar : PubMed/NCBI
|
20
|
Xie WH, Ding J, Xie XX, Yang XH, Wu XF,
Chen ZX, Guo QL, Gao WY, Wang XZ and Li D: Hepatitis B virus X
protein promotes liver cell pyroptosis under oxidative stress
through NLRP3 inflammasome activation. Inflamm Res. 69:683–696.
2020. View Article : Google Scholar : PubMed/NCBI
|
21
|
Lee PH, Yamamoto TN, Gurusamy D, Sukumar
M, Yu Z, Hu-Li J, Kawabe T, Gangaplara A, Kishton RJ, Henning AN,
et al: Host conditioning with IL-1β improves the antitumor function
of adoptively transferred T cells. J Exp Med. 216:2619–2634. 2019.
View Article : Google Scholar :
|
22
|
Huebener P, Pradere JP, Hernandez C, Gwak
GY, Caviglia JM, Mu X, Loike JD and Schwabe RF: The HMGB1/RAGE axis
triggers neutrophil-mediated injury amplification following
necrosis. J Clin Invest. 125:539–550. 2015. View Article : Google Scholar : PubMed/NCBI
|
23
|
Wu X, Zhang H, Qi W, Zhang Y, Li J, Li Z,
Lin Y, Bai X, Liu X, Chen X, et al: Nicotine promotes
atherosclerosis via ROS-NLRP3-mediated endothelial cell pyroptosis.
Cell Death Dis. 9:1712018. View Article : Google Scholar : PubMed/NCBI
|
24
|
Takahashi M: NLRP3 inflammasome as a novel
player in myocardial infarction. Int Heart J. 55:101–105. 2014.
View Article : Google Scholar : PubMed/NCBI
|
25
|
Kovacs SB and Miao EA: Gasdermins:
Effectors of pyroptosis. Trends Cell Biol. 27:673–684. 2017.
View Article : Google Scholar : PubMed/NCBI
|
26
|
Agar C, van Os GM, Mörgelin M, Sprenger
RR, Marquart JA, Urbanus RT, Derksen RH, Meijers JC and de Groot
PG: Beta2-glycoprotein I can exist in 2 conformations: Implications
for our understanding of the antiphospholipid syndrome. Blood.
116:1336–1343. 2010. View Article : Google Scholar : PubMed/NCBI
|
27
|
Zhou H, Sheng L, Wang H, Xie H, Mu Y, Wang
T and Yan J: Anti-β2GPI/β2GPI stimulates activation of THP-1 cells
through TLR4/MD-2/MyD88 and NF-κB signaling pathways. Thromb Res.
132:742–749. 2013. View Article : Google Scholar
|
28
|
Cho JS, Guo Y, Ramos RI, Hebroni F,
Plaisier SB, Xuan C, Granick JL, Matsushima H, Takashima A, Iwakura
Y, et al: Neutrophil-derived IL-1β is sufficient for abscess
formation in immunity against staphylococcus aureus in mice. PLoS
Pathog. 8:e10030472012. View Article : Google Scholar
|
29
|
Mulla MJ, Salmon JE, Chamley LW, Brosens
JJ, Boeras CM, Kavathas PB and Abrahams VM: A role for uric acid
and the Nalp3 inflammasome in antiphospholipid antibody-induced
IL-1β production by human first trimester trophoblast. PLoS One.
8:e652372013. View Article : Google Scholar
|
30
|
Guo Z, Yu S, Chen X, Ye R, Zhu W and Liu
X: NLRP3 is involved in ischemia/reperfusion injury. CNS Neurol
Disord Drug Targets. 15:699–712. 2016. View Article : Google Scholar : PubMed/NCBI
|
31
|
Wang W and Zhang T: Caspase-1-mediated
pyroptosis of the predominance for driving CD4[Formula: See text] T
cells death: A nonlocal spatial mathematical model. Bull Math Biol.
80:540–582. 2018. View Article : Google Scholar : PubMed/NCBI
|
32
|
Karmakar M, Katsnelson M, Malak HA, Greene
NG, Howell SJ, Hise AG, Camilli A, Kadioglu A, Dubyak GR and
Pearlman E: Neutrophil IL-1β processing induced by pneumolysin is
mediated by the NLRP3/ASC inflammasome and caspase-1 activation and
is dependent on K+ efflux. J Immunol. 194:1763–1775. 2015.
View Article : Google Scholar : PubMed/NCBI
|
33
|
Shi J, Zhao Y, Wang K, Shi X, Wang Y,
Huang H, Zhuang Y, Cai T, Wang F and Shao F: Cleavage of GSDMD by
inflammatory caspases determines pyroptotic cell death. Nature.
526:660–665. 2015. View Article : Google Scholar : PubMed/NCBI
|
34
|
Ismael S, Zhao L, Nasoohi S and Ishrat T:
Inhibition of the NLRP3-inflammasome as a potential approach for
neuroprotection after stroke. Sci Rep. 8:59712018. View Article : Google Scholar : PubMed/NCBI
|
35
|
An H, Qian C and Cao X: Regulation of
Toll-like receptor signaling in the innate immunity. Sci China Life
Sci. 53:34–43. 2010. View Article : Google Scholar : PubMed/NCBI
|
36
|
Xing Y, Cao R and Hu HM: TLR and NLRP3
inflammasome-dependent innate immune responses to tumor-derived
autophagosomes (DRibbles). Cell Death Dis. 7:e23222016. View Article : Google Scholar : PubMed/NCBI
|
37
|
Wang Y, Zhu X, Yuan S, Wen S, Liu X, Wang
C, Qu Z, Li J, Liu H, Sun L and Liu F: TLR4/NF-κB signaling induces
GSDMD-Related pyroptosis in tubular cells in diabetic kidney
disease. Front Endocrinol (Lausanne). 10:6032019. View Article : Google Scholar
|
38
|
Bat-Erdene U, Quan E, Chan K, Lee BM,
Matook W, Lee KY and Rosales JL: Neutrophil TLR4 and PKR are
targets of breast cancer cell glycosaminoglycans and effectors of
glycosaminoglycan-induced APRIL secretion. Oncogenesis. 7:452018.
View Article : Google Scholar : PubMed/NCBI
|
39
|
Bonnet MC, Weil R, Dam E, Hovanessian AG
and Meurs EF: PKR stimulates NF-kappaB irrespective of its kinase
function by interacting with the IkappaB kinase complex. Mol Cell
Biol. 20:4532–4542. 2000. View Article : Google Scholar : PubMed/NCBI
|
40
|
Zhang P and Samuel CE: Induction of
protein kinase PKR-dependent activation of interferon regulatory
factor 3 by vaccinia virus occurs through adapter IPS-1 signaling.
J Biol Chem. 283:34580–34587. 2008. View Article : Google Scholar : PubMed/NCBI
|
41
|
Barlan AU, Griffin TM, McGuire KA and
Wiethoff CM: Adenovirus membrane penetration activates the NLRP3
inflammasome. J Virol. 85:146–155. 2011. View Article : Google Scholar :
|
42
|
Adamiak M, Ciechanowicz A, Skoda M, Cymer
M, Tracz M, Xu B and Ratajczak MZ: Novel Evidence that purinergic
signaling-Nlrp3 inflammasome axis regulates circadian rhythm of
hematopoietic stem/progenitor cells circulation in peripheral
blood. Stem Cell Rev Rep. 16:335–343. 2020. View Article : Google Scholar : PubMed/NCBI
|
43
|
Dyer MR, Chen Q, Haldeman S, Yazdani H,
Hoffman R, Loughran P, Tsung A, Zuckerbraun BS, Simmons RL and Neal
MD: Deep vein thrombosis in mice is regulated by platelet HMGB1
through release of neutrophil-extracellular traps and DNA. Sci Rep.
8:20682018. View Article : Google Scholar : PubMed/NCBI
|
44
|
Tulotta C and Ottewell P: The role of
IL-1B in breast cancer bone metastasis. Endocr Relat Cancer.
25:R421–R434. 2018. View Article : Google Scholar : PubMed/NCBI
|
45
|
Schulze J, Zierath D, Tanzi P, Cain K,
Shibata D, Dressel A and Becker K: Severe stroke induces
long-lasting alterations of high-mobility group box 1. Stroke.
44:246–248. 2013. View Article : Google Scholar
|
46
|
Yoshida H, Russell J, Senchenkova EY,
Almeida Paula LD and Granger DN: Interleukin-1beta mediates the
extra-intestinal thrombosis associated with experimental colitis.
Am J Pathol. 177:2774–2781. 2010. View Article : Google Scholar : PubMed/NCBI
|
47
|
Khodabandehlou K, Masehi-Lano JJ, Poon C,
Wang J and Chung EJ: Targeting cell adhesion molecules with
nanoparticles using in vivo and flow-based in vitro models of
atherosclerosis. Exp Biol Med (Maywood). 242:799–812. 2017.
View Article : Google Scholar
|
48
|
Hosseinkhani B, Kuypers S, van den Akker
NMS, Molin DGM and Michiels L: Extracellular vesicles work as a
functional inflammatory mediator between vascular endothelial cells
and immune cells. Front Immunol. 9:17892018. View Article : Google Scholar : PubMed/NCBI
|
49
|
Raschi E, Chighizola CB, Grossi C, Ronda
N, Gatti R, Meroni PL and Borghi MO: β2-glycoprotein I,
lipopolysaccharide and endothelial TLR4: Three players in the two
hit theory for anti-phospholipid-mediated thrombosis. J Autoimmun.
55:42–50. 2014. View Article : Google Scholar : PubMed/NCBI
|
50
|
Xia L, Zhou H, Wang T, Xie Y, Wang T, Wang
X and Yan J: Activation of mTOR is involved in
anti-β2GPI/β2GPI-induced expression of tissue
factor and IL-8 in monocytes. Thromb Res. 157:103–110. 2017.
View Article : Google Scholar : PubMed/NCBI
|