|
1
|
Blaser MJ and Berg DE: Helicobacter pylori
genetic diversity and risk of human disease. J Clin Invest.
107:767–773. 2001. View
Article : Google Scholar : PubMed/NCBI
|
|
2
|
Safavi M, Sabourian R and Foroumadi A:
Treatment of Helicobacter pylori infection: Current and future
insights. World J Clin Cases. 4:5–19. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Robinson K, Argent RH and Atherton JC: The
inflammatory and immune response to Helicobacter pyloriinfection.
Best Pract Res Clin Gastroenterol. 21:237–259. 2007. View Article : Google Scholar
|
|
4
|
Conteduca V, Sansonno D, Lauletta G, Russi
S, Ingravallo G and Dammacco F: H. pylori infection and gastric
cancer: State of the art (review). Int J Oncol. 42:5–18. 2013.
View Article : Google Scholar
|
|
5
|
International Agency for Research on
Cancer: IARC Monographs on the Evaluation of Carcinogenic Risk to
Humans, Vol 61: Schistosomotes, Liver Flukes and Helicobacter
pylori. IARC; Lyon: 1994
|
|
6
|
Watanabe T, Tada M, Nagai H, Sasaki S and
Nakao M: Helicobacter pylori infection induces gastric cancer in
mongolian gerbils. Gastroenterology. 115:642–648. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Honda S, Fujioka T, Tokieda M, Satoh R,
Nishizono A and Nasu M: Development of Helicobacter pylori-induced
gastric carcinoma in Mongolian gerbils. Cancer Res. 58:4255–4259.
1998.PubMed/NCBI
|
|
8
|
Franco AT, Israel DA, Washington MK,
Krishna U, Fox JG, Rogers AB, Neish AS, Collier-Hyams L,
Perez-Perez GI, Hatakeyama M, et al: Activation of beta-catenin by
carcinogenic Helicobacter pylori. Proc Natl Acad Sci USA.
102:10646–10651. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Montano V, Didelot X, Foll M, Linz B,
Reinhardt R, Suerbaum S, Moodley Y and Jensen JD: Worldwide
population structure, long-term demography, and local adaptation of
Helicobacter pylori. Genetics. 200:947–963. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Miszczyk E, Rudnicka K, Moran AP, Fol M,
Kowalewicz-Kulbat M, Druszczyńska M, Matusiak A, Walencka M,
Rudnicka W and Chmiela M: Interaction of Helicobacter pylori with
C-type lectin dendritic cell-specific ICAM grabbing nonintegrin. J
Biomed Biotechnol. 2012:2064632012. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Pacifico L, Osborn JF, Tromba V,
Romaggioli S, Bascetta S and Chiesa C: Helicobacter pylori
infection and extragastric disorders in children: A critical
update. World J Gastroenterol. 20:1379–1401. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Chmiela M, Gajewski A and Rudnicka K:
Helicobacter pylori vs coronary heart disease - searching for
connections. World J Cardiol. 7:187–203. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Buzás GM: Metabolic consequences of
Helicobacter pylori infection and eradication. World J
Gastroenterol. 20:5226–5234. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Hu Y, He C, Liu JP, Li NS, Peng C, Yang-Ou
YB, Yang XY, Lu NH and Zhu Y: Analysis of key genes and signaling
pathways involved in Helicobacter pylori-associated gastric cancer
based on The Cancer Genome Atlas database and RNA sequencing data.
Helicobacter. 23:e125302018. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Chen S, Duan G, Zhang R and Fan Q:
Helicobacter pylori cytotoxin-associated gene A protein upregulates
α-enolase expression via Src/MEK/ERK pathway: Implication for
progression of gastric cancer. Int J Oncol. 45:764–770. 2014.
View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Khatoon J, Prasad KN, Prakash Rai R,
Ghoshal UC and Krishnani N: Association of heterogenicity of
Helicobacter pylori cag pathogenicity island with peptic ulcer
diseases and gastric cancer. Br J Biomed Sci. 74:121–126. 2017.
View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Livak KJ and Schmittgen TD: Analysis of
relative gene expression data using real-time quantitative PCR and
the 2(-Δ Δ C(T)) Method. Methods. 25:402–408. 2001. View Article : Google Scholar
|
|
18
|
Lin SL, Yeh JL, Tsai PC, Chang TH, Huang
WC, Lee ST, Wassler M, Geng YJ and Sulistyowati E: Inhibition of
neointima hyperplasia, inflammation, and reactive oxygen species in
balloon-injured arteries by HVJ envelope vector-mediated delivery
of superoxide dismutase gene. Transl Stroke Res. Sep 6–2018, (Epub
ahead of print) http://doi.org/10.1007/s12975-018-0660-9urisimpledoi.org/10.1007/s12975-018-0660-9.
View Article : Google Scholar
|
|
19
|
Jia Q, Feng M, Wang Y and Xue S: Gastric
cancer cells in collagen gel matrix: Three-dimensional growth and
differential expression of adhesion molecules (CD44s, CD54,
E-cadherin). J Biomed Mater Res A. 84:917–925. 2008. View Article : Google Scholar
|
|
20
|
Tafreshi M, Guan J, Gorrell RJ, Chew N,
Xin Y, Deswaerte V, Rohde M, Daly RJ, Peek RM Jr, Jenkins BJ, et
al: Helicobacter pylori type IV secretion system and its adhesin
subunit, CagL, mediate potent inflammatory responses in primary
human endothelial cells. Front Cell Infect Microbiol. 8:222018.
View Article : Google Scholar :
|
|
21
|
Sierra JC, Asim M, Verriere TG, Piazuelo
MB, Suarez G, Romero-Gallo J, Delgado AG, Wroblewski LE, Barry DP,
Peek RM Jr, et al: Epidermal growth factor receptor inhibition
downregulates Helicobacter pylori-induced epithelial inflammatory
responses, DNA damage and gastric carcinogenesis. Gut.
67:1247–1260. 2018. View Article : Google Scholar
|
|
22
|
Müller A and Solnick JV: Inflammation,
immunity, and vaccine development for Helicobacter pylori.
Helicobacter. 16(Suppl 1): 26–32. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Nguyen TT, Kim SJ, Park JM, Hahm KB and
Lee HJ: Repressed TGF-β signaling through CagA-Smad3 interaction as
pathogenic mechanisms of Helicobacter pylori-associated gastritis.
J Clin Biochem Nutr. 57:113–120. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Blaser MJ and Atherton JC: Helicobacter
pylori persistence: Biology and disease. J Clin Invest.
113:321–333. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Wang J, Wu J, Cheng Y, Jiang Y and Li G:
Over-expression of microRNA-223 inhibited the proinflammatory
responses in Helicobacter pylori-infection macrophages by
down-regulating IRAK-1. Am J Transl Res. 8:615–622. 2016.PubMed/NCBI
|
|
26
|
Panpetch W, Spinler JK, Versalovic J and
Tumwasorn S: Characterization of Lactobacillus salivarius strains
B37 and B60 capable of inhibiting IL-8 production in Helicobacter
pylori-stimulated gastric epithelial cells. BMC Microbiol.
16:2422016. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Chongruksut W, Limpakan Yamada S,
Chakrabandhu B, Ruengorn C and Nanta S: Correlation of Helicobacter
pylori and interleukin-8 mRNA expression in high risk gastric
cancer population prediction. World J Gastrointest Oncol.
8:215–221. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Koussoulas V, Vassiliou S,
Giamarellos-Bourboulis EJ, Tassias G, Kotsaki A, Barbatzas C and
Tzivras M: Implications for a role of interleukin-23 in the
pathogenesis of chronic gastritis and of peptic ulcer disease. Clin
Exp Immunol. 156:97–101. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Ohnishi N, Yuasa H, Tanaka S, Sawa H,
Miura M, Matsui A, Higashi H, Musashi M, Iwabuchi K, Suzuki M, et
al: Transgenic expression of Helicobacter pylori CagA induces
gastrointestinal and hematopoietic neoplasms in mouse. Proc Natl
Acad Sci USA. 105:1003–1008. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Park HS, Wijerathne CUB, Jeong HY, Seo CS,
Ha H and Kwun HJ: Gastroprotective effects of Hwanglyeonhaedok-tang
against Helicobacter pylori-induced gastric cell injury. J
Ethnopharmacol. 216:239–250. 2018. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Akeel M, Shehata A, Elhafey A, Elmakki E,
Aboshouk T, Ageely H and Mahfouz M: Helicobacter pylorivacA, cagA
and iceA genotypes in dyspeptic patients from southwestern region,
Saudi Arabia: distribution and association with clinical outcomes
and histopathological changes. BMC Gastroenterol. 19:162019.
View Article : Google Scholar
|
|
32
|
Lee KS, Kalantzis A, Jackson CB, O’Connor
L, Murata-Kamiya N, Hatakeyama M, Judd LM, Giraud AS and Menheniott
TR: Helicobacter pylori CagA Triggers expression of the
bactericidal lectin REG3c via gastric STAT3 activation. PLoS One.
7:e307862012. View Article : Google Scholar
|
|
33
|
Palframan SL, Kwok T and Gabriel K:
Vacuolating cytotoxin A (VacA), a key toxin for Helicobacter pylori
pathogenesis. Front Cell Infect Microbiol. 2:922012. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Raju D, Hussey S, Ang M, Terebiznik MR,
Sibony M, Galindo-Mata E, Gupta V, Blanke SR, Delgado A,
Romero-Gallo J, et al: Vacuolating cytotoxin and variants in
Atg16L1 that disrupt autophagy promote Helicobacter pylori
infection in humans. Gastroenterology. 142:1160–1171. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Rassow J and Meinecke M: Helicobacter
pylori VacA: A new perspective on an invasive chloride channel.
Microbes Infect. 14:1026–1033. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Hisatsune J, Yamasaki E, Nakayama M,
Shirasaka D, Kurazono H, Katagata Y, Inoue H, Han J, Sap J, Yahiro
K, et al: Helicobacter pylori VacA enhances prostaglandin E2
production through induction of cyclooxygenase 2 expression via a
p38 mitogen-activated protein kinase/activating transcription
factor 2 cascade in AZ-521 cells. Infect Immun. 75:4472–4481. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Amieva MR and El-Omar EM: Host-bacterial
interactions in Helicobacter pylori infection. Gastroenterology.
134:306–323. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Wang P, Mei J, Zhang N, Tao J, Tian H and
Fu GH: Helicobacter pylori upregulates the expression of p16(INK4)
in gastric cancer cells. Hepatogastroenterology. 58:846–853.
2011.PubMed/NCBI
|
|
39
|
Zhang GX, Gu YH, Zhao ZQ, Xu SF, Zhang HJ,
Wang HD and Hao B: Coordinate increase of telomerase activity and
c-Myc expression in Helicobacter pylori-associated gastric
diseases. World J Gastroenterol. 10:1759–1762. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
40
|
Saf C, Gulcan EM, Ozkan F, Cobanoglu Saf
SP and Vitrinel A: Assessment of p21, p53 expression, and Ki-67
proliferative activities in the gastric mucosa of children with
Helicobacter pylori gastritis. Eur J Gastroenterol Hepatol.
27:155–161. 2015. View Article : Google Scholar
|
|
41
|
Petersson F, Franzén LE and Borch K:
Characterization of the gastric cardia in volunteers from the
general population Type of mucosa, Helicobacter pylori infection,
inflammation, mucosal proliferative activity, p53 and p21
expression, and relations to gastritis. Dig Dis Sci. 55:46–53.
2010. View Article : Google Scholar
|
|
42
|
Wei J, Nagy TA, Vilgelm A, Zaika E, Ogden
SR, Romero-Gallo J, Piazuelo MB, Correa P, Washington MK, El-Rifai
W, et al: Regulation of p53 tumor suppressor by Helicobacter pylori
in gastric epithelial cells. Gastroenterology. 139:1333–1343. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
43
|
Xu H, Chaturvedi R, Cheng Y, Bussiere FI,
Asim M, Yao MD, Potosky D, Meltzer SJ, Rhee JG, Kim SS, et al:
Spermine oxidation induced by Helicobacter pylori results in
apoptosis and DNA damage: Implications for gastric carcinogenesis.
Cancer Res. 64:8521–8525. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
44
|
Gamboa-Dominguez A, Ubbelohde T,
Saqui-Salces M, Romano-Mazzoti L, Cervantes M, Domínguez-Fonseca C,
de la Luz Estreber M and Ruíz-Palacios GM: Salt and stress
synergize H. pylori-induced gastric lesions, cell proliferation,
and p21 expression in Mongolian gerbils. Dig Dis Sci. 52:1517–1526.
2007. View Article : Google Scholar : PubMed/NCBI
|
|
45
|
Kurosawa A, Miwa H, Hirose M, Tsune I,
Nagahara A and Sato N: Inhibition of cell proliferation and
induction of apoptosis by Helicobacter pylori through increased
phosphorylated p53, p21 and Bax expression in endothelial cells. J
Med Microbiol. 51:385–391. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
46
|
Suzuki M, Mimuro H, Kiga K, Fukumatsu M,
Ishijima N, Morikawa H, Nagai S, Koyasu S, Gilman RH, Kersulyte D,
et al: Helicobacter pylori CagA phosphorylation-independent
function in epithelial proliferation and inflammation. Cell Host
Microbe. 5:23–34. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
47
|
Murata-Kamiya N, Kurashima Y, Teishikata
Y, Yamahashi Y, Saito Y, Higashi H, Aburatani H, Akiyama T, Peek RM
Jr, Azuma T, et al: Helicobacter pylori CagA interacts with
E-cadherin and deregulates the beta-catenin signal that promotes
intestinal transdifferentiation in gastric epithelial cells.
Oncogene. 26:4617–4626. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
48
|
Gagnaire A, Nadel B, Raoult D, Neefjes J
and Gorvel JP: Collateral damage: Insights into bacterial
mechanisms that predispose host cells to cancer. Nat Rev Microbiol.
15:109–128. 2017. View Article : Google Scholar : PubMed/NCBI
|
|
49
|
Chauhan N, Tay ACY, Marshall BJ and Jain
U: Helicobacter pylori VacA, a distinct toxin exerts diverse
functionalities in numerous cells: An overview. Helicobacter.
24:e125442019. View Article : Google Scholar
|
|
50
|
Kumar S and Dhiman M: Inflammasome
activation and regulation during Helicobacter pylori pathogenesis.
Microb Pathog. 125:468–474. 2018. View Article : Google Scholar : PubMed/NCBI
|
|
51
|
Lundgren A, Trollmo C, Edebo A,
Svennerholm AM and Lundin BS: Helicobacter pylori-specific
CD4+ T cells home to and accumulate in the human
Helicobacter pylori-infected gastric mucosa. Infect Immun.
73:5612–5619. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
52
|
Garay J, Piazuelo MB, Majumdar S, Li L,
Trillo-Tinoco J, Del Valle L, Schneider BG, Delgado AG, Wilson KT,
Correa P, et al: The homing receptor CD44 is involved in the
progression of precancerous gastric lesions in patients infected
with Helicobacter pylori and in development of mucous metaplasia in
mice. Cancer Lett. 371:90–98. 2016. View Article : Google Scholar :
|
|
53
|
Maeda S, Akanuma M, Mitsuno Y, Hirata Y,
Ogura K, Yoshida H, Shiratori Y and Omata M: Distinct mechanism of
Helicobacter pylori-mediated NF-kappa B activation between gastric
cancer cells and monocytic cells. J Biol Chem. 276:44856–44864.
2001. View Article : Google Scholar : PubMed/NCBI
|
