|
1
|
Halliwell B and Gutteridge JMC: Free
Radicals in Biology and Medicine. 4th edition. Oxford University
Press; Oxford, United Kingdom: 2007
|
|
2
|
Fridovich I: Oxygen toxicity: a radical
explanation. J Exp Biol. 201:1203–1209. 1998.PubMed/NCBI
|
|
3
|
Papa S and Skulachev VP: Reactive oxygen
species, mitochondria, apoptosis and aging. Mol Cell Biochem.
174:305–319. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Rhee SG, Kang SW, Chang TS, Jeong W and
Kim K: Peroxiredoxin, a novel family of peroxidases. IUBMB Life.
52:35–41. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Rhee SG, Yang KS, Kang SW, Woo HA and
Chang TS: Controlled elimination of intracellular
H2O2: regulation of peroxiredoxin, catalase,
and glutathione peroxidase via post-translational modification.
Antioxid Redox Signal. 7:619–626. 2005.PubMed/NCBI
|
|
6
|
Rhee SG, Chae HZ and Kim K:
Peroxiredoxins: a historical overview and speculative preview of
novel mechanisms and emerging concepts in cell signaling. Free
Radic Biol Med. 38:1543–5152. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Fujii J and Ikeda Y: Advances in our
understanding of peroxiredoxin, a multifunctional, mammalian redox
protein. Redox Rep. 7:123–130. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Chang XZ, Li DQ, Hou YF, Wu J, Lu JS, Di
GH, Jin W, Ou ZL, Shen ZZ and Shao ZM: Identification of the
functional role of peroxiredoxin 6 in the progression of breast
cancer. Breast Cancer Res. 9:R762007. View
Article : Google Scholar : PubMed/NCBI
|
|
9
|
Iraqui I, Faye G, Ragu S, Masurel-Heneman
A, Kolodner RD and Huang ME: Human peroxiredoxin PrxI is an
orthologue of yeast Tsa1, capable of suppressing genome instability
in Saccharomyces cerevisiae. Cancer Res. 68:1055–1063. 2008.
View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Song IS, Kim SU, Oh NS, Kim J, Yu DY,
Huang SM, Kim JM, Lee DS and Kim NS: Peroxiredoxin I contributes to
TRAIL resistance through suppression of redox-sensitive caspase
activation in human hepatoma cells. Carcinogenesis. 30:1106–1114.
2009. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Neumann CA, Krause DS, Carman CV, Das S,
Dubey DP, Abraham JL, Bronson RT, Fujiwara Y, Orkin SH and Van
Etten RA: Essential role for the peroxiredoxin Prdx1 in erythrocyte
antioxidant defence and tumour suppression. Nature. 424:561–565.
2003. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Fang J, Nakamura T, Cho DH, Gu Z and
Lipton SA: S-nitrosylation of peroxiredoxin 2 promotes oxidative
stress-induced neuronal cell death in Parkinson’s disease. Proc
Natl Acad Sci USA. 104:18742–18747. 2007.PubMed/NCBI
|
|
13
|
Cha MK, Suh KH and Kim IH: Overexpression
of peroxiredoxin I and thioredoxin1 in human breast carcinoma. J
Exp Clin Cancer Res. 28:932009. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Karihtala P, Mantyniemi A, Kang SW,
Kinnula VL and Soini Y: Peroxiredoxins in breast carcinoma. Clin
Cancer Res. 9:3418–3424. 2003.PubMed/NCBI
|
|
15
|
Quan C, Cha EJ, Lee HL, Han KH, Lee KM and
Kim WJ: Enhanced expression of peroxiredoxin I and VI correlates
with development, recurrence and progression of human bladder
cancer. J Urol. 175:1512–1516. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Lehtonen ST, Svensk AM, Soini Y, Paakko P,
Hirvikoski P, Kang SW, Saily M and Kinnula VL: Peroxiredoxins, a
novel protein family in lung cancer. Int J Cancer. 111:514–521.
2004. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Yanagawa T, Iwasa S, Ishii T, Tabuchi K,
Yusa H, Onizawa K, Omura K, Harada H, Suzuki H and Yoshida H:
Peroxiredoxin I expression in oral cancer: a potential new tumor
marker. Cancer Lett. 156:27–35. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Chang JW, Jeon HB, Lee JH, Yoo JS, Chun
JS, Kim JH and Yoo YJ: Augmented expression of peroxiredoxin I in
lung cancer. Biochem Biophys Res Commun. 289:507–512. 2001.
View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Rhee SG, Kang SW, Jeong W, Chang TS, Yang
KS and Woo HA: Intracellular messenger function of hydrogen
peroxide and its regulation by peroxiredoxins. Curr Opin Cell Biol.
17:183–189. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Rhee SG: Cell signaling.
H2O2, a necessary evil for cell signaling.
Science. 312:1882–1883. 2006.PubMed/NCBI
|
|
21
|
Okado-Matsumoto A, Matsumoto A, Fujii J
and Taniguchi N: Peroxiredoxin IV is a secretable protein with
heparin-binding properties under reduced conditions. J Biochem.
127:493–501. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Tavender TJ, Sheppard AM and Bulleid NJ:
Peroxiredoxin IV is an endoplasmic reticulum-localized enzyme
forming oligomeric complexes in human cells. Biochem J.
411:191–199. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Schulte J, Struck J, Bergmann A and Kohrle
J: Immunoluminometric assay for quantification of peroxiredoxin 4
in human serum. Clin Chim Acta. 411:1258–1263. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Tavender TJ and Bulleid NJ: Peroxiredoxin
IV protects cells from oxidative stress by removing
H2O2 produced during disulphide formation. J
Cell Sci. 123:2672–2679. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Bertolotti M, Yim SH, Garcia-Manteiga JM,
Masciarelli S, Kim YJ, Kang MH, Iuchi Y, Fujii J, Vene R,
Rubartelli A, Rhee SG and Sitia R: B- to plasma-cell terminal
differentiation entails oxidative stress and profound reshaping of
the antioxidant responses. Antioxid Redox Signal. 13:1133–1144.
2010. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Ikeda Y, Ito R, Ihara H, Okada T and Fujii
J: Expression of N-terminally truncated forms of rat
peroxiredoxin-4 in insect cells. Protein Expr Purif. 72:1–7. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Matsumoto A, Okado A, Fujii T, Fujii J,
Egashira M, Niikawa N and Taniguchi N: Cloning of the peroxiredoxin
gene family in rats and characterization of the fourth member. FEBS
Lett. 443:246–250. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Yoshida MC, Masuda R, Sasaki M, Takeichi
N, Kobayashi H, Dempo K and Mori M: New mutation causing hereditary
hepatitis in the laboratory rat. J Hered. 78:361–365.
1987.PubMed/NCBI
|
|
29
|
Li Y, Togashi Y, Sato S, Emoto T, Kang JH,
Takeichi N, Kobayashi H, Kojima Y, Une Y and Uchino J: Spontaneous
hepatic copper accumulation in Long-Evans Cinnamon rats with
hereditary hepatitis. A model of Wilson’s disease. J Clin Invest.
87:1858–1861. 1991.PubMed/NCBI
|
|
30
|
Wu J, Forbes JR, Chen HS and Cox DW: The
LEC rat has a deletion in the copper transporting ATPase gene
homologous to the Wilson disease gene. Nat Genet. 7:541–545. 1994.
View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Masuda R, Yoshida MC, Sasaki M, Dempo K
and Mori M: High susceptibility to hepatocellular carcinoma
development in LEC rats with hereditary hepatitis. Jpn J Cancer
Res. 79:828–835. 1988. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Marquez-Quinones A, Cipak A, Zarkovic K,
Fattel-Fazenda S, Villa-Trevino S, Waeg G, Zarkovic N and Gueraud
F: HNE-protein adducts formation in different pre-carcinogenic
stages of hepatitis in LEC rats. Free Radic Res. 44:119–127. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Samuele A, Mangiagalli A, Armentero MT,
Fancellu R, Bazzini E, Vairetti M, Ferrigno A, Richelmi P, Nappi G
and Blandini F: Oxidative stress and pro-apoptotic conditions in a
rodent model of Wilson’s disease. Biochim Biophys Acta.
1741:325–330. 2005.PubMed/NCBI
|
|
34
|
Madhani M, Barchowsky A, Klei L, Ross CR,
Jackson SK, Swartz HM and James PE: Antibacterial peptide PR-39
affects local nitric oxide and preserves tissue oxygenation in the
liver during septic shock. Biochim Biophys Acta. 1588:232–240.
2002. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Sato H, Shiiya A, Kimata M, Maebara K,
Tamba M, Sakakura Y, Makino N, Sugiyama F, Yagami K, Moriguchi T,
Takahashi S and Bannai S: Redox imbalance in cystine/glutamate
transporter-deficient mice. J Biol Chem. 280:37423–37429. 2005.
View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Khan N, Williams BB, Hou H, Li H and
Swartz HM: Repetitive tissue pO2 measurements by
electron paramagnetic resonance oximetry: current status and future
potential for experimental and clinical studies. Antioxid Redox
Signal. 9:1169–1182. 2007.
|
|
37
|
Nagaoka Y, Iuchi Y, Ikeda Y and Fujii J:
Glutathione reductase is expressed at high levels in pancreatic
islet cells. Redox Rep. 9:321–324. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Fujii T, Fujii J and Taniguchi N:
Augmented expression of peroxiredoxin VI in rat lung and kidney
after birth implies an antioxidative role. Eur J Biochem.
268:218–225. 2001. View Article : Google Scholar : PubMed/NCBI
|
