|
1
|
Randazzo PA, Inoue H and Bharti S: Arf
GAPs as regulators of the actin cytoskeleton. Biol Cell.
99:583–600. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Spang A, Shiba Y and Randazzo PA: Arf
GAPs: gatekeepers of vesicle generation. FEBS Lett. 584:2646–2651.
2010. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Randazzo PA and Hirsch DS: Arf GAPs:
multifunctional proteins that regulate membrane traffic and actin
remodelling. Cell Signal. 16:401–413. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
D’Souza-Schorey C and Chavrier P: ARF
proteins: roles in membrane traffic and beyond. Nat Rev Mol Cell
Biol. 7:347–358. 2006.
|
|
5
|
Bharti S, Inoue H, Bharti K, Hirsch DS,
Nie Z, Yoon HY, Artym V, Yamada KM, Mueller SC, Barr VA and
Randazzo PA: Src-dependent phosphorylation of ASAP1 regulates
podosomes. Mol Cell Biol. 27:8271–8283. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Randazzo PA, Andrade J, Miura K, Brown MT,
Long YQ, Stauffer S, Roller P and Cooper JA: The Arf
GTPase-activating protein ASAP1 regulates the actin cytoskeleton.
Proc Natl Acad Sci USA. 97:4011–4016. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
de Curtis I: Cell migration: GAPs between
membrane traffic and the cytoskeleton. EMBO Rep. 2:277–281.
2001.PubMed/NCBI
|
|
8
|
Ismail SA, Vetter IR, Sot B and
Wittinghofer A: The structure of an Arf-ArfGAP complex reveals a
Ca2+ regulatory mechanism. Cell. 141:812–821. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Okabe H, Furukawa Y, Kato T, Hasegawa S,
Yamaoka Y and Nakamura Y: Isolation of development and
differentiation enhancing factor-like 1 (DDEFL1) as a drug target
for hepatocellular carcinomas. Int J Oncol. 24:43–48.
2004.PubMed/NCBI
|
|
10
|
Fang Z, Miao Y, Ding X, Deng H, Liu S,
Wang F, Zhou R, Watson C, Fu C, Hu Q, et al: Proteomic
identification and functional characterization of a novel ARF6
GTPase-activating protein, ACAP4. Mol Cell Proteomics. 5:1437–1449.
2006. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Ding X, Deng H, Wang D, Zhou J, Huang Y,
Zhao X, Yu X, Wang M, Wang F, Ward T, et al: Phospho-regulated
ACAP4-Ezrin interaction is essential for histamine-stimulated
parietal cell secretion. J Biol Chem. 285:18769–18780. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Yu X, Wang F, Liu H, Adams G, Aikhionbare
F, Liu D, Cao X, Fan L, Hu G, Chen Y, et al: ACAP4 protein
cooperates with Grb2 protein to orchestrate epidermal growth
factor-stimulated integrin β1 recycling in cell migration. J Biol
Chem. 286:43735–43747. 2011.PubMed/NCBI
|
|
13
|
Ha VL, Bharti S, Inoue H, Vass WC, Campa
F, Nie Z, de Gramont A, Ward Y and Randazzo PA: ASAP3 is a focal
adhesion-associated Arf GAP that functions in cell migration and
invasion. J Biol Chem. 283:14915–14926. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Pellegrin S and Mellor H: Actin stress
fibres. J Cell Sci. 120:3491–3499. 2007. View Article : Google Scholar
|
|
15
|
He TC, Zhou S, da Costa LT, Yu J, Kinzler
KW and Vogelstein B: A simplified system for generating recombinant
adenoviruses. Proc Natl Acad Sci USA. 95:2509–2514. 1998.
View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Qiu L, Wu X, Chau JF, Szeto IY, Tam WY,
Guo Z, Chung SK, Oates PJ, Chung SS and Yang JY: Aldose reductase
regulates hepatic peroxisome proliferator-activated receptor α
phosphorylation and activity to impact lipid homeostasis. J Biol
Chem. 283:17175–17183. 2008.PubMed/NCBI
|
|
17
|
Herman IM: Actin isoforms. Curr Opin Cell
Biol. 5:48–55. 1993. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Chou CC, Davis RC, Fuller ML, Slovin JP,
Wong A, Wright J, Kania S, Shaked R, Gatti RA and Salser WA:
Gamma-actin: unusual mRNA 3′-untranslated sequence conservation and
amino acid substitutions that may be cancer related. Proc Natl Acad
Sci USA. 84:2575–2579. 1987.
|
|
19
|
Zhu M, Yang T, Wei S, DeWan AT, Morell RJ,
Elfenbein JL, Fisher RA, Leal SM, Smith RJ and Friderici KH:
Mutations in the γ-actin gene (ACTG1) are associated with dominant
progressive deafness (DFNA20/26). Am J Hum Genet. 73:1082–1091.
2003.
|
|
20
|
van Wijk E, Krieger E, Kemperman MH, De
Leenheer EM, Huygen PL, Cremers CW, Cremers FP and Kremer H: A
mutation in the gamma actin 1 (ACTG1) gene causes autosomal
dominant hearing loss (DFNA20/26). J Med Genet. 40:879–884.
2003.PubMed/NCBI
|
|
21
|
de Heer AM, Huygen PL, Collin RW, Oostrik
J, Kremer H and Cremers CW: Audiometric and vestibular features in
a second Dutch DFNA20/26 family with a novel mutation in ACTG1. Ann
Otol Rhinol Laryngol. 118:382–390. 2009.PubMed/NCBI
|
|
22
|
Liu P, Li H, Ren X, Mao H, Zhu Q, Zhu Z,
Yang R, Yuan W, Liu J, Wang Q and Liu M: Novel ACTG1 mutation
causing autosomal dominant non-syndromic hearing impairment in a
Chinese family. J Genet Genomics. 35:553–558. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Morin M, Bryan KE, Mayo-Merino F, Goodyear
R, Mencia A, Modamio-Hoybjor S, del Castillo I, Cabalka JM,
Richardson G, Moreno F, et al: In vivo and in vitro effects of two
novel gamma-actin (ACTG1) mutations that cause DFNA20/26 hearing
impairment. Hum Mol Genet. 18:3075–3089. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Rendtorff ND, Zhu M, Fagerheim T, Antal
TL, Jones M, Teslovich TM, Gillanders EM, Barmada M, Teig E, Trent
JM, et al: A novel missense mutation in ACTG1 causes dominant
deafness in a Norwegian DFNA20/26 family, but ACTG1 mutations are
not frequent among families with hereditary hearing impairment. Eur
J Hum Genet. 14:1097–1105. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Perrin BJ, Sonnemann KJ and Ervasti JM:
β-actin and γ-actin are each dispensable for auditory hair cell
development but required for Stereocilia maintenance. PLoS Genet.
6:e10011582010.
|
|
26
|
Kruth KA and Rubenstein PA: Two
deafness-causing (DFNA20/26) actin mutations affect
Arp2/3-dependent actin regulation. J Biol Chem. 287:27217–27226.
2012. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Belyantseva IA, Perrin BJ, Sonnemann KJ,
Zhu M, Stepanyan R, McGee J, Frolenkov GI, Walsh EJ, Friderici KH,
Friedman TB and Ervasti JM: γ-actin is required for cytoskeletal
maintenance but not development. Proc Natl Acad Sci USA.
106:9703–9708. 2009.
|
|
28
|
Sonnemann KJ, Fitzsimons DP, Patel JR, Liu
Y, Schneider MF, Moss RL and Ervasti JM: Cytoplasmic γ-actin is not
required for skeletal muscle development but its absence leads to a
progressive myopathy. Dev Cell. 11:387–397. 2006.
|
|
29
|
Riviere JB, van Bon BW, Hoischen A,
Kholmanskikh SS, O’Roak BJ, Gilissen C, Gijsen S, Sullivan CT,
Christian SL, Abdul-Rahman OA, et al: De novo mutations in the
actin genes ACTB and ACTG1 cause Baraitser-Winter syndrome. Nat
Genet. 44:440–444. S1–S2. 2012. View
Article : Google Scholar : PubMed/NCBI
|
|
30
|
Bunnell TM and Ervasti JM: Delayed
embryonic development and impaired cell growth and survival in
Actg1 null mice. Cytoskeleton (Hoboken). 67:564–572. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Sun Q, Wang Y, Zhang Y, Liu F, Cheng X,
Hou N, Zhao X and Yang X: Expression profiling reveals
dysregulation of cellular cytoskeletal genes in HBx-induced
hepatocarcinogenesis. Cancer Biol Ther. 6:668–674. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Shum MS, Pasquier E, Po’uha ST, O’Neill
GM, Chaponnier C, Gunning PW and Kavallaris M: γ-Actin regulates
cell migration and modulates the ROCK signaling pathway. FASEB J.
25:4423–4433. 2011.
|
|
33
|
Po’uha ST, Honore S, Braguer D and
Kavallaris M: Partial depletion of gamma-actin suppresses
microtubule dynamics. Cytoskeleton (Hoboken). 70:148–160.
2013.PubMed/NCBI
|
|
34
|
Patel RA, Liu Y, Wang B, Li R and Sebti
SM: Identification of novel ROCK inhibitors with anti-migratory and
anti-invasive activities. Oncogene. Feb 11–2013.(Epub ahead of
print).
|
