|
1
|
Lozsadi DA and Larner AJ: Prevalence and
causes of seizures at the time of diagnosis of probable Alzheimer’s
disease. Dement Geriatr Cogn Disord. 22:121–124. 2006.
|
|
2
|
Del Vecchio RA, Gold LH, Novick SJ, Wong G
and Hyde LA: Increased seizure threshold and severity in young
transgenic CRND8 mice. Neurosci Lett. 367:164–167. 2004.PubMed/NCBI
|
|
3
|
Shoham S and Ebstein RP: The distribution
of beta-amyloid precursor protein in rat cortex after systemic
kainate-induced seizures. Exp Neurol. 147:361–376. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Sheng JG, Boop FA, Mrak RE and Griffin WS:
Increased neuronal beta-amyloid precursor protein expression in
human temporal lobe epilepsy: association with interleukin-1 alpha
immunoreactivity. J Neurochem. 63:1872–1879. 1994. View Article : Google Scholar
|
|
5
|
Willoughby DA, Johnson SA, Pasinetti GM,
et al: Amyloid precursor protein mRNA encoding the Kunitz protease
inhibitor domain is increased by kainic acid-induced seizures in
rat hippocampus. Exp Neurol. 118:332–339. 1992. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Ferreira A, Caceres A and Kosik KS:
Intraneuronal compartments of the amyloid precursor protein. J
Neurosci. 13:3112–3123. 1993.PubMed/NCBI
|
|
7
|
Wieser HG, Blume WT, Fish D, et al: ILAE
Commission Report. Proposal for a new classification of outcome
with respect to epileptic seizures following epilepsy surgery.
Epilepsia. 42:282–286. 2001. View Article : Google Scholar
|
|
8
|
Radhakrishnan VV, Rao MB, Radhakrishnan K,
et al: Pathology of temporal lobe epilepsy: An analysis of 100
consecutive surgical specimens from patients with medically
refractory epilepsy. Neurol India. 47:196–201. 1999.PubMed/NCBI
|
|
9
|
Larner AJ: Axonal sprouting and
synaptogenesis in temporal lobe epilepsy: possible pathogenetic and
therapeutic roles of neurite growth inhibitory factors. Seizure.
4:249–258. 1995. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Nadler JV: The recurrent mossy fiber
pathway of the epileptic brain. Neurochem Res. 28:1649–1658. 2003.
View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Yamazaki T, Selkoe DJ and Koo EH:
Trafficking of cell surface beta-amyloid precursor protein:
retrograde and transcytotic transport in cultured neurons. J Cell
Biol. 129:431–442. 1995. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Kins S, Lauther N, Szodorai A and
Beyreuther K: Subcellular trafficking of the amyloid precursor
protein gene family and its pathogenic role in Alzheimer’s disease.
Neurodegener Dis. 3:218–226. 2006.PubMed/NCBI
|
|
13
|
Smith DH, Uryu K, Saatman KE, Trojanowski
JQ and McIntosh TK: Protein accumulation in traumatic brain injury.
Neuromolecular Med. 4:59–72. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Bramlett HM, Kraydieh S, Green EJ and
Dietrich WD: Temporal and regional patterns of axonal damage
following traumatic brain injury: a beta-amyloid precursor protein
immunocytochemical study in rats. J Neuropathol Exp Neurol.
56:1132–1141. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Perez RG, Zheng H, Van der Ploeg LH and
Koo EH: The beta-amyloid precursor protein of Alzheimer’s disease
enhances neuron viability and modulates neuronal polarity. J
Neurosci. 17:9407–9414. 1997.
|
|
16
|
Gordon-Krajcer W and Gajkowska B:
Excitotoxicity-induced expression of amyloid precursor protein
(beta-APP) in the hippocampus and cortex of rat brain. An
electron-microscopy and biochemical study. Folia Neuropathol.
39:163–173. 2001.PubMed/NCBI
|
|
17
|
Zhu JL, Zhu SH and Ren L: Expression of
beta-amyloid precursor protein in diffuse axonal injury of rats. Fa
Yi Xue Za Zhi. 21:165–168. 2005.(In Chinese).
|
|
18
|
Pollard H, Khrestchatisky M, Moreau J,
Ben-Ari Y and Represa A: Correlation between reactive sprouting and
microtubule protein expression in epileptic hippocampus.
Neuroscience. 61:773–787. 1994. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Blurton-Jones M and Laferla FM: Pathways
by which Abeta facilitates tau pathology. Curr Alzheimer Res.
3:437–448. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Pavia J, de Ceballos ML and Sanchez de la
Cuesta F: Alzheimer’s disease: relationship between muscarinic
cholinergic receptors, beta-amyloid and tau proteins. Fundam Clin
Pharmacol. 12:473–481. 1998.
|
|
21
|
Wang ZF, Li HL, Li XC, et al: Effects of
endogenous beta-amyloid overproduction on tau phosphorylation in
cell culture. J Neurochem. 98:1167–1175. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Ghribi O, Larsen B, Schrag M and Herman
MM: High cholesterol content in neurons increases BACE,
beta-amyloid, and phosphorylated tau levels in rabbit hippocampus.
Exp Neurol. 200:460–467. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Peel AL, Sorscher N, Kim JY, Galvan V,
Chen S and Bredesen DE: Tau phosphorylation in Alzheimer’s disease:
potential involvement of an APP-MAP kinase complex.
Neuromolecular Med. 5:205–218. 2004.
|
|
24
|
Kalaria RN, Bhatti SU, Lust WD and Perry
G: The amyloid precursor protein in ischemic brain injury and
chronic hypoperfusion. Ann NY Acad Sci. 695:190–193. 1993.
View Article : Google Scholar : PubMed/NCBI
|
