|
1
|
Lotze MT and Tracey KJ: High-mobility
group box 1 protein (HMGB1): nuclear weapon in the immune arsenal.
Nat Rev Immunol. 5:331–342. 2005. View
Article : Google Scholar : PubMed/NCBI
|
|
2
|
Vakeva AP, Agah A, Rollins SA, Matis LA,
Li L and Stahl GL: Myocardial infarction and apoptosis after
myocardial ischemia and reperfusion: role of the terminal
complement components and inhibition by anti-C5 therapy.
Circulation. 97:2259–2267. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Frangogiannis NG, Smith CW and Entman ML:
The inflammatory response in myocardial infarction. Cardiovasc Res.
53:31–47. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Andrassy M, Volz HC, Igwe JC, et al:
High-mobility group box-1 in ischemia-reperfusion injury of the
heart. Circulation. 117:3216–3226. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Hu X, Fu W and Jiang H: HMGB1: a potential
therapeutic target for myocardial ischemia and reperfusion injury.
Int J Cardiol. 155:4892012. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Otterbein LE, Soares MP, Yamashita K and
Bach FH: Heme oxygenase-1: unleashing the protective properties of
heme. Trends Immunol. 24:449–455. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Hwa JS, Jin YC, Lee YS, et al:
2-methoxycinnamaldehyde from Cinnamomum cassia reduces rat
myocardial ischemia and reperfusion injury in vivo due to HO-1
induction. J Ethnopharmacol. 139:605–615. 2012.
|
|
8
|
Liu SX, Zhang Y, Wang YF, et al:
Upregulation of heme oxygenase-1 expression by hydroxysafflor
yellow A conferring protection from anoxia/reoxygenation-induced
apoptosis in H9c2 cardiomyocytes. Int J Cardiol. 160:95–101. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Ha YM, Ham SA, Kim YM, et al:
β1-adrenergic receptor-mediated HO-1 induction, via PI3K
and p38 MAPK, by isoproterenol in RAW 264.7 cells leads to
inhibition of HMGB1 release in LPS-activated RAW 264.7 cells and
increases in survival rate of CLP-induced septic mice. Biochem
Pharmacol. 82:769–777. 2011.
|
|
10
|
Jin YC, Kim CW, Kim YM, et al:
Cryptotanshinone, a lipophilic compound of Salvia
miltiorrriza root, inhibits TNF-α-induced expression of
adhesion molecules in HUVEC and attenuates rat myocardial
ischemia/reperfusion injury in vivo. Eur J Pharmacol. 614:91–97.
2009.PubMed/NCBI
|
|
11
|
Wang J, Yang H, Hu X, et al:
Dobutamine-mediated heme oxygenase-1 induction via PI3K and p38
MAPK inhibits high mobility group box 1 protein release and
attenuates rat myocardial ischemia/reperfusion injury in vivo. J
Surg Res. 183:509–516. 2013. View Article : Google Scholar
|
|
12
|
Yan L, Tang Q, Shen D, et al: SOCS-1
inhibits TNF-α-induced cardiomyocyte apoptosis via ERK1/2 pathway
activation. Inflammation. 31:180–188. 2008.
|
|
13
|
Yang J, Jiang H, Yang J, Ding JW, Chen LH,
Li S and Zhang XD: Valsartan preconditioning protects against
myocardial ischemia-reperfusion injury through TLR4/NF-κB signaling
pathway. Mol Cell Biochem. 330:39–46. 2009.PubMed/NCBI
|
|
14
|
Inoue K, Kawahara K, Biswas KK, et al:
HMGB1 expression by activated vascular smooth muscle cells in
advanced human atherosclerosis plaques. Cardiovasc Pathol.
16:136–143. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Arroyo-Espliguero R, Avanzas P, Quiles J
and Kaski JC: Predictive value of coronary artery stenoses and
C-reactive protein levels in patients with stable coronary artery
disease. Atherosclerosis. 204:239–243. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Andersson U, Wang H, Palmblad K, et al:
High mobility group 1 protein (HMG-1) stimulates proinflammatory
cytokine synthesis in human monocytes. J Exp Med. 192:565–570.
2000. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Takamiya R, Hung CC, Hall SR, et al:
High-mobility group box 1 contributes to lethality of endotoxemia
in heme oxygenase-1-deficient mice. Am J Respir Cell Mol Biol.
41:129–135. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Tsoyi K, Lee TY, Lee YS, et al:
Heme-oxygenase-1 induction and carbon monoxide-releasing molecule
inhibit lipopolysaccharide (LPS)-induced high-mobility group box 1
release in vitro and improve survival of mice in LPS- and cecal
ligation and puncture-induced sepsis model in vivo. Mol Pharmacol.
76:173–182. 2009. View Article : Google Scholar
|
|
19
|
Guirao X, Kumar A, Katz J, et al:
Catecholamines increase monocyte TNF receptors and inhibit TNF
through β2-adrenoreceptor activation. Am J Physiol.
273:E1203–E1208. 1997.PubMed/NCBI
|
|
20
|
Kappel M, Poulsen TD, Galbo H and Pedersen
BK: Effects of elevated plasma noradrenaline concentration on the
immune system in humans. Eur J Appl Physiol Occup Physiol.
79:93–98. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Sanders VM, Baker RA, Ramer-Quinn DS,
Kasprowicz DJ, Fuchs BA and Street NE: Differential expression of
the beta2-adrenergic receptor by Th1 and Th2 clones: implications
for cytokine production and B cell help. J Immunol. 158:4200–4210.
1997.PubMed/NCBI
|
|
22
|
Muthu K, He LK, Szilagyi A, Strotmon P,
Gamelli RL and Shankar R: β-adrenergic stimulation increases
macrophage CD14 expression and E. coli phagocytosis through
PKA signaling mechanisms. J Leukoc Biol. 88:715–724. 2010.
|
|
23
|
Liu S, Morris SM Jr, Nie S, Shapiro RA and
Billiar TR: cAMP induces CD14 expression in murine macrophages via
increased transcription. J Leukoc Biol. 67:894–901. 2000.PubMed/NCBI
|
|
24
|
Gegner JA, Ulevitch RJ and Tobias PS:
Lipopolysaccharide (LPS) signal transduction and clearance. Dual
roles for LPS binding protein and membrane CD14. J Biol Chem.
270:5320–5325. 1995. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Tsung A, Klune JR, Zhang X, et al: HMGB1
release induced by liver ischemia involves Toll-like receptor 4
dependent reactive oxygen species production and calcium-mediated
signaling. J Exp Med. 204:2913–2923. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Yu Y, Liu M, Zhang L, et al: Heat shock
transcription factor 1 inhibits H2O2-induced
cardiomyocyte death through suppression of high-mobility group box
1. Mol Cell Biochem. 364:263–269. 2012.PubMed/NCBI
|
|
27
|
Iles KE, Dickinson DA, Wigley AF, Welty
NE, Blank V and Forman HJ: HNE increases HO-1 through activation of
the ERK pathway in pulmonary epithelial cells. Free Radic Biol Med.
39:355–364. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Ryter SW and Choi AM: Heme oxygenase-1:
redox regulation of a stress protein in lung and cell culture
models. Antioxid Redox Signal. 7:80–91. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Eom HW, Park SY, Kim YH, et al: Bambusae
Caulis in Taeniam modulates neuroprotective and
anti-neuroinflammatory effects in hippocampal and microglial cells
via HO-1- and Nrf-2-mediated pathways. Int J Mol Med. 30:1512–1520.
2012.PubMed/NCBI
|
|
30
|
Jin GH, Park SY, Kim E, et al:
Anti-inflammatory activity of Bambusae Caulis in Taeniam through
heme oxygenase-1 expression via Nrf-2 and p38 MAPK signaling in
macrophages. Environ Toxicol Pharmacol. 34:315–323. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Senthil Kumar KJ, Liao JW, Xiao JH, Gokila
Vani M and Wang SY: Hepatoprotective effect of lucidone against
alcohol-induced oxidative stress in human hepatic HepG2 cells
through the up-regulation of HO-1/Nrf-2 antioxidant genes. Toxicol
In Vitro. 26:700–708. 2012.PubMed/NCBI
|
|
32
|
Ryu EY, Park AJ, Park SY, et al:
Inhibitory effects of Ginkgo biloba extract on inflammatory
mediator production by Porphyromonas gingivalis
lipopolysaccharide in murine macrophages via Nrf-2 mediated heme
oxygenase-1 signaling pathways. Inflammation. 35:1477–1486. 2012.
View Article : Google Scholar : PubMed/NCBI
|
