|
1
|
Krysko DV, Leybaert L, Vandenabeele P and
D’Herde K: Gap junctions and the propagation of cell survival and
cell death signals. Apoptosis. 10:459–469. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Peixoto PM, Ryu SY, Pruzansky DP,
Kuriakose M, Gilmore A and Kinnally KW: Mitochondrial apoptosis is
amplified through gap junctions. Biochem Biophys Res Commun.
390:38–43. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Sahores M and Mendoza-Naranjo A: Gap
junctions as therapeutic targets in brain injury following
hypoxia-ischemia. Recent Pat CNS Drug Discov. 3:209–315. 2008.
View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Spray DC, Hanstein R, Lopez-Quintero SV,
Stout RF Jr, Suadicani SO and Thi MM: Gap junctions and Bystander
effects: Good Samaritans and executioners. Wiley Interdiscip Rev
Membr Transp Signal. 2:1–15. 2013. View
Article : Google Scholar : PubMed/NCBI
|
|
5
|
Davidson JO, Green CR, Bennel L, Nicholson
LF, Danesh-Meyer H, O’Carroll SJ and Gunn AJ: A key role for
connexin hemichannels in spreading ischemic brain injury. Curr Drug
Targets. 14:36–46. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Rami A, Volkmann T and Winckler J:
Effective reduction of neuronal death by inhibiting gap junctional
intercellular communication in a rodent model of global transient
cerebral ischemia. Exp Neurol. 170:297–304. 2001. View Article : Google Scholar
|
|
7
|
Xie M, Yi C, Luo X, et al: Glial gap
junctionsal communication involvement in hippocampal damage after
middle cerebral artery occlusion. Ann Neurol. 70:121–132. 2011.
View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Lin JH, Weigel H, Cotrina ML, et al:
Gap-junction-mediated propagation and amplification of cell injury.
Nat Neurosci. 1:494–500. 1998. View
Article : Google Scholar : PubMed/NCBI
|
|
9
|
Blanc EM, Bruce-Keller AJ and Mattson MP:
Astrocytic gap junctional communication decreases neuronal
vulnerability to oxidative stress-induced disruption of
Ca2+ homeostasis and cell death. J Neurochem.
70:958–970. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Ozog MA, Siushansian R and Naus CC:
Blocking gap junctional coupling increases glutamate-induced
neurotoxicity in neuron-astrocyte co-cultures. J Neuropathol Exp
Neurol. 61:132–141. 2002.PubMed/NCBI
|
|
11
|
Lee J, Yim YS, Ko SJ, Kim DG and Kim CH:
Gap junctions contribute to astrocytic resistance against zinc
toxicity. Brain Res Bull. 86:314–318. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Chew SS, Johnson CS, Green CR and
Danesh-Meyer HV: Role of connexin43 in central nervous system
injury. Exp Neurol. 225:250–261. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Bendel O, Alkass K, Bueters T, von Euler M
and von Euler G: Reproducible loss of CA1 neurons following carotid
artery occlusion combined with halothane-induced hypotension. Brain
Res. 1033:135–142. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Colbourne F, Li H and Buchan AM:
Continuing postischemic neuronal death in CA1: influence of
ischemia duration and cytoprotective doses of NBQX and SNX-111 in
rats. Stroke. 30:662–668. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Rawanduzy A, Hansen A, Hansen TW and
Nedergaard M: Effective reduction of infarct volume by gap junction
blockade in a rodent model of stroke. J Neurosurg. 87:916–920.
1997.
|
|
16
|
Kaido T, Kempski O, Heimann A, Heers C and
Bartsch D: Cluster analysis of mRNA expression levels identifies
multiple sequential patterns following focal cerebral ischemia.
Turk Neurosurg. 22:441–447. 2012.
|
|
17
|
Longa EZ, Weinstein PR, Carlson S and
Cummins R: Reversible middle cerebral artery occlusion without
craniectomy in rats. Stroke. 20:84–91. 1989. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Zivin JA: Factors determining the
therapeutic window for stroke. Neurology. 50:599–603. 1998.
View Article : Google Scholar : PubMed/NCBI
|
|
19
|
George A, Marziniak M, Schäfers M, Toyka
KV and Sommer C: Thalidomide treatment in chronic constrictive
neuropathy decreases endoneurial tumor necrosis factor-alpha,
increases interleukin-10 and has long-term effects on spinal cord
dorsal horn met-enkephalin. Pain. 88:267–275. 2000. View Article : Google Scholar
|
|
20
|
Narahashi T, Aistrup GL, Lindstrom JM,
Marszalec W, Nagata K, Wang F and Yeh JZ: Ion channel modulation as
the basis for general anesthesia. Toxicol Lett. 100:185–191. 1998.
View Article : Google Scholar
|
|
21
|
Contreras JE, Sánchez HA, Véliz LP,
Bukauskas FF, Bennett MV and Sáez JC: Role of connexin-based gap
junction channels and hemichannels in ischemia-induced cell death
in nervous tissue. Brain Res Brain Res Rev. 47:290–303. 2004.
View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Retamal MA, Schalper KA, Shoji KF,
Orellana JA, Bennett MV and Sáez JC: Possible involvement of
different connexin43 domains in plasma membrane permeabilization
induced by ischemia-reperfusion. J Membr Biol. 218:49–63. 2007.
View Article : Google Scholar
|
|
23
|
Rouach N, Avignone E, Même W, Koulakoff A,
Venance L, Blomstrand F and Giaume C: Gap junctions and connexin
expression in the normal and pathological central nervous system.
Biol Cell. 94:457–475. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Talhouk RS, Zeinieh MP, Mikati MA and
El-Sabban ME: Gap junctional intercellular communication in
hypoxia-ischemia-induced neuronal injury. Prog Neurobiol. 84:57–76.
2008. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Lin JH, Lou N, Kang N, et al: A central
role of connexin 43 in hypoxic preconditioning. J Neurosci.
28:681–695. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Nakase T, Fushiki S and Naus CC:
Astrocytic gap junctions composed of connexin 43 reduce apoptotic
neuronal damage in cerebral ischemia. Stroke. 34:1987–1993. 2003.
View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Nakase T, Fushiki S, Söhl G, Theis M,
Willecke K and Naus CC: Neuroprotective role of astrocytic gap
junctions in ischemic stroke. Cell Commun Adhes. 10:413–417. 2003.
View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Nakase T, Sähl G, Theis M, Willecke K and
Naus CC: Increased apoptosis and inflammation after focal brain
ischemia in mice lacking connexin43 in astrocytes. Am J Pathol.
164:2067–2075. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Siushansian R, Bechberger JF, Cechetto DF,
Hachinski VC and Naus CC: Connexin43 null mutation increases
infarct size after stroke. J Comp Neurol. 440:387–394. 2001.
View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Wang F, Hai J and Jing Y: Gap junctions
communication involved in brain protection following focal ischemia
and reperfusion in rats. Neural Regen Res. 9:677–682. 2009.(In
Chinese).
|
|
31
|
Perez Velazquez JL, Kokarovtseva L,
Sarbaziha R, Jeyapalan Z and Leshchenko Y: Role of gap junctionsal
coupling in astrocytic networks in the determination of global
ischaemia-induced oxidative stress and hippocampal damage. Eur J
Neurosci. 23:1–10. 2006.PubMed/NCBI
|
|
32
|
Saito R, Graf R, Hübel K, Fujita T, Rosner
G and Heiss WD: Reduction of infarct volume by halothane: effect on
cerebral blood flow or perifocal spreading depression-like
depolarizations. J Cereb Blood Flow Metab. 17:857–864.
1997.PubMed/NCBI
|
|
33
|
de Pina-Benabou MH, Szostak V, Kyrozis A,
et al: Blockade of gap junctions in vivo provides neuroprotection
after perinatal global ischemia. Stroke. 36:2232–2237.
2005.PubMed/NCBI
|
|
34
|
Martinou JC, Dubois-Dauphin M, Staple JK,
et al: Overexpression of BCL-2 in transgenic mice protects neurons
from naturally occurring cell death and experimental ischemia.
Neuron. 13:1017–1030. 1994. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Chen J, Simon RP, Nagayama T, Zhu R,
Loeffert JE, Watkins SC and Graham SH: Suppression of endogenous
bcl-2 expression by antisense treatment exacerbates ischemic
neuronal death. J Cereb Blood Flow Metab. 20:1033–1039. 2000.
View Article : Google Scholar : PubMed/NCBI
|
|
36
|
MacManus JP and Linnik MD: Gene expression
induced by cerebral ischemia: an apoptotic perspective. J Cereb
Blood Flow Metab. 17:815–832. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Han B, Wang Q, Cui G, Shen X and Zhu Z:
Post-treatment of Bax-inhibiting peptide reduces neuronal death and
behavioral deficits following global cerebral ischemia. Neurochem
Int. 58:224–233. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Xi HJ, Zhang TH, Tao T, Song CY, Lu SJ,
Cui XG and Yue ZY: Propofol improved neurobehavioral outcome of
cerebral ischemia-reperfusion rats by regulating Bcl-2 and Bax
expression. Brain Res. 1410:24–32. 2011. View Article : Google Scholar : PubMed/NCBI
|
