|
1
|
Yang W, Lu J, Weng J, et al: Prevalence of
diabetes among men and women in China. New Engl J Med.
362:1090–1101. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Ceriello A: New insights on oxidative
stress and diabetic complications may lead to a “causal”
antioxidant therapy. Diabetes Care. 26:1589–1596. 2003.
|
|
3
|
Collins AJ, Kasiske B, Herzog C, et al:
Excerpts from the United States Renal Data System 2003 Annual Data
Report: atlas of end-stage renal disease in the United States. Am J
Kidney Dis. 42:A5–A7. S1–S230. 2003.
|
|
4
|
Caramori ML, Fioretto P and Mauer M: Low
glomerular filtration rate in normoal buminuric type 1 diabetic
patients: an indicator of more advanced glomerular lesions.
Diabetes. 52:1036–1040. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Wada J and Makino H: Inflammation and the
pathogenesis of diabetic nephropathy. Clin Sci (Lond). 124:139–152.
2013. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Ha H, Yu MR and Kim KH: Melatonin and
taurine reduce early glomerulopathy in diabetic rats. Free Radic
Biol Med. 26:944–950. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Li SY, Fu ZJ and Lo AC: Hypoxia-induced
oxidative stress in ischemic retinopathy. Oxid Med Cell Longev.
2012:e4267692012.
|
|
8
|
Pan HZ, Zhang L, Guo MY, et al: The
oxidative stress status in diabetes mellitus and diabetic
nephropathy. Acta Diabetol. 47:71–76. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Mansouri E, Panahi M, Ghaffari MA, et al:
Effects of grape seed proanthocyanidin extract on oxidative stress
induced by diabetes in rat kidney. Iran Biomed J. 15:100–106.
2011.PubMed/NCBI
|
|
10
|
Kanasaki K, Taduri G and Koya D: Diabetic
nephropathy: the role of inflammation in fibroblast activation and
kidney fibrosis. Front Endocrinol (Lausanne). 4:72013.PubMed/NCBI
|
|
11
|
Lim AK and Tesch GH: Inflammation in
diabetic nephropathy. Mediators Inflamm. 2012:146–154. 2012.
|
|
12
|
Rivero A, Mora C, Muros M, et al:
Pathogenic perspectives for the role of inflammation in diabetic
nephropathy. Clin Sci (Lond). 116:479–492. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Gui D, Huang J, Guo Y, et al:
Astragaloside IV ameliorates renal injury in streptozotocin-induced
diabetic rats through inhibiting NF-κB-mediated inflammatory genes
expression. Cytokine. 61:970–977. 2013.PubMed/NCBI
|
|
14
|
Zhang XY, Li WG, Wu YJ, et al:
Proanthocyanidin from grape seeds enhances doxorubicin-induced
antitumor effect and reverses drug resistance in
doxorubicin-resistant K562/DOX cells. Can J Physiol Pharm.
83:309–318. 2005. View
Article : Google Scholar : PubMed/NCBI
|
|
15
|
Boghdady NA: Antioxidant and antiapoptotic
effects of proanthocyanidin and ginkgo biloba extract against
doxorubicin-induced cardiac injury in rats. Cell Biochem Funct.
31:344–351. 2013. View
Article : Google Scholar : PubMed/NCBI
|
|
16
|
Mansouri E, Panahi M, Ghaffari MA and
Ghorbani A: Effects of grape seed proanthocyanidin extract on
oxidative stress induced by diabetes in rat kidney. Iran Biomed J.
15:100–106. 2011.PubMed/NCBI
|
|
17
|
Li X, Xiao Y, Gao H, et al: Grape seed
proanthocyanidins ameliorate diabetic nephropathy via modulation of
levels of AGE, RAGE and CTGF. Nephron Exp Nephrol. 111:e31–e41.
2009. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Kolset SO, Reinholt FP and Jenssen T:
Diabetic nephropathy and extracellular matrix. J Histochem
Cytochem. 60:976–986. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Furness PN: Basement membrane synthesis
and degradation. J Pathol. 183:1–3. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Wu K, Setty S, Mauer SM, et al: Altered
kidney matrix gene expression in early stages of experimental
diabetes. Acta Anat (Basel). 158:155–165. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Han SY, Jee YH, Han KH, et al: An
imbalance between matrix metalloproteinase-2 and tissue inhibitor
of matrix metalloproteinase-2 contributes to the development of
early diabetic nephropathy. Nephrol Dial Transplant. 21:2406–2416.
2006. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Hadler-Olsen E, Winberg JO, Reinholt FP,
et al: Proteases in Plasma and Kidney of db/db Mice as Markers of
Diabetes-Induced Nephropathy. ISRN Endocrinol. 2011:e8326422011.
View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Efficacy of atenolol and captopril in
reducing risk of macrovascular and microvascular complications in
type 2 diabetes: UKPDS 39. UK Prospective Diabetes Study Group.
BMJ. 317:713–720. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Pinent M, Blay M, Bladé MC, et al: Grape
seed-derived procyanidins have an antihyperglycemic effect in
streptozotocin-induced diabetic rats and insulin-omimetic activity
in insulin-sensitive cell lines. Endocrinology. 145:4985–4990.
2004. View Article : Google Scholar
|
|
25
|
El-Alfy AT, Ahmed AA and Fatani AJ:
Protective effect of red grape seeds proanthocyanidins against
induction of diabetes by alloxan in rats. Pharmacol Res.
52:264–270. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Li BY, Cheng M, Gao HQ, et al:
Back-regulation of six oxidative stress proteins with grape seed
proanthocyanidin extracts in rat diabetic nephropathy. J Cell
Biochem. 104:668–679. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Cui Xiaopei, Liu Xiangju, Feng Hua, et al:
Grape seed proanthocyanidin extracts enhance endothelial nitric
oxide synthase expression through 5′-AMP activated protein
kinase/Surtuin 1-Krüpple like factor 2 pathway and modulate blood
pressure in ouabain induced hypertensive rats. Biol Pharm Bull.
35:2192–2197. 2012.PubMed/NCBI
|
|
28
|
Wu S, Guo R, Guo R, et al: Effects of
grape seed proanthocyanidin extracts on renal vascular hypertension
in rats. Chin J Pathophysiol. 27:593–595. 2011.
|
|
29
|
Ha H and Lee HB: Reactive oxygen species
as glucose signaling molecules in mesangial cells cultured under
high glucose. Kidney Int Suppl. 77:S19–S25. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Scivittaro V, Ganz MB and Weiss MF: AGEs
induce oxidative stress and activate protein kinase C-beta (II) in
neonatal mesangial cells. Am J Physiol Renal Physiol.
278:F676–F683. 2000.PubMed/NCBI
|
|
31
|
Tomlinson DR: Mitogen-activated protein
kinases as glucose transducers for diabetic complications.
Diabetologia. 42:1271–1281. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Suzuki S, Hinokio Y, Komatu K, et al:
Oxidative damage to mitochondrial DNA and its relationship to
diabetic complications. Diabetes Res Clin Pract. 45:161–168. 1999.
View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Kedziora K, Szram S, Komatowski T, et al:
Effect of vitamin E and vitamin C supplementation on antioxidative
state and renal glomendar basement membrane thickness in diabetic
kidney. Nephron Exp Nephrol. 95:134–143. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Bhatti F, Mankhey RW, Asico L, et al:
Mechanisms of antioxidant and pro-oxidant effects of alpha-lipoic
acid in the diabetic and nondiabetic kidney. Kidney Int.
67:1371–1380. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Naito Y, Uchiyama K, Aoi W, et al:
Prevention of diabetic nephropathy by treatment with astaxanthm in
diabetic db/db mice. Biofactors. 20:49–59. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Tam FW, Riser BL, Meeran K, et al: Urinary
monocyte chemoattractant protein-1 (MCP-1) and connective tissue
growth factor (CCN2) as prognostic markers for progression of
diabetic nephropathy. Cytokine. 47:37–42. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Navarro-González JF, Jarque A, Muros M, et
al: Tumor necrosis factor-alpha as a therapeutic target for
diabetic nephropathy. Cytokine Growth Factor Rev. 20:165–173.
2009.PubMed/NCBI
|
|
38
|
Rivero A, Mora C, Muros M, et al:
Pathogenic perspectives for the role of inflammation in diabetic
nephropathy. Clin Sci (Lond). 116:479–492. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
39
|
Chow FY, Nikolic-Paterson DJ, Ma FY, et
al: Monocyte chemoattractant protein-1-induced tissue inflammation
is critical for the development of renal injury but not type 2
diabetes in obese db/db mice. Diabetologia. 50:471–480. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
40
|
Navarro JF, Mora C, Muros M, et al:
Urinary tumour necrosis factor-alpha excretion independently
correlates with clinical markers of glomerular and
tubulointerstitial injury in type 2 diabetic patients. Nephrol Dial
Transpl. 21:3428–3434. 2006. View Article : Google Scholar
|
|
41
|
Okada S, Shikata K, Matsuda M, et al:
Intercellular adhesion molecule-1-deficient mice are resistant
against renal injury after induction of diabetes. Diabetes.
52:2586–2593. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
42
|
Anatoliotakis N, Deftereos S, Bouras G, et
al: Myeloperoxidase: expressing inflammation and oxidative stress
in cardiovascular disease. Curr Top Med Chem. 13:115–138. 2013.
View Article : Google Scholar : PubMed/NCBI
|
|
43
|
Crowley SD: The cooperative roles of
inflammation and oxidative stress in the pathogenesis of
hypertension. Antioxid Redox Signal. 20:102–120. 2014. View Article : Google Scholar : PubMed/NCBI
|
