|
1
|
Moro T, Kyomoto M, Ishihara K, et al:
Grafting of poly(2-methacryloyloxyethyl phosphorylcholine) on
polyethylene liner in artificial hip joints reduces production of
wear particles. J Mech Behav Biomed Mater. 31:100–106. 2014.
View Article : Google Scholar
|
|
2
|
Fujii J, Yasunaga Y, Yamasaki A and Ochi
M: Wear debris stimulates bone-resorbing factor expression in the
fibroblasts and osteoblasts. Hip Int. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Antonios JK, Yao Z, Li C, Rao AJ and
Goodman SB: Macrophage polarization in response to wear particles
in vitro. Cell Mol Immunol. 10:471–482. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Nich C, Takakubo Y, Pajarinen J, et al:
Macrophages-Key cells in the response to wear debris from joint
replacements. J Biomed Mater Res A. 101:3033–3045. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Shimamura M, Na kagami H, Osa ko MK, et
al: OPG/RANKL/RANK axis is a critical inflammatory signaling system
in ischemic brain in mice. Proc Natl Acad Sci USA. 111:8191–8196.
2014. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Ma B, Zhang Q, Wu D, et al: Strontium
fructose 1,6-diphosphate prevents bone loss in a rat model of
postmenopausal osteoporosis via the OPG/RANKL/RANK pathway. Acta
Pharmacol Sin. 33:479–489. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Zhang X, Paun A, Claudio E, Wang H and
Siebenlist U: The tumor promoter and NF-κB modulator Bcl-3
regulates splenic B cell development. J Immunol. 191:5984–5992.
2013. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Dwyer BJ, Olynyk JK, Ramm GA and
Tirnitz-Parker JE: TWEAK and LTβ signaling during chronic liver
disease. Front Immunol. 5:392014. View Article : Google Scholar
|
|
9
|
Martin P, Mora I, Cortes A, et al:
Relevant role of cGMP-dependent protein kinase (PKG) in the
progression of fibrosis induced by TNF-like weak inducer of
apoptosis (TWEAK). Am J Physiol Renal Physiol. 307:F75–F85. 2014.
View Article : Google Scholar
|
|
10
|
Winkles JA: The TWEAK-Fn14
cytokine-receptor axis: discovery, biology and therapeutic
targeting. Nat Rev Drug Discov. 7:411–425. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Yang Q, Xu Y, Feng G, et al: p38 MAPK
signal pathway involved in anti-inflammatory effect of
Chaihu-Shugan-San and Shen-ling-bai-zhu-San on hepatocyte in
non-alcoholic steatohepatitis rats. Afr J Tradit Complement Altern
Med. 11:213–221. 2014.PubMed/NCBI
|
|
12
|
Moreira V, Lomonte B, Vinolo MA, Curi R,
Gutierrez JM and Teixeira C: An Asp49 phospholipase A2 from snake
venom induces cyclooxygenase-2 expression and prostaglandin E2
production via activation of NF-kappa B, p38MAPK, and PKC in
Macrophages. Mediators Inflamm. 2014:2014. View Article : Google Scholar
|
|
13
|
Wang X and Liu Y: Regulation of innate
immune response by MAP kinase phosphatase-1. Cell Signal.
19:1372–1382. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Zhi-Chun L, Qiao-Ling Z, Zhi-Qin L,
Xiao-Zhao L, Xiao-xia Z and Rong T: Tumor necrosis factor-like weak
inducer of apoptosis (TWEAK) mediates p38 mitogen-activated protein
kinase activation and signal transduction in peripheral blood
mononuclear cells from patients with lupus nephritis. Inflammation.
35:935–943. 2012. View Article : Google Scholar
|
|
15
|
Li H, Mittal OK, Paul PK, et al: Tumor
necrosis factor-related weak inducer of apoptosis augments matrix
metalloproteinase 9 (MMP-9) production in skeletal muscle through
the activation of nuclear factor-kappaB-inducing kinase and p38
mitogen-activated protein kinase: A potential role of MMP-9 in
myopathy. J Biol Chem. 284:4439–4450. 2009. View Article : Google Scholar :
|
|
16
|
Wisniacki N, Amaravadi L, Galluppi GR, et
al: Safety, tolerability, pharmacokinetics, and pharmacodynamics of
anti-TWEAK monoclonal antibody in patients with rheumatoid
arthritis. Clin Ther. 35:1137–1149. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Xue K, Qi L, Zhou G and Liu K: A two-step
method of constructing mature cartilage using bone marrow-derived
mesenchymal stem cells. Cells Tissues Organs. 197:484–495. 2013.
View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Schultz DC, Bazel S, Wright LM, et al:
Western blotting and enzymatic activity analysis of cathepsin D in
breast tissue and sera of patients with breast cancer and benign
breast disease and of normal controls. Cancer Res. 54:48–54.
1994.PubMed/NCBI
|
|
19
|
Xiao W, Chen X and He M: Inhibition of the
Jagged/Notch pathway inhibits retinoblastoma cell proliferation via
suppressing the PI3K/Akt, Src, p38MAPK and Wnt/βcatenin signaling
pathways. Mol Med Rep. 10:453–458. 2014.PubMed/NCBI
|
|
20
|
Purdue PE, Koulouvaris P, Potter HG,
Nestor BJ and Sculco TP: The cellular and molecular biology of
periprosthetic osteolysis. Clin Orthop Relat Res. 454:251–261.
2007. View Article : Google Scholar
|
|
21
|
Gallo J, Goodman SB, Konttinen YT and
Raska M: Particle disease: biologic mechanisms of periprosthetic
osteolysis in total hip arthroplasty. Innate Immun. 19:213–224.
2013. View Article : Google Scholar :
|
|
22
|
Ingham E and Fisher J: The role of
macrophages in osteolysis of total joint replacement. Biomaterials.
26:1271–1286. 2005. View Article : Google Scholar
|
|
23
|
Pajarinen J, Kouri VP, Jamsen E, Li TF,
Mandelin J and Konttinen YT: The response of macrophages to
titanium particles is determined by macrophage polarization. Acta
Biomater. 9:9229–9240. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Goodman SB, Ma T, Spanogle J, et al:
Effects of a p38 MAP kinase inhibitor on bone ingrowth and tissue
differentiation in rabbit chambers. J Biomed Mater Res A.
81:310–316. 2007. View Article : Google Scholar
|
|
25
|
Tsirigotis M, Baldwin RM, Tang MY, Lorimer
IA and Gray DA: Activation of p38MAPK contributes to expanded
polyglutamine-induced cytotoxicity. PloS One. 3:e21302008.
View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Desplat-Jégo S, Feuillet L, Creidy R, et
al: TWEAK is expressed at the cell surface of monocytes during
multiple sclerosis. J Leukoc Biol. 85:132–135. 2009. View Article : Google Scholar
|
|
27
|
Mao X, Pan X, Peng X, Cheng T and Zhang X:
Inhibition of titanium particle-induced inflammation by the
proteasome inhibitor bortezomib in murine macrophage-like RAW264.7
cells. Inflammation. 35:1411–1418. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Goodman SB, Gibon E, Pajarinen J, et al:
Novel biological strategies for treatment of wear particle-induced
periprosthetic osteolysis of orthopaedic implants for joint
replacement. J R Soc Interface. 11:201309622014. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
van Kuijk AW, Wijbrandts CA, Vinkenoog M,
Zheng TS, Reedquist KA and Tak PP: TWEAK and its receptor Fn14 in
the synovium of patients with rheumatoid arthritis compared to
psoriatic arthritis and its response to tumour necrosis factor
blockade. Ann Rheum Dis. 69:301–304. 2010. View Article : Google Scholar
|
|
30
|
Perper SJ, Browning B, Burkly LC, et al:
TWEAK is a novel arthritogenic mediator. J Immunol. 177:2610–2620.
2006. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Neale SD and Athanasou NA: Cytokine
receptor profile of arthroplasty macrophages, foreign body giant
cells and mature osteoclasts. Acta Orthop Scand. 70:452–458. 1999.
View Article : Google Scholar
|
