|
1
|
Siegel R, Naishadham D and Jemal A: Cancer
statistics, 2013. CA Cancer J Clin. 63:11–30. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
DeSantis C, Ma J, Bryan L and Jemal A:
Breast cancer statistics, 2013. CA Cancer J Clin. 64:52–62. 2014.
View Article : Google Scholar
|
|
3
|
Murphy IG, Dillon MF, Doherty AO, et al:
Analysis of patients with false negative mammography and
symptomatic breast carcinoma. J Surg Oncol. 96:457–463. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Chen J and Wang X: MicroRNA-21 in breast
cancer: diagnostic and prognostic potential. Clin Transl Oncol.
16:225–233. 2014. View Article : Google Scholar
|
|
5
|
Angell JE, Lindner DJ, Shapiro PS, Hofmann
ER and Kalvakolanu DV: Identification of GRIM-19, a novel cell
death-regulatory gene induced by the interferon-beta and retinoic
acid combination, using a genetic approach. J Biol Chem.
275:33416–33426. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Fearnley IM, Carroll J, Shannon RJ,
Runswick MJ, Walker JE and Hirst J: GRIM-19, a cell death
regulatory gene product, is a subunit of bovine mitochondrial
NADH:ubiquinone oxidoreductase (complex I). J Biol Chem.
276:38345–38348. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Huang G, Lu H, Hao A, et al: GRIM-19, a
cell death regulatory protein, is essential for assembly and
function of mitochondrial complex I. Mol Cell Biol. 24:8447–8456.
2004. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Li F, Ren W, Zhao Y, et al: Downregulation
of GRIM-19 is associated with hyperactivation of p-STAT3 in
hepatocellular carcinoma. Med Oncol. 29:3046–3054. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Alchanati I, Nallar SC, Sun P, et al: A
proteomic analysis reveals the loss of expression of the cell death
regulatory gene GRIM-19 in human renal cell carcinomas. Oncogene.
25:7138–7147. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Zhou Y, Li M, Wei Y, et al:
Down-regulation of GRIM-19 expression is associated with
hyperactivation of STAT3-induced gene expression and tumor growth
in human cervical cancers. J Interferon Cytokine Res. 29:695–703.
2009. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Fan XY, Jiang ZF, Cai L and Liu RY:
Expression and clinical significance of GRIM-19 in lung cancer. Med
Oncol. 29:3183–3189. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Alchanati I, Nallar SC, Sun P, et al: A
proteomic analysis reveals the loss of expression of the cell death
regulatory gene GRIM-19 in human renal cell carcinomas. Oncogene.
25:7138–7147. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Wen LJ, Gao LF, Jin CS, et al: Small
interfering RNA survivin and GRIM-19 co-expression salmonella
plasmid inhibited the growth of laryngeal cancer cells in vitro and
in vivo. Int J Clin Exp Pathol. 6:2071–2081. 2013.PubMed/NCBI
|
|
14
|
Fusco A, Viglietto G and Santoro M: Point
mutation in GRIM-19: a new genetic lesion in Hurthle cell thyroid
carcinomas. Br J Cancer. 92:1817–1818. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Máximo V, Botelho T, Capela J, et al:
Somatic and germline mutation in GRIM-19, a dual function gene
involved in mitochondrial metabolism and cell death, is linked to
mitochondrion-rich (Hurthle cell) tumours of the thyroid. Br J
Cancer. 92:1892–1898. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Nallar SC, Kalakonda S, Lindner DJ, et al:
Tumor-derived mutations in the gene associated with retinoid
interferon-induced mortality (GRIM-19) disrupt its anti-signal
transducer and activator of transcription 3 (STAT3) activity and
promote oncogenesis. J Biol Chem. 288:7930–7941. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Lu Y, Fukuyama S, Yoshida R, et al: Loss
of SOCS3 gene expression converts STAT3 function from
anti-apoptotic to pro-apoptotic. J Biol Chem. 281:36683–36690.
2006. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Huang Y, Yang M, Yang H and Zeng Z:
Upregulation of the GRIM-19 gene suppresses invasion and metastasis
of human gastric cancer SGC-7901 cell line. Exp Cell Res.
316:2061–2070. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Huang G, Chen Y, Lu H and Cao X: Coupling
mitochondrial respiratory chain to cell death: an essential role of
mitochondrial complex I in the interferon-beta and retinoic
acid-induced cancer cell death. Cell Death Differ. 14:327–337.
2007. View Article : Google Scholar
|
|
20
|
Hao H, Liu J, Liu G, et al: Depletion of
GRIM-19 accelerates hepatocellular carcinoma invasion via inducing
EMT and loss of contact inhibition. J Cell Physiol. 227:1212–1219.
2012. View Article : Google Scholar
|
|
21
|
Zhou T, Chao L, Rong G, Wang C, Ma R and
Wang X: Downregulation of GRIM-19 is associated with STAT3
overexpression in breast carcinomas. Hum Pathol. 44:1773–1779.
2013. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Bu X, Zhao C, Wang W and Zhang N: GRIM-19
inhibits the STAT3 signaling pathway and sensitizes gastric cancer
cells to radiation. Gene. 512:198–205. 2013. View Article : Google Scholar
|
|
23
|
Zhang L, Gao L, Li Y, et al: Effects of
plasmid-based Stat3-specific short hairpin RNA and GRIM-19 on PC-3
M tumor cell growth. Clin Cancer Res. 14:559–568. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Zhang Y, Hao H, Zhao S, et al:
Downregulation of GRIM-19 promotes growth and migration of human
glioma cells. Cancer Sci. 102:1991–1999. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Wang T, Yan XB, Zhao JJ, et al: Gene
associated with retinoid-interferon-induced mortality-19 suppresses
growth of lung adenocarcinoma tumor in vitro and in vivo. Lung
Cancer. 72:287–293. 2011. View Article : Google Scholar
|
|
26
|
Wang GM, Ren ZX, Wang PS, et al:
Plasmid-based Stat3-specific siRNA and GRIM-19 inhibit the growth
of thyroid cancer cells in vitro and in vivo. Oncol Rep.
32:573–580. 2014.PubMed/NCBI
|
|
27
|
Zhang J, Yang J, Roy SK, et al: The cell
death regulator GRIM-19 is an inhibitor of signal transducer and
activator of transcription 3. Proc Natl Acad Sci USA.
100:9342–9347. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Darnell JE Jr: STATs and gene regulation.
Science. 277:1630–1635. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Kalakonda S, Nallar SC, Lindner DJ, Hu J,
Reddy SP and Kalvakolanu DV: Tumor-suppressive activity of the cell
death activator GRIM-19 on a constitutively active signal
transducer and activator of transcription 3. Cancer Res.
67:6212–6220. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Lufei C, Ma J, Huang G, et al: GRIM-19, a
death-regulatory gene product, suppresses Stat3 activity via
functional interaction. EMBO J. 22:1325–1335. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Boyle ST and Kochetkova M: Breast cancer
stem cells and the immune system: Promotion, evasion and therapy. J
Mammary Gland Biol Neoplasia. 19:203–211. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Xie TX, Wei D, Liu M, et al: Stat3
activation regulates the expression of matrix metalloproteinase-2
and tumor invasion and metastasis. Oncogene. 23:3550–3560. 2004.
View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Farina AR, Tacconelli A, Vacca A, Maroder
M, Gulino A and Mackay AR: Transcriptional upregulation of matrix
metalloproteinase-9 expression during spontaneous epithelial to
neuroblast phenotype conversion by SK-N-SH neuroblastoma cells,
involved in enhanced invasivity, depends upon GT-box and nuclear
factor kappaB elements. Cell Growth Differ. 10:353–367.
1999.PubMed/NCBI
|
|
34
|
Bond M, Fabunmi RP, Baker AH and Newby AC:
Synergistic upregulation of metalloproteinase-9 by growth factors
and inflammatory cytokines: an absolute requirement for
transcription factor NF-kappa B. FEBS Lett. 435:29–34. 1998.
View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Niu G, Wright KL, Huang M, et al:
Constitutive Stat3 activity up-regulates VEGF expression and tumor
angiogenesis. Oncogene. 21:2000–2008. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Cho JY, Chung HC, Noh SH, Roh JK, Min JS
and Kim BS: High level of urokinase-type plasminogen activator is a
new prognostic marker in patients with gastric carcinoma. Cancer.
79:878–883. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Wei D, Le X, Zheng L, et al: Stat3
activation regulates the expression of vascular endothelial growth
factor and human pancreatic cancer angiogenesis and metastasis.
Oncogene. 22:319–329. 2003. View Article : Google Scholar : PubMed/NCBI
|
