|
1
|
Yao YM and Sheng ZY: Progress of basis
research on traumatic sepsis in China. Chin J Trauma. 19:9–12.
2003.
|
|
2
|
Koh IH, Menchaca-Diaz JL, Koh TH, Souza
RL, Shu CM, Rogerio VE and Liberatore AM: Microcirculatory
evaluation in sepsis: A difficult task. Shock. 34(Suppl 1): 27–33.
2010. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Aird WC: The role of the endothelium in
severe sepsis and multiple organ dysfunction syndrome. Blood.
101:3765–3777. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Lee WL and Liles WC: Endothelial
activation, dysfunction and permeability during severe infections.
Curr Opin Hematol. 18:191–196. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Lee WL and Slutsky AS: Sepsis and
endothelial permeability. N Engl J Med. 363:689–691. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Schouten M, Wiersinga WJ, Levi M and van
der Poll T: Inflammation, endothelium, and coagulation in sepsis. J
Leukoc Biol. 83:536–545. 2008. View Article : Google Scholar
|
|
7
|
Wang H, Yang H and Tracey KJ:
Extracellular role of HMGB1 in inflammation and sepsis. J Intern
Med. 255:320–331. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Wang H, Yang H, Czura CJ, Sama AE and
Tracey KJ: HMGB1 as a late mediator of lethal systemic
inflammation. Am J Respir Crit Care Med. 164:1768–1773. 2001.
View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Karlsson S, Pettilä V, Tenhunen J,
Laru-Sompa R, Hynninen M and Ruokonen E: HMGB1 as a predictor of
organ dysfunction and outcome in patients with severe sepsis.
Intensive Care Med. 34:1046–1053. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Kokkola R, Andersson A, Mullins G, Ostberg
T, Treutiger CJ, Arnold B, Nawroth P, Andersson U, Harris RA and
Harris HE: RAGE is the major receptor for the proinflammatory
activity of HMGB1 in rodent macrophages. Scand J Immunol. 61:1–9.
2005. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Mullins GE, Sunden-Cullberg J, Johansson
AS, Rouhiainen A, Erlandsson-Harris H, Yang H, Tracey KJ, Rauvala
H, Palmblad J, Andersson J and Treutiger CJ: Activation of human
umbilical vein endothelial cells leads to relocation and release of
high-mobility group box chromosomal protein 1. Scand J Immunol.
60:566–573. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Gill SE, Taneja R, Rohan M, Wang L and
Mehta S: Pulmonary microvascular albumin leak is associated with
endothelial cell death in murine sepsis-induced lung injury in
vivo. PloS One. 9:e885012014. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Hornburger MC, Mayer BA, Leonhardt S,
Willer EA, Zahler S, Beyerle A, Rajalingam K, Vollmar AM and Fürst
R: A novel role for inhibitor of apoptosis (IAP) proteins as
regulators of endothelial barrier function by mediating RhoA
activation. FASEB J. 28:1938–1946. 2014. View Article : Google Scholar
|
|
14
|
Aird WC: Endothelium as an organ system.
Crit Care Med. 32(5 Suppl): S271–S279. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Leclerc J, Pu Q, Corseaux D, Haddad E,
Decoene C, Bordet R, Six I, Jude B and Vallet B: A single endotoxin
injection in the rabbit causes prolonged blood vessel dysfunction
and a procoagulant state. Crit Care Med. 28:3672–3678. 2000.
View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Mutunga M, Fulton B, Bullock R, Batchelor
A, Gascoigne A, Gillespie JI and Baudouin SV: Circulating
endothelial cells in patients with septic shock. Am J Respir Crit
Care Med. 163:195–200. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
McKenzie JA and Ridley AJ: Roles of
Rho/ROCK and MLCK in TNF-alpha-induced changes in endothelial
morphology and permeability. J Cell Physiol. 213:221–228. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
18
|
van Nieuw Amerongen GP, van Delft S,
Vermeer MA, Collard JG and van Hinsbergh VW: Activation of RhoA by
thrombin in endothelial hyperpermeability: Role of Rho kinase and
protein tyrosine kinases. Circ Res. 87:335–340. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Zheng YJ, Zhou B, Song ZF, Li L, Wu J,
Zhang RY and Tang YQ: Study of Astragalus mongholicus
polysaccharides on endothelial cells permeability induced by HMGB1.
Carbohydr Polym. 92:934–941. 2013. View Article : Google Scholar
|
|
20
|
Dellinger RP, Levy MM, Rhodes A, Annane D,
Gerlach H, Opal SM, Sevransky JE, Sprung CL, Douglas IS, Jaeschke
R, et al: Surviving Sepsis Campaign: International guidelines for
management of severe sepsis and septic shock, 2012. Intensive Care
Med. 39:165–228. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Lagu T, Rothberg MB, Shieh MS, Pekow PS,
Steingrub JS and Lindenauer PK: Hospitalizations, costs, and
outcomes of severe sepsis in the United States 2003 to 2007. Crit
Care Med. 40:754–761. 2012. View Article : Google Scholar
|
|
22
|
Bilgin YM, van de Watering LM, Versteegh
MI, van Oers MH and Brand A: Effects of allogeneic leukocytes in
blood transfusions during cardiac surgery on inflammatory mediators
and postoperative complications. Crit Care Med. 38:546–552. 2010.
View Article : Google Scholar
|
|
23
|
Wang H, Bloom O, Zhang M, Vishnubhakat JM,
Ombrellino M, Che J, Frazier A, Yang H, Ivanova S, Borovikova L, et
al: HMG-1 as a late mediator of endotoxin lethality in mice.
Science. 285:248–251. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Bitto A, Barone M, David A, Polito F,
Familiari D, Monaco F, Giardina M, David T, Messina R, Noto A, et
al: High mobility group box-1 expression correlates with poor
outcome in lung injury patients. Pharmacol Res. 61:116–120. 2010.
View Article : Google Scholar
|
|
25
|
Fiuza C, Bustin M, Talwar S, Tropea M,
Gerstenberger E, Shelhamer JH and Suffredini AF:
Inflammation-promoting activity of HMGB1 on human microvascular
endothelial cells. Blood. 101:2652–2660. 2003. View Article : Google Scholar
|
|
26
|
Liu MW, Wang YH, Qian CY and Li H:
Xuebijing exerts protective effects on lung permeability leakage
and lung injury by upregulating Toll-interacting protein expression
in rats with sepsis. Int J Mol Med. 34:1492–1504. 2014.PubMed/NCBI
|
|
27
|
Bannerman DD and Goldblum SE: Direct
effects of endotoxin on the endothelium: Barrier function and
injury. Lab Invest. 79:1181–1199. 1999.PubMed/NCBI
|
|
28
|
Goldenberg NM, Steinberg BE, Slutsky AS
and Lee WL: Broken barriers: A new take on sepsis pathogenesis. Sci
Transl Med. 3:88ps252011.PubMed/NCBI
|
|
29
|
Jones TJ, Adapala RK, Geldenhuys WJ,
Bursley C, AbouAlaiwi WA, Nauli SM and Thodeti CK: Primary cilia
regulates the directional migration and barrier integrity of
endothelial cells through the modulation of hsp27 dependent actin
cytoskeletal organization. J Cell Physiol. 227:70–76. 2012.
View Article : Google Scholar
|
|
30
|
Corada M, Mariotti M, Thurston G, Smith K,
Kunkel R, Brockhaus M, Lampugnani MG, Martin-Padura I, Stoppacciaro
A, Ruco L, et al: Vascular endothelial-cadherin is an important
determinant of microvascular integrity in vivo. Proc Natl Acad Sci
USA. 96:9815–9820. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Ng CT, Fong LY, Sulaiman MR, Moklas MA,
Yong YK, Hakim MN and Ahmad Z: Interferon-gamma increases
endothelial permeability by causing activation of p38 MAP kinase
and actin cytoskeleton alteration. J Interferon Cytokine Res.
35:513–522. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Muradashvili N, Tyagi N, Tyagi R, Munjal C
and Lominadze D: Fibrinogen alters mouse brain endothelial cell
layer integrity affecting vascular endothelial cadherin. Biochem
Biophys Res Commun. 413:509–514. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Wesche-Soldato DE, Swan RZ, Chung CS and
Ayala A: The apoptotic pathway as a therapeutic target in sepsis.
Curr Drug Targets. 8:493–500. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Schmid FX, Vudattu N, Floerchinger B,
Hilker M, Eissner G, Hoenicka M, Holler E and Birnbaum DE:
Endothelial apoptosis and circulating endothelial cells after
bypass grafting with and without cardiopulmonary bypass. Eur J
Cardiothorac Surg. 29:496–500. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Fu H, Wang QS, Luo Q, Tan S, Su H, Tang
SL, Zhao ZL and Huang LP: Simvastatin inhibits apoptosis of
endothelial cells induced by sepsis through upregulating the
expression of Bcl-2 and downregulating Bax. World J Emerg Med.
4:291–297. 2014. View Article : Google Scholar
|
|
36
|
Kluz J, Kopeć W, Jakobsche-Policht U and
Adamiec R: Circulating endothelial cells, endothelial apoptosis and
soluble markers of endothelial dysfunction in patients with
systemic lupus erythematosus-related vasculitis. Int Angiol.
28:192–201. 2009.PubMed/NCBI
|
|
37
|
Xiao D and Zhang L: Upregulation of Bax
and Bcl-2 following prenatal cocaine exposure induces apoptosis in
fetal rat brain. Int J Med Sci. 5:295–302. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Liang Y, Li X, Zhang X, Li Z, Wang L, Sun
Y, Liu Z and Ma X: Elevated levels of plasma TNF-α are associated
with microvascular endothelial dysfunction in patients with sepsis
through activating the NF-κB and p38 mitogen-activated protein
kinase in endothelial cells. Shock. 41:275–281. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
39
|
Andersson U, Wang H, Palmblad K, Aveberger
AC, Bloom O, Erlandsson-Harris H, Janson A, Kokkola R, Zhang M,
Yang H and Tracey KJ: High mobility group 1 protein (HMG-1)
stimulates proinflammatory cytokine synthesis in human monocytes. J
Exp Med. 192:565–570. 2000. View Article : Google Scholar : PubMed/NCBI
|
