|
1
|
Marvar PJ, Thabet SR, Guzik TJ, Lob HE,
McCann LA, Weyand C, Gordon FJ and Harrison DG: Central and
peripheral mechanisms of T-lymphocyte activation and vascular
inflammation produced by angiotensin II-induced hypertension. Circ
Res. 107:263–270. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Jia LX, Qi GM, Liu O, Li TT, Yang M, Cui
W, Zhang WM, Qi YF and Du J: Inhibition of platelet activation by
clopidogrel prevents hypertension-induced cardiac inflammation and
fibrosis. Cardiovasc Drugs Ther. 27:521–530. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Mantovani A and Garlanda C:
Platelet-macrophage partnership in innate immunity and
inflammation. Nat Immunol. 14:768–770. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
van Gils JM, Zwaginga JJ and Hordijk PL:
Molecular and functional interactions among monocytes, platelets,
and endothelial cells and their relevance for cardiovascular
diseases. J Leukoc Biol. 85:195–204. 2009. View Article : Google Scholar
|
|
5
|
von Hundelshausen P and Weber C: Platelets
as immune cells: Bridging inflammation and cardiovascular disease.
Circ Res. 100:27–40. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
National Research Council (US) Institute
for Laboratory Animal Research: Guide for the Care and Use of
Laboratory Animals. National Academies Press; Washington, DC: pp.
85–23. 1996
|
|
7
|
Gresele P, Momi S, Berrettini M, Nenci GG,
Schwarz HP, Semeraro N and Colucci M: Activated human protein C
prevents thrombin-induced thromboembolism in mice. Evidence that
activated protein c reduces intravascular fibrin accumulation
through the inhibition of additional thrombin generation. J Clin
Invest. 101:667–676. 1998. View
Article : Google Scholar : PubMed/NCBI
|
|
8
|
Burger PC and Wagner DD: Platelet
P-selectin facilitates atherosclerotic lesion development. Blood.
101:2661–2666. 2003. View Article : Google Scholar
|
|
9
|
Huang XR, Chung AC, Yang F, Yue W, Deng C,
Lau CP, Tse HF and Lan HY: Smad3 mediates cardiac inflammation and
fibrosis in angiotensin II-induced hypertensive cardiac remodeling.
Hypertension. 55:1165–1171. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Livak KJ and Schmittgen TD: Analysis of
relative gene expression data using real-time quantitative PCR and
the 2(-Delta Delta C(T)) method. Methods. 25:402–408. 2001.
View Article : Google Scholar
|
|
11
|
Ma F, Li Y, Jia L, Han Y, Cheng J, Li H,
Qi Y and Du J: Macrophage-stimulated cardiac fibroblast production
of IL-6 is essential for TGF β/Smad activation and cardiac fibrosis
induced by angiotensin II. PLoS One. 7:e351442012. View Article : Google Scholar
|
|
12
|
Usher MG, Duan SZ, Ivaschenko CY, Frieler
RA, Berger S, Schütz G, Lumeng CN and Mortensen RM: Myeloid
mineralocorticoid receptor controls macrophage polarization and
cardiovascular hypertrophy and remodeling in mice. J Clin Invest.
120:3350–3364. 2010. View
Article : Google Scholar : PubMed/NCBI
|
|
13
|
Totani L and Evangelista V:
Platelet-leukocyte interactions in cardiovascular disease and
beyond. Arterioscler Thromb Vasc Biol. 30:2357–2361. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Honsho S, Nishikawa S, Amano K, Zen K,
Adachi Y, Kishita E, Matsui A, Katsume A, Yamaguchi S, Nishikawa K,
et al: Pressure-mediated hypertrophy and mechanical stretch induces
IL-1 release and subsequent IGF-1 generation to maintain
compensative hypertrophy by affecting Akt and JNK pathways. Circ
Res. 105:1149–1158. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Bujak M, Dobaczewski M, Chatila K, Mendoza
LH, Li N, Reddy A and Frangogiannis NG: Interleukin-1 receptor type
I signaling critically regulates infarct healing and cardiac
remodeling. Am J Pathol. 173:57–67. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Guo L, Sun G, Wang G, Ning W and Zhao K:
Soluble P-selectin promotes acute myocardial infarction onset but
not severity. Mol Med Rep. 11:2027–2033. 2015.
|
|
17
|
Spencer CG, Gurney D, Blann AD, Beevers DG
and Lip GY; ASCOT Steering Committee Anglo-Scandinavian Cardiac
Outcomes Trial: Von willebrand factor, soluble P-selectin, and
target organ damage in hypertension: A substudy of the
angloscandinavian cardiac outcomes trial (ASCOT). Hypertension.
40:61–66. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Piqueras L, Kubes P, Alvarez A, O'Connor
E, Issekutz AC, Esplugues JV and Sanz MJ: Angiotensin II induces
leukocyte-endothelial cell interactions in vivo via AT(1) and AT(2)
receptor-mediated P-selectin upregulation. Circulation.
102:2118–2123. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Kawai Y and Montgomery RR: Endothelial
cell processing of von Willebrand proteins. Ann N Y Acad Sci.
509:60–70. 1987. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Larsson PT, Schwieler JH and Wallén NH:
Platelet activation during angiotensin II infusion in healthy
volunteers. Blood Coagul Fibrinolysis. 11:61–69. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Kuebler WM: Selectins revisited: The
emerging role of platelets in inflammatory lung disease. J Clin
Invest. 116:3106–3108. 2006. View
Article : Google Scholar : PubMed/NCBI
|
|
22
|
Wang HB, Wang JT, Zhang L, Geng ZH, Xu WL,
Xu T, Huo Y, Zhu X, Plow EF, Chen M and Geng JG: P-selectin primes
leukocyte integrin activation during inflammation. Nat Immunol.
8:882–892. 2007. View
Article : Google Scholar : PubMed/NCBI
|
|
23
|
Pan L, Li Y, Jia L, Qin Y, Qi G, Cheng J,
Qi Y, Li H and Du J: Cathepsin S deficiency results in abnormal
accumulation of autophagosomes in macrophages and enhances Ang
II-induced cardiac inflammation. PLoS One. 7:e353152012. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Dai DF, Johnson SC, Villarin JJ, Chin MT,
Nieves-Cintrón M, Chen T, Marcinek DJ, Dorn GW II, Kang YJ, Prolla
TA, et al: Mitochondrial oxidative stress mediates angiotensin
II-induced cardiac hypertrophy and Galphaq overexpression-induced
heart failure. Circ Res. 108:837–846. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Izumiya Y, Kim S, Izumi Y, Yoshida K,
Yoshiyama M, Matsuzawa A, Ichijo H and Iwao H: Apoptosis
signal-regulating kinase 1 plays a pivotal role in angiotensin
II-induced cardiac hypertrophy and remodeling. Circ Res.
93:874–883. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Bujak M, Dobaczewski M, Chatila K, Mendoza
LH, Li N, Reddy A and Frangogiannis NG: Interleukin-1 receptor type
I signaling critically regulates infarct healing and cardiac
remodeling. Am J Pathol. 173:57–67. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Ferroni P, Guagnano MT, Falco A, Paoletti
V, Manigrasso MR, Michetti N, Santilli F, Guadagni F, Basili S and
Davì G: Association of low-grade inflammation and platelet
activation in patients with hypertension with microalbuminuria.
Clin Sci (Lond). 114:449–455. 2008. View Article : Google Scholar
|
|
28
|
Davì G, Gresele P, Violi F, Basili S,
Catalano M, Giammarresi C, Volpato R, Nenci GG, Ciabattoni G and
Patrono C: Diabetes mellitus, hypercholesterolemia, and
hypertension but not vascular disease per se are associated with
persistent platelet activation in vivo. Evidence derived from the
study of peripheral arterial disease. Circulation. 96:69–75. 1997.
View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Nadar SK, Blann AD, Kamath S, Beevers DG
and Lip GY: Platelet indexes in relation to target organ damage in
high-risk hypertensive patients: A substudy of the
anglo-scandinavian cardiac outcomes trial (ASCOT). J Am Coll
Cardiol. 44:415–422. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Liu Y, Gao XM, Fang L, Jennings NL, Su Y,
Q X, Samson AL, Kiriazis H, Wang XF, Shan L, et al: Novel role of
platelets in mediating inflammatory responses and ventricular
rupture or remodeling following myocardial infarction. Arterioscler
Thromb Vasc Biol. 31:834–841. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Hara T, Shimizu K, Ogawa F, Yanaba K,
Iwata Y, Muroi E, Takenaka M, Komura K, Hasegawa M, Fujimoto M and
Sato S: Platelets control leukocyte recruitment in a murine model
of cutaneous arthus reaction. Am J Pathol. 176:259–269. 2010.
View Article : Google Scholar :
|
|
32
|
Larsen E, Celi A, Gilbert GE, Furie BC,
Erban JK, Bonfanti R, Wagner DD and Furie B: PADGEM protein: A
receptor that mediates the interaction of activated platelets with
neutrophils and monocytes. Cell. 59:305–312. 1989. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Theilmeier G, Lenaerts T, Remacle C,
Collen D, Vermylen J and Hoylaerts MF: Circulating activated
platelets assist THP-1 monocytoid/endothelial cell interaction
under shear stress. Blood. 94:2725–2734. 1999.PubMed/NCBI
|
|
34
|
Mollmann H, Nef HM and Troidl C: Turning
the right screw: Targeting the interleukin-6 receptor to reduce
unfavourable tissue remodelling after myocardial infarction.
Cardiovasc Res. 87:395–396. 2010. View Article : Google Scholar
|
|
35
|
Staufenberger S, Jacobs M, Brandstatter K,
Hafner M, Regitz-Zagrosek V, Ertl G and Schorb W: Angiotensin II
type 1 receptor regulation and differential trophic effects on rat
cardiac myofibroblasts after acute myocardial infarction. J Cell
Physiol. 187:326–335. 2001. View
Article : Google Scholar : PubMed/NCBI
|
|
36
|
Lijnen PJ, Petrov VV and Fagard RH:
Angiotensin II-induced stimulation of collagen secretion and
production in cardiac fibroblasts is mediated via angiotensin II
subtype 1 receptors. J Renin Angiotensin Aldosterone Syst.
2:117–122. 2001.
|
|
37
|
Hafizi S, Wharton J, Morgan K, Allen SP,
Chester AH, Catravas JD, Polak JM and Yacoub MH: Expression of
functional angiotensin-converting enzyme and AT1 receptors in
cultured human cardiac fibroblasts. Circulation. 98:2553–2559.
1998. View Article : Google Scholar : PubMed/NCBI
|
