|
1
|
Gonzalez C, Sanz-Alfayate G, Agapito MT,
Gomez-Niño A, Rocher A and Obeso A: Significance of ROS in oxygen
sensing in cell systems with sensitivity to physiological hypoxia.
Respir Physiol Neurobiol. 132:17–41. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Baran CP, Zeigler MM, Tridandapani S and
Marsh CB: The role of ROS and RNS in regulating life and death of
blood monocytes. Curr Pharm Des. 10:855–866. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Irani K: Oxidant signaling in vascular
cell growth, death, and survival: A review of the roles of reactive
oxygen species in smooth muscle and endothelial cell mitogenic and
apoptotic signaling. Circ Res. 87:179–183. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Perez-Vizcaino F, Cogolludo A and Moreno
L: Reactive oxygen species signaling in pulmonary vascular smooth
muscle. Respir Physiol Neurobiol. 174:212–220. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Zorov DB, Juhaszova M and Sollott SJ:
Mitochondrial ROS-induced ROS release: An update and review.
Biochim Biophys Acta. 1757:509–517. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Zelko IN, Mariani TJ and Folz RJ:
Superoxide dismutase multigene family: A comparison of the CuZn-SOD
(SOD1), Mn-SOD (SOD2), and EC-SOD (SOD3) gene structures,
evolution, and expression. Free Radic Biol Med. 33:337–349. 2002.
View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Wilcox CS: Reactive oxygen species: Roles
in blood pressure and kidney function. Curr Hypertens Rep.
4:160–166. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Chen TJ, Jeng JY, Lin CW, Wu CY and Chen
YC: Quercetin inhibition of ROS-dependent and -independent
apoptosis in rat glioma C6 cells. Toxicology. 223:113–126. 2006.
View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Dasmahapatra G, Rahmani M, Dent P and
Grant S: The tyrphostin adaphostin interacts synergistically with
proteasome inhibitors to induce apoptosis in human leukemia cells
through a reactive oxygen species (ROS)-dependent mechanism. Blood.
107:232–240. 2006. View Article : Google Scholar
|
|
10
|
Wallach-Dayan SB, Izbicki G, Cohen PY,
Gerstl-Golan R, Fine A and Breuer R: Bleomycin initiates apoptosis
of lung epithelial cells by ROS but not by Fas/FasL pathway. Am J
Physiol Lung Cell Mol Physiol. 290:L790–L796. 2006. View Article : Google Scholar
|
|
11
|
Saeki K, Hayakawa S, Isemura M and Miyase
T: Importance of a pyrogallol-type structure in catechin compounds
for apoptosis-inducing activity. Phytochemistry. 53:391–394. 2000.
View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Yamada J, Yoshimura S, Yamakawa H, Sawada
M, Nakagawa M, Hara S, Kaku Y, Iwama T, Naganawa T, Banno Y, et al:
Cell permeable ROS scavengers, Tiron and Tempol, rescue PC12 cell
death caused by pyrogallol or hypoxia/reoxygenation. Neurosci Res.
45:1–8. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Kim SW, Han YW, Lee ST, Jeong HJ, Kim SH,
Kim IH, Lee SO, Kim DG, Kim SH, Kim SZ and Park WH: A superoxide
anion generator, pyrogallol, inhibits the growth of HeLa cells via
cell cycle arrest and apoptosis. Mol Carcinog. 47:114–125. 2008.
View Article : Google Scholar
|
|
14
|
Han YH, Kim SZ, Kim SH and Park WH:
Pyrogallol inhibits the growth of lung cancer Calu-6 cells via
caspase-dependent apoptosis. Chem Biol Interact. 177:107–114. 2009.
View Article : Google Scholar
|
|
15
|
Han YH, Kim SH, Kim SZ and Park WH:
Pyrogallol inhibits the growth of human pulmonary adenocarcinoma
A549 cells by arresting cell cycle and triggering apoptosis. J
Biochem Mol Toxicol. 23:36–42. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Park WH, Park MN, Han YH and Kim SW:
Pyrogallol inhibits the growth of gastric cancer SNU-484 cells via
induction of apoptosis. Int J Mol Med. 22:263–268. 2008.PubMed/NCBI
|
|
17
|
You BR and Park WH: Gallic acid-induced
lung cancer cell death is related to glutathione depletion as well
as reactive oxygen species increase. Toxicol In Vitro.
24:1356–1362. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
You BR, Kim SH and Park WH: Reactive
oxygen species, glutathione, and thioredoxin influence suberoyl
bishydroxamic acid-induced apoptosis in A549 lung cancer cells.
Tumour Biol. 36:3429–3439. 2015. View Article : Google Scholar
|
|
19
|
You BR, Shin HR, Han BR and Park WH: PX-12
induces apoptosis in Calu-6 cells in an oxidative stress-dependent
manner. Tumour Biol. 36:2087–2095. 2015. View Article : Google Scholar
|
|
20
|
You BR and Park WH: Arsenic trioxide
induces human pulmonary fibroblast cell death via increasing ROS
levels and GSH depletion. Oncol Rep. 28:749–757. 2012.PubMed/NCBI
|
|
21
|
Li PF, Dietz R and von Harsdorf R:
Reactive oxygen species induce apoptosis of vascular smooth muscle
cell. FEBS Lett. 404:249–252. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Johnson TM, Yu ZX, Ferrans VJ, Lowenstein
RA and Finkel T: Reactive oxygen species are downstream mediators
of p53-dependent apoptosis. Proc Natl Acad Sci USA. 93:11848–11852.
1996. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Rao GN and Berk BC: Active oxygen species
stimulate vascular smooth muscle cell growth and proto-oncogene
expression. Circ Res. 70:593–599. 1992. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Rao GN, Lassègue B, Griendling KK and
Alexander RW: Hydrogen peroxide stimulates transcription of c-jun
in vascular smooth muscle cells: Role of arachidonic acid.
Oncogene. 8:2759–2764. 1993.PubMed/NCBI
|
|
25
|
Brown MR, Miller FJ Jr, Li WG, Ellingson
AN, Mozena JD, Chatterjee P, Engelhardt JF, Zwacka RM, Oberley LW,
Fang X, et al: Overexpression of human catalase inhibits
proliferation and promotes apoptosis in vascular smooth muscle
cells. Circ Res. 85:524–533. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Han YH, Moon HJ, You BR, Kim SZ, Kim SH
and Park WH: Pyrogallol-induced endothelial cell death is related
to GSH depletion rather than ROS level changes. Oncol Rep.
23:287–292. 2010.
|
|
27
|
Estrela JM, Ortega A and Obrador E:
Glutathione in cancer biology and therapy. Crit Rev Clin Lab Sci.
43:143–181. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Han YH, Kim SZ, Kim SH and Park WH:
Induction of apoptosis in arsenic trioxide-treated lung cancer A549
cells by buthionine sulfoximine. Mol Cells. 26:158–164.
2008.PubMed/NCBI
|
|
29
|
Han YH, Kim SZ, Kim SH and Park WH:
Enhancement of arsenic trioxide-induced apoptosis in HeLa cells by
diethyldithiocarbamate or buthionine sulfoximine. Int J Oncol.
33:205–213. 2008.PubMed/NCBI
|
|
30
|
Wu XX, Ogawa O and Kakehi Y: Enhancement
of arsenic trioxide-induced apoptosis in renal cell carcinoma cells
by L-buthionine sulfoximine. Int J Oncol. 24:1489–1497.
2004.PubMed/NCBI
|
