|
1
|
Li HY, Ruan YW, Ren CR, Cui Q and So KF:
Mechanisms of secondary degeneration after partial optic nerve
transection. Neural Regen Res. 9:565–574. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Schwartz M: Optic nerve crush: Protection
and regeneration. Brain Res Bull. 62:467–471. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Xu F, Huang H, Wu Y, Lu L, Jiang L, Chen
L, Zeng S, Li L and Li M: Upregulation of Gem relates to retinal
ganglion cells apoptosis after optic nerve crush in adult rats. J
Mol Histol. 45:565–571. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Wu Y, Xu F, Huang H, Chen L, Wen M, Jiang
L, Lu L, Li L, Song D, Zeng S, et al: Up-regulation of SKIP relates
to retinal ganglion cells apoptosis after optic nerve crush in
vivo. J Mol Histol. 45:715–721. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Suzuki Y, Demoliere C, Kitamura D,
Takeshita H, Deuschle U and Watanabe T: HAX-1, a novel
intracellular protein, localized on mitochondria, directly
associates with HS1, a substrate of Src family tyrosine kinases. J
Immunol. 158:2736–2744. 1997.PubMed/NCBI
|
|
6
|
Simmen T: Hax-1: A regulator of calcium
signaling and apoptosis progression with multiple roles in human
disease. Expert Opin Ther Targets. 15:741–751. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Yap SV, Koontz JM and
Kontrogianni-Konstantopoulos A: HAX-1: A family of apoptotic
regulators in health and disease. J Cell Physiol. 226:2752–2761.
2011. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Fadeel B and Grzybowska E: HAX-1: A
multifunctional protein with emerging roles in human disease.
Biochim Biophys Acta. 1790:1139–1148. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Hu J, Mu C and Hao J: Cerebral ischemia
reduces expression of Hs1-associated protein X-1 (Hax-1) in mouse
brain. Neurosci Lett. 534:338–343. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Rami A, Kim M, Niquet J and Langhagen A:
Alterations in the expression of the anti-apoptotic factor HAX-1
upon seizures-induced hippocampal injury in the neonatal rat brain.
Neurochem Res. 37:116–125. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Shi W, Zhao W, Shen A, Shao B, Wu X, Yang
J, Ni L, Wu Q and Chen J: Traumatic brain injury induces an
up-regulation of Hs1-associated protein X-1 (Hax-1) in rat brain
cortex. Neurochem Res. 36:375–382. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Suzuki H, Oku H, Horie T, Morishita S,
Tonari M, Oku K, Okubo A, Kida T, Mimura M, Fukumoto M, et al:
Changes in expression of aquaporin-4 and aquaporin-9 in optic nerve
after crushing in rats. PLoS One. 9:e1146942014. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Mac Nair CE, Fernandes KA, Schlamp CL,
Libby RT and Nickells RW: Tumor necrosis factor alpha has an early
protective effect on retinal ganglion cells after optic nerve
crush. J Neuroinflammation. 11:1942014. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Yang L, Miao L, Liang F, Huang H, Teng X,
Li S, Nuriddinov J, Selzer ME and Hu Y: The mTORC1 effectors S6K1
and 4E-BP play different roles in CNS axon regeneration. Nat
Commun. 5:54162014. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Huang SP and Tsai RK: Efficacy of
granulocyte-colony stimulating factor treatment in a rat model of
anterior ischemic optic neuropathy. Neural Regen Res. 9:1502–1505.
2014. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Morgan-Warren PJ, Berry M, Ahmed Z, Scott
RA and Logan A: Exploiting mTOR signaling: A novel translatable
treatment strategy for traumatic optic neuropathy? Invest
Ophthalmol Vis Sci. 54:6903–6916. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Watanabe M: Regeneration of optic nerve
fibers of adult mammals. Dev Growth Differ. 52:567–576. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Schmitt HM, Pelzel HR, Schlamp CL and
Nickells RW: Histone deacetylase 3 (HDAC3) plays an important role
in retinal ganglion cell death after acute optic nerve injury. Mol
Neurodegener. 9:392014. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Shibeeb O, Wood JP, Casson RJ and Chidlow
G: Effects of a conventional photocoagulator and a 3-ns pulse laser
on preconditioning responses and retinal ganglion cell survival
after optic nerve crush. Exp Eye Res. 127:77–90. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Chen M, Xiang Z and Cai J: The
anti-apoptotic and neuro-protective effects of human umbilical cord
blood mesenchymal stem cells (hUCB-MSCs) on acute optic nerve
injury is transient. Brain Res. 1532:63–75. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Chao JR, Parganas E, Boyd K, Hong CY,
Opferman JT and Ihle JN: Hax1-mediated processing of HtrA2 by Parl
allows survival of lymphocytes and neurons. Nature. 452:98–102.
2008. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Han Y, Chen YS, Liu Z, Bodyak N, Rigor D,
Bisping E, Pu WT and Kang PM: Overexpression of HAX-1 protects
cardiac myocytes from apoptosis through caspase-9 inhibition. Circ
Res. 99:415–423. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Cilenti L, Soundarapandian MM, Kyriazis
GA, Stratico V, Singh S, Gupta S, Bonventre JV, Alnemri ES and
Zervos AS: Regulation of HAX-1 anti-apoptotic protein by Omi/HtrA2
protease during cell death. J Biol Chem. 279:50295–50301. 2004.
View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Wang Y, Zhang H, Liu Y, Li P, Cao Z and
Cao Y: Erythropoietin (EPO) protects against high glucose-induced
apoptosis in retinal ganglional cells. Cell Biochem Biophys.
71:749–755. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Cao Y, Li X, Shi P, Wang LX and Sui ZG:
Effects of L-carnitine on high glucose-induced oxidative stress in
retinal ganglion cells. Pharmacology. 94:123–130. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Rathnasamy G, Sivakumar V, Rangarajan P,
Foulds WS, Ling EA and Kaur C: NF-kB-mediated nitric oxide
production and activation of caspase-3 cause retinal ganglion cell
death in the hypoxic neonatal retina. Invest Ophthalmol Vis Sci.
55:5878–5889. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Lee AY, Lee Y, Park YK, Bae KH, Cho S, Lee
DH, Park BC, Kang S and Park SG: HS 1-associated protein X-1 is
cleaved by caspase-3 during apoptosis. Mol Cells. 25:86–90.
2008.PubMed/NCBI
|
|
28
|
Yedavalli VS, Shih HM, Chiang YP, Lu CY,
Chang LY, Chen MY, Chuang CY, Dayton AI, Jeang KT and Huang LM:
Human immunodeficiency virus type 1 Vpr interacts with
antiapoptotic mitochondrial protein HAX-1. J Virol. 79:13735–13746.
2005. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Li B, Hu Q, Wang H, Man N, Ren H, Wen L,
Nukina N, Fei E and Wang G: Omi/HtrA2 is a positive regulator of
autophagy that facilitates the degradation of mutant proteins
involved in neurodegenerative diseases. Cell Death Differ.
17:1773–1784. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Zurawa-Janicka D, Skorko-Glonek J and
Lipinska B: HtrA proteins as targets in therapy of cancer and other
diseases. Expert Opin Ther Targets. 14:665–679. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Bhuiyan MS and Fukunaga K: Mitochondrial
serine protease HtrA2/Omi as a potential therapeutic target. Curr
Drug Targets. 10:372–383. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Sun H, Li L, Zhou F, Zhu L, Ke K, Tan X,
Xu W, Rui Y, Zheng H, Zhou Z and Yang H: The member of high
temperature requirement family HtrA2 participates in neuronal
apoptosis after intracerebral hemorrhage in adult rats. J Mol
Histol. 44:369–379. 2013. View Article : Google Scholar : PubMed/NCBI
|
