|
1
|
Ho K, Nichols CG, Lederer WJ, Lytton J,
Vassilev PM, Kanazirska MV and Hebert SC: Cloning and expression of
an inwardly rectifying ATP-regulated potassium channel. Nature.
362:31–38. 1993. View
Article : Google Scholar : PubMed/NCBI
|
|
2
|
Lu M, Wang T, Yan Q, Yang X, Dong K,
Knepper MA, Wang W, Giebisch G, Shull GE and Hebert SC: Absence of
small conductance K+ channel (SK) activity in apical membranes of
thick ascending limb and cortical collecting duct in ROMK
(Bartter's) knockout mice. J Biol Chem. 277:37881–37887. 2002.
View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Giebisch G: Renal potassium transport:
Mechanisms and regulation. Am J Physiol. 274:F817–F833.
1998.PubMed/NCBI
|
|
4
|
Palmer LG: Potassium secretion and the
regulation of distal nephron K channels. Am J Physiol.
277:F821–F825. 1999.PubMed/NCBI
|
|
5
|
Wang W, Sackin H and Giebisch G: Renal
potassium channels and their regulation. Annu Rev Physiol.
54:81–96. 1992. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Nakamura T and Mizuno S: The discovery of
hepatocyte growth factor (HGF) and its significance for cell
biology, life sciences and clinical medicine. Proc Jpn Acad Ser B
Phys Biol Sci. 86:588–610. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Mizuno S and Nakamura T: Prevention of
neutrophil extravasation by hepatocyte growth factor leads to
attenuations of tubular apoptosis and renal dysfunction in mouse
ischemic kidneys. Am J Pathol. 166:1895–1905. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Ohnishi H, Mizuno S and Nakamura T:
Inhibition of tubular cell proliferation by neutralizing endogenous
HGF leads to renal hypoxia and bone marrow-derived cell engraftment
in acute renal failure. Am J Physiol Renal Physiol. 294:F326–F335.
2008. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Kato T, Funakoshi H, Kadoyama K, Noma S,
Kanai M, Ohya-Shimada W, Mizuno S, Doe N, Taniguchi T and Nakamura
T: Hepatocyte growth factor overexpression in the nervous system
enhances learning and memory performance in mice. J Neurosci Res.
90:1743–1755. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
MacGregor GG, Xu JZ, McNicholas CM,
Giebisch G and Hebert SC: Partially active channels produced by PKA
site mutation of the cloned renal K+ channel, ROMK2 (kir1.2). Am J
Physiol. 275:F415–F422. 1998.PubMed/NCBI
|
|
11
|
Yoo D, Kim BY, Campo C, Nance L, King A,
Maouyo D and Welling PA: Cell surface expression of the ROMK (Kir
1.1) channel is regulated by the aldosterone-induced kinase, SGK-1,
and protein kinase A. J Biol Chem. 278:23066–23075. 2003.
View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Lang F and Cohen P: Regulation and
physiological roles of serum- and glucocorticoid-induced protein
kinase isoforms. Sci STKE. 2001:re172001.PubMed/NCBI
|
|
13
|
Náray-Fejes-Tóth A, Canessa C, Cleaveland
ES, Aldrich G and Fejes-Tóth G: SGK is an aldosterone-induced
kinase in the renal collecting duct. Effects on epithelial na+
channels. J Biol Chem. 274:16973–16978. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Shelly C and Herrera R: Activation of SGK1
by HGF, Rac1 and integrin-mediated cell adhesion in MDCK cells:
PI-3K-dependent and -independent pathways. J Cell Sci.
115:1985–1993. 2002.PubMed/NCBI
|
|
15
|
Bosch X, Poch E and Grau JM:
Rhabdomyolysis and acute kidney injury. N Engl J Med. 361:62–72.
2009. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Ukai T: The Great Hanshin-Awaji Earthquake
and the problems with emergency medical care. Ren Fail. 19:633–645.
1997. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Nagano T, Mori-Kudo I, Tsuchida A,
Kawamura T, Taiji M and Noguchi H: Ameliorative effect of
hepatocyte growth factor on glycerol-induced acute renal failure
with acute tubular necrosis. Nephron. 91:730–738. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Ishibashi K, Sasaki S, Sakamoto H,
Nakamura Y, Hata T, Nakamura T and Marumo F: Hepatocyte growth
factor is a paracrine factor for renal epithelial cells:
Stimulation of DNA synthesis and NA,K-ATPase activity. Biochem
Biophys Res Commun. 182:960–965. 1992. View Article : Google Scholar : PubMed/NCBI
|
