|
1
|
Krishna SG, Kruger AJ, Patel N, Hinton A,
Yadav D and Conwell DL: Cholecystectomy during index admission for
acute biliary pancreatitis lowers 30-day readmission rates.
Pancreas. 47:996–1002. 2018. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Pandol SJ, Saluja AK, Imrie CW and Banks
PA: Acute pancreatitis: Bench to the bedside. Gastroenterology.
132:1127–1151. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Gao B, Wang D, Sun W, Meng X, Zhang W and
Xue D: Differentially expressed microRNA identification and target
gene function analysis in starvation-induced autophagy of AR42J
pancreatic acinar cells. Mol Med Rep. 14:590–598. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Dawra R, Sah RP, Dudeja V, Rishi L,
Talukdar R, Garg P and Saluja AK: Intra-acinar trypsinogen
activation mediates early stages of pancreatic injury but not
inflammation in mice with acute pancreatitis. Gastroenterology.
141:2210–2217.e2. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Sato H, Siow RC, Bartlett S, Taketani S,
Ishii T, Bannai S and Mann GE: Expression of stress proteins heme
oxygenase-1 and −2 in acute pancreatitis and pancreatic islet
betaTC3 and acinar AR42J cells. FEBS Lett. 405:219–223. 1997.
View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Ma B, Wu L, Lu M, Gao B, Qiao X, Sun B,
Xue D and Zhang W: Differentially expressed kinase genes associated
with trypsinogen activation in rat pancreatic acinar cells treated
with taurolithocholic acid 3-sulfate. Mol Med Rep. 7:1591–1596.
2013. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Yang Z, Yang W, Lu M, Li Z, Qiao X, Sun B,
Zhang W and Xue D: Role of the c-Jun N-terminal kinase signaling
pathway in the activation of trypsinogen in rat pancreatic acinar
cells. Int J Mol Med. 41:1119–1126. 2018. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Zhao Q, Wei Y, Pandol SJ, Li L and
Habtezion A: STING signaling promotes inflammation in experimental
acute pancreatitis. Gastroenterology. 154:1822–1835.e2. 2018.
View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Liu Y, Chen XD, Yu J, Chi JL, Long FW,
Yang HW, Chen KL, Lv ZY, Zhou B, Peng ZH, et al: Deletion Of XIAP
reduces the severity of acute pancreatitis via regulation of cell
death and nuclear factor-κB activity. Cell Death Dis. 8:e26852017.
View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Kowalik AS, Johnson CL, Chadi SA, Weston
JY, Fazio EN and Pin CL: Mice lacking the transcription factor
Mist1 exhibit an altered stress response and increased sensitivity
to caerulein-induced pancreatitis. Am J Physiol Gastrointest Liver
Physiol. 292:G1123–G1132. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Sendler M, Weiss FU, Golchert J, Homuth G,
van den Brandt C, Mahajan UM, Partecke LI, Döring P, Gukovsky I,
Gukovskaya AS, et al: Cathepsin B-mediated activation of
trypsinogen in endocytosing macrophages increases severity of
pancreatitis in mice. Gastroenterology. 154:704–718.e10. 2018.
View Article : Google Scholar : PubMed/NCBI
|
|
12
|
An F, Zhan Q, Xia M, Jiang L, Lu G, Huang
M, Guo J and Liu S: From moderately severe to severe
hypertriglyceridemia induced acute pancreatitis: Circulating miRNAs
play role as potential biomarkers. PLoS One. 9:e1110582014.
View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Gashler A and Sukhatme VP: Early growth
response protein 1 (Egr-1): prototype of a zinc-finger family of
transcription factors. Prog Nucleic Acid Res Mol Biol. 50:191–224.
1995. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Kaufmann A, Rössler OG and Thiel G:
Expression of the transcription factor Egr-1 in pancreatic acinar
cells following stimulation of cholecystokinin or Gαq-coupled
designer receptors. Cell Physiol Biochem. 33:1411–1425. 2014.
View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Zhan X, Wan J, Zhang G, Song L, Gui F,
Zhang Y, Li Y, Guo J, Dawra RK, Saluja AK, et al: Elevated
intracellular trypsin exacerbates acute pancreatitis and chronic
pancreatitis in mice. Am J Physiol Gastrointest Liver Physiol.
316:G816–G825. 2019. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Sandoval J, Pereda J, Pérez S, Finamor I,
Vallet-Sánchez A, Rodríguez JL, Franco L, Sastre J and López-Rodas
G: Epigenetic regulation of early- and late-response genes in acute
pancreatitis. J Immunol. 197:4137–4150. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Bartel DP: MicroRNAs: Target recognition
and regulatory functions. Cell. 136:215–233. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Zhang J, Ning X, Cui W, Bi M, Zhang D and
Zhang J: Transforming growth factor (TGF)-β-induced microRNA-216a
promotes acute pancreatitis via Akt and TGF-β pathway in mice. Dig
Dis Sci. 60:127–135. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Tian R, Wang RL, Xie H, Jin W and Yu KL:
Overexpressed miRNA-155 dysregulates intestinal epithelial apical
junctional complex in severe acute pancreatitis. World J
Gastroenterol. 19:8282–8291. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Ambros V: MicroRNAs and developmental
timing. Curr Opin Genet Dev. 21:511–517. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Gerasimenko JV, Flowerdew SE, Voronina SG,
Sukhomlin TK, Tepikin AV, Petersen OH and Gerasimenko OV: Bile
acids induce Ca2+ release from both the endoplasmic
reticulum and acidic intracellular calcium stores through
activation of inositol trisphosphate receptors and ryanodine
receptors. J Biol Chem. 281:40154–40163. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Voronina SG, Barrow SL, Gerasimenko OV,
Petersen OH and Tepikin AV: Effects of secretagogues and bile acids
on mitochondrial membrane potential of pancreatic acinar cells:
Comparison of different modes of evaluating DeltaPsim. J Biol Chem.
279:27327–27338. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Livak KJ and Schmittgen TD: Analysis of
relative gene expression data using real-time quantitative PCR and
the 2(-Delta Delta C(T)) method. Methods. 25:402–408. 2001.
View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Song Z, Huang Y, Liu C, Lu M, Li Z, Sun B,
Zhang W and Xue D: miR-352 participates in the regulation of
trypsinogen activation in pancreatic acinar cells by influencing
the function of autophagic lysosomes. Oncotarget. 9:10868–10879.
2018. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Sherwood MW, Prior IA, Voronina SG, Barrow
SL, Woodsmith JD, Gerasimenko OV, Petersen OH and Tepikin AV:
Activation of trypsinogen in large endocytic vacuoles of pancreatic
acinar cells. Proc Natl Acad Sci USA. 104:5674–5679. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Vaz J, Akbarshahi H and Andersson R:
Controversial role of toll-like receptors in acute pancreatitis.
World J Gastroenterol. 19:616–630. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Kylänpää L, Rakonczay Z Jr and O'Reilly
DA: The clinical course of acute pancreatitis and the inflammatory
mediators that drive it. Int J Inflam. 2012:3606852012. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Ohmuraya M and Yamamura K: Autophagy and
acute pancreatitis: A novel autophagy theory for trypsinogen
activation. Autophagy. 4:1060–1062. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Halangk W, Lerch MM, Brandt-Nedelev B,
Roth W, Ruthenbuerger M, Reinheckel T, Domschke W, Lippert H,
Peters C and Deussing J: Role of cathepsin B in intracellular
trypsinogen activation and the onset of acute pancreatitis. J Clin
Invest. 106:773–781. 2000. View
Article : Google Scholar : PubMed/NCBI
|
|
30
|
Chen WD, Zhang JL, Wang XY, Hu ZW and Qian
YB: The JAK2/STAT3 signaling pathway is required for inflammation
and cell death induced by cerulein in AR42J cells. Eur Rev Med
Pharmacol Sci. 23:1770–1777. 2019.PubMed/NCBI
|
|
31
|
Meng S, Wang H, Xue D and Zhang W:
Screening and validation of differentially expressed extracellular
miRNAs in acute pancreatitis. Mol Med Rep. 16:6412–6418. 2017.
View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Sukhatme VP, Cao XM, Chang LC, Tsai-Morris
CH, Stamenkovich D, Ferreira PC, Cohen DR, Edwards SA, Shows TB,
Curran T, et al: A zinc finger-encoding gene coregulated with c-fos
during growth and differentiation, and after cellular
depolarization. Cell. 53:37–43. 1988. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Yan L, Wang Y, Liang J, Liu Z, Sun X and
Cai K: MiR-301b promotes the proliferation, mobility, and
epithelial-to-mesenchymal transition of bladder cancer cells by
targeting EGR1. Biochem Cell Biol. 95:571–577. 2017. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Yan SF, Fujita T, Lu J, Okada K, Shan Zou
Y, Mackman N, Pinsky DJ and Stern DM: Egr-1, a master switch
coordinating upregulation of divergent gene families underlying
ischemic stress. Nat Med. 6:1355–1361. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Thiel G and Cibelli G: Regulation of life
and death by the zinc finger transcription factor Egr-1. J Cell
Physiol. 193:287–292. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Alagappan D, Balan M, Jiang Y, Cohen RB,
Kotenko SV and Levison SW: Egr-1 is a critical regulator of
EGF-receptor-mediated expansion of subventricular zone neural stem
cells and progenitors during recovery from hypoxia-hypoglycemia.
ASN Neuro. 5:183–193. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Ji B, Chen XQ, Misek DE, Kuick R, Hanash
S, Ernst S, Najarian R and Logsdon CD: Pancreatic gene expression
during the initiation of acute pancreatitis: identification of
EGR-1 as a key regulator. Physiol Genomics. 14:59–72. 2003.
View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Wan H, Yuan Y, Liu J and Chen G:
Pioglitazone, a PPAR-γ activator, attenuates the severity of
cerulein-induced acute pancreatitis by modulating early growth
response-1 transcription factor. Transl Res. 160:153–161. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
39
|
Gong LB, He L, Liu Y, Chen XQ and Jiang B:
Expression of early growth response factor-1 in rats with
cerulein-induced acute pancreatitis and its significance. World J
Gastroenterol. 11:5022–5024. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
40
|
Russo MW, Sevetson BR and Milbrandt J:
Identification of NAB1, a repressor of NGFI-A- and Krox20-mediated
transcription. Proc Natl Acad Sci USA. 92:6873–6877. 1995.
View Article : Google Scholar : PubMed/NCBI
|
|
41
|
Svaren J, Sevetson BR, Apel ED, Zimonjic
DB, Popescu NC and Milbrandt J: NAB2, a corepressor of NGFI-A
(Egr-1) and Krox20, is induced by proliferative and differentiative
stimuli. Mol Cell Biol. 16:3545–3553. 1996. View Article : Google Scholar : PubMed/NCBI
|
|
42
|
Qu Z, Wolfraim LA, Svaren J, Ehrengruber
MU, Davidson N and Milbrandt J: The transcriptional corepressor
NAB2 inhibits NGF-induced differentiation of PC12 cells. J Cell
Biol. 142:1075–1082. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
43
|
Zhang G, Li S, Lu J, Ge Y, Wang Q, Ma G,
Zhao Q, Wu D, Gong W, Du M, et al: LncRNA MT1JP functions as a
ceRNA in regulating FBXW7 through competitively binding to
miR-92a-3p in gastric cancer. Mol Cancer. 17:872018. View Article : Google Scholar : PubMed/NCBI
|
|
44
|
Setyowati Karolina D, Sepramaniam S, Tan
HZ, Armugam A and Jeyaseelan K: miR-25 and miR-92a regulate insulin
I biosynthesis in rats. RNA Biol. 10:1365–1378. 2013. View Article : Google Scholar : PubMed/NCBI
|
