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Molecular Medicine Reports
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Print ISSN: 1791-2997 Online ISSN: 1791-3004
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February-2022 Volume 25 Issue 2

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International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

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International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

Molecular Medicine Reports

Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

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Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

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Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

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Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

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International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

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Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

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International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

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Correction Open Access

[Corrigendum] Silencing of RIPK4 inhibits epithelial‑mesenchymal transition by inactivating the Wnt/β‑catenin signaling pathway in osteosarcoma

  • Authors:
    • Zhigang Yi
    • Yanchuan Pu
    • Ruoyan Gou
    • Yonggang Chen
    • Xiaojun Ren
    • Wenzhong Liu
    • Ping Dong

  • View Affiliations / Copyright

    Affiliations: Department of Pediatric Orthopedics and Pediatrics, Second Hospital of Lanzhou University, Lanzhou, Gansu 730000, P.R. China, Department of Orthopedics, Wuwei City People's Hospital, Wuwei, Gansu 733000, P.R. China
    Copyright: © Yi et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
  • Article Number: 69
    |
    Published online on: January 4, 2022
       https://doi.org/10.3892/mmr.2022.12585
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Mol Med Rep 21: [Related article:] 1154-1162, 2020; DOI: 10.3892/mmr.2020.10939

Subsequently to the publication of the above article, the authors have realized that the same western blotting data shown for the vimentin bands for the U2OS cell line in Fig. 3B on p. 1159 had inadvertently been re-used for the β-catenin bands for the U2OS cell line in Fig. 5A on p. 1161.

Figure 5.

Silencing of RIPK4 suppresses the inactivation of the Wnt/β-catenin signaling pathway. (A) Western blot analysis revealed that RIPK4 silencing decreased total and nuclear β-catenin expression in MG-63 and U2OS cells. β-actin was used as the loading control for total expression. Lamin B1 was used as a loading control in the nucleus. (B) Immunofluorescent staining revealed that RIPK4 silencing reduced the levels of β-catenin. The targeted proteins were stained green and the nuclei were stained with DAPI (blue; magnification, ×200). RIPK4, receptor interacting protein kinase 4; DAPI, 4,6-diamidino-2-phenylindole; NC, negative control; siRNA, small interfering RNA.

The authors have re-examined their original data, and have realized that Fig. 5 was assembled incorrectly. The corrected version of Fig. 5, showing the correct β-catenin bands for the U2OS cell line, is shown opposite. Note that this error did not quantitatively affect either the results or the overall conclusions reported in this paper. The authors are grateful to the Editor of Molecular Medicine Reports for allowing them this opportunity to publish a Corrigendum, and they apologize to the readership for any inconvenience caused.

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Copy and paste a formatted citation
Spandidos Publications style
Yi Z, Pu Y, Gou R, Chen Y, Ren X, Liu W and Dong P: [Corrigendum] Silencing of RIPK4 inhibits epithelial‑mesenchymal transition by inactivating the Wnt/β‑catenin signaling pathway in osteosarcoma. Mol Med Rep 25: 69, 2022.
APA
Yi, Z., Pu, Y., Gou, R., Chen, Y., Ren, X., Liu, W., & Dong, P. (2022). [Corrigendum] Silencing of RIPK4 inhibits epithelial‑mesenchymal transition by inactivating the Wnt/β‑catenin signaling pathway in osteosarcoma. Molecular Medicine Reports, 25, 69. https://doi.org/10.3892/mmr.2022.12585
MLA
Yi, Z., Pu, Y., Gou, R., Chen, Y., Ren, X., Liu, W., Dong, P."[Corrigendum] Silencing of RIPK4 inhibits epithelial‑mesenchymal transition by inactivating the Wnt/β‑catenin signaling pathway in osteosarcoma". Molecular Medicine Reports 25.2 (2022): 69.
Chicago
Yi, Z., Pu, Y., Gou, R., Chen, Y., Ren, X., Liu, W., Dong, P."[Corrigendum] Silencing of RIPK4 inhibits epithelial‑mesenchymal transition by inactivating the Wnt/β‑catenin signaling pathway in osteosarcoma". Molecular Medicine Reports 25, no. 2 (2022): 69. https://doi.org/10.3892/mmr.2022.12585
Copy and paste a formatted citation
x
Spandidos Publications style
Yi Z, Pu Y, Gou R, Chen Y, Ren X, Liu W and Dong P: [Corrigendum] Silencing of RIPK4 inhibits epithelial‑mesenchymal transition by inactivating the Wnt/β‑catenin signaling pathway in osteosarcoma. Mol Med Rep 25: 69, 2022.
APA
Yi, Z., Pu, Y., Gou, R., Chen, Y., Ren, X., Liu, W., & Dong, P. (2022). [Corrigendum] Silencing of RIPK4 inhibits epithelial‑mesenchymal transition by inactivating the Wnt/β‑catenin signaling pathway in osteosarcoma. Molecular Medicine Reports, 25, 69. https://doi.org/10.3892/mmr.2022.12585
MLA
Yi, Z., Pu, Y., Gou, R., Chen, Y., Ren, X., Liu, W., Dong, P."[Corrigendum] Silencing of RIPK4 inhibits epithelial‑mesenchymal transition by inactivating the Wnt/β‑catenin signaling pathway in osteosarcoma". Molecular Medicine Reports 25.2 (2022): 69.
Chicago
Yi, Z., Pu, Y., Gou, R., Chen, Y., Ren, X., Liu, W., Dong, P."[Corrigendum] Silencing of RIPK4 inhibits epithelial‑mesenchymal transition by inactivating the Wnt/β‑catenin signaling pathway in osteosarcoma". Molecular Medicine Reports 25, no. 2 (2022): 69. https://doi.org/10.3892/mmr.2022.12585
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