International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.
International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.
Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.
Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.
Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.
Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.
Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.
International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.
Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.
Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.
Publishes open-access research on using epigenetics to advance understanding and treatment of human disease.
An International Open Access Journal Devoted to General Medicine.
Oncol Lett 14: [Related article:] 1884-1888, 2017; DOI: 10.3892/ol.2017.6360
Subsequently to the publication of this paper, an interested reader drew to the authors’ attention that the “Control” lane in the E2F1 panel of Fig. 2 appeared to be strikingly similar to the “24 h” TGF-β treatment lane in the E2F1 panel of Fig. 4.
Although the authors were unable to locate the original raw data to evaluate how the errors in figure compilation had occurred, they were able to repeat these experiments, and thereby could confirm that: i) E2F1 truly may can be down-regulated in the MM1s cell line through siRNA targeting E2F1; and ii) TGF-β can induce the immediate expression of E2F1 in the MM1s cell line within 24 h.
The revised versions of Figs. 2 and 4, containing the new data, are shown opposite. The authors regret the errors that were made in the compilation of the orginal figures, and are grateful to the editor of Oncology Letters for allowing them the opportunity to publish a Corrigendum. Furthermore, they apologise to the readership for any inconvenience caused.