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Oncology Letters
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Print ISSN: 1792-1074 Online ISSN: 1792-1082
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February-2021 Volume 21 Issue 2

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International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

Molecular Medicine Reports

Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

Oncology Reports

Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

Oncology Letters

Oncology Letters

Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

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Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

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Molecular and Clinical Oncology

International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

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Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

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International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

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International Journal of Epigenetics

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Correction Open Access

[Corrigendum] TGF‑β induces growth suppression in multiple myeloma MM.1S cells via E2F1

  • Authors:
    • Xialei Liu
    • Hui Guo
    • Yuting Wei
    • Chaonong Cai
    • Baimeng Zhang
    • Jian Li
  • View Affiliations / Copyright

    Affiliations: Department of General Surgery 3, The Fifth Affiliated Hospital of Sun Yat‑sen University, Zhuhai, Guangdong 519000, P.R. China, Department of Ultrasound, The Fifth Affiliated Hospital of Sun Yat‑sen University, Zhuhai, Guangdong 519000, P.R. China, Department of Hemodialysis, The Fifth Affiliated Hospital of Sun Yat‑sen University, Zhuhai, Guangdong 519000, P.R. China
    Copyright: © Liu et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
  • Article Number: 83
    |
    Published online on: December 2, 2020
       https://doi.org/10.3892/ol.2020.12344
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Article

Oncol Lett 14: [Related article:] 1884-1888, 2017; DOI: 10.3892/ol.2017.6360

Subsequently to the publication of this paper, an interested reader drew to the authors’ attention that the “Control” lane in the E2F1 panel of Fig. 2 appeared to be strikingly similar to the “24 h” TGF-β treatment lane in the E2F1 panel of Fig. 4.

Figure 2.

The efficiency of E2F1 knockdown by siRNA was verified by immunoblotting using an E2F1-specific antibody. Ctl, cells without transfection; siRNA, small interfering RNA; Ctl siRNA, cells transfected with unrelated siRNA controls; E2F1 siRNA, cells transfected with anti-E2F1 siRNA.

Figure 4.

Western blot analysis of the phosphorylation state of pRb and total E2F1 protein levels in TGF-β-treated cells shows TGF-β induces E2F1 protein expression levels transiently. TGF-β, transforming growth factor-β; pRb, retinoblastoma tumor-suppressor protein.

Although the authors were unable to locate the original raw data to evaluate how the errors in figure compilation had occurred, they were able to repeat these experiments, and thereby could confirm that: i) E2F1 truly may can be down-regulated in the MM1s cell line through siRNA targeting E2F1; and ii) TGF-β can induce the immediate expression of E2F1 in the MM1s cell line within 24 h.

The revised versions of Figs. 2 and 4, containing the new data, are shown opposite. The authors regret the errors that were made in the compilation of the orginal figures, and are grateful to the editor of Oncology Letters for allowing them the opportunity to publish a Corrigendum. Furthermore, they apologise to the readership for any inconvenience caused.

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Copy and paste a formatted citation
Spandidos Publications style
Liu X, Guo H, Wei Y, Cai C, Zhang B and Li J: [Corrigendum] TGF‑β induces growth suppression in multiple myeloma MM.1S cells via E2F1. Oncol Lett 21: 83, 2021.
APA
Liu, X., Guo, H., Wei, Y., Cai, C., Zhang, B., & Li, J. (2021). [Corrigendum] TGF‑β induces growth suppression in multiple myeloma MM.1S cells via E2F1. Oncology Letters, 21, 83. https://doi.org/10.3892/ol.2020.12344
MLA
Liu, X., Guo, H., Wei, Y., Cai, C., Zhang, B., Li, J."[Corrigendum] TGF‑β induces growth suppression in multiple myeloma MM.1S cells via E2F1". Oncology Letters 21.2 (2021): 83.
Chicago
Liu, X., Guo, H., Wei, Y., Cai, C., Zhang, B., Li, J."[Corrigendum] TGF‑β induces growth suppression in multiple myeloma MM.1S cells via E2F1". Oncology Letters 21, no. 2 (2021): 83. https://doi.org/10.3892/ol.2020.12344
Copy and paste a formatted citation
x
Spandidos Publications style
Liu X, Guo H, Wei Y, Cai C, Zhang B and Li J: [Corrigendum] TGF‑β induces growth suppression in multiple myeloma MM.1S cells via E2F1. Oncol Lett 21: 83, 2021.
APA
Liu, X., Guo, H., Wei, Y., Cai, C., Zhang, B., & Li, J. (2021). [Corrigendum] TGF‑β induces growth suppression in multiple myeloma MM.1S cells via E2F1. Oncology Letters, 21, 83. https://doi.org/10.3892/ol.2020.12344
MLA
Liu, X., Guo, H., Wei, Y., Cai, C., Zhang, B., Li, J."[Corrigendum] TGF‑β induces growth suppression in multiple myeloma MM.1S cells via E2F1". Oncology Letters 21.2 (2021): 83.
Chicago
Liu, X., Guo, H., Wei, Y., Cai, C., Zhang, B., Li, J."[Corrigendum] TGF‑β induces growth suppression in multiple myeloma MM.1S cells via E2F1". Oncology Letters 21, no. 2 (2021): 83. https://doi.org/10.3892/ol.2020.12344
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