1
|
Chen W, Zheng R, Baade PD, Zhang S, Zeng
H, Bray F, Jemal A, Yu XQ and He J: Cancer statistics in China,
2015. CA Cancer J Clin. 66:115–132. 2016. View Article : Google Scholar : PubMed/NCBI
|
2
|
Bonelli P, Borrelli A, Tuccillo FM,
Silvestro L, Palaia R and Buonaguro FM: Precision medicine in
gastric cancer. World J Gastrointest Oncol. 11:804–829. 2019.
View Article : Google Scholar : PubMed/NCBI
|
3
|
Symeonidis D, Diamantis A, Bompou E and
Tepetes K: Current role of lymphadenectomy in gastric cancer
surgery. J BUON. 24:1761–1767. 2019.PubMed/NCBI
|
4
|
Zhang F, Huang X, Song Y, Gao P, Zhou C,
Guo Z, Shi J, Wu Z and Wang Z: Conversion surgery for stage IV
gastric cancer. Front Oncol. 9:11582019. View Article : Google Scholar : PubMed/NCBI
|
5
|
Shang Q, Yang Z, Jia R and Ge S: The novel
roles of circRNAs in human cancer. Mol Cancer. 18:62019. View Article : Google Scholar : PubMed/NCBI
|
6
|
Chen LL and Yang L: Regulation of circRNA
biogenesis. RNA Biol. 12:381–388. 2015. View Article : Google Scholar : PubMed/NCBI
|
7
|
Jeck WR, Sorrentino JA, Wang K, Slevin MK,
Burd CE, Liu J, Marzluff WF and Sharpless NE: Circular RNAs are
abundant, conserved, and associated with ALU repeats. RNA.
19:141–157. 2013. View Article : Google Scholar : PubMed/NCBI
|
8
|
Chen X, Yang T, Wang W, Xi W, Zhang T, Li
Q, Yang A and Wang T: Circular RNAs in immune responses and immune
diseases. Theranostics. 9:588–607. 2019. View Article : Google Scholar : PubMed/NCBI
|
9
|
Rong D, Tang W, Li Z, Zhou J, Shi J, Wang
H and Cao H: Novel insights into circular RNAs in clinical
application of carcinomas. OncoTargets Ther. 10:2183–2188. 2017.
View Article : Google Scholar
|
10
|
Han B, Chao J and Yao H: Circular RNA and
its mechanisms in disease: From the bench to the clinic. Pharmacol
Ther. 187:31–44. 2018. View Article : Google Scholar : PubMed/NCBI
|
11
|
Dong Y, He D, Peng Z, Peng W, Shi W, Wang
J, Li B, Zhang C and Duan C: Circular RNAs in cancer: An emerging
key player. J Hematol Oncol. 10:22017. View Article : Google Scholar : PubMed/NCBI
|
12
|
He J, Xie Q, Xu H, Li J and Li Y: Circular
RNAs and cancer. Cancer Lett. 396:138–144. 2017. View Article : Google Scholar : PubMed/NCBI
|
13
|
Dang Y, Ouyang X, Zhang F, Wang K, Lin Y,
Sun B, Wang Y, Wang L and Huang Q: Circular RNAs expression
profiles in human gastric cancer. Sci Rep. 7:90602017. View Article : Google Scholar : PubMed/NCBI
|
14
|
Sui W, Shi Z, Xue W, Ou M, Zhu Y, Chen J,
Lin H, Liu F and Dai Y: Circular RNA and gene expression profiles
in gastric cancer based on microarray chip technology. Oncol Rep.
37:1804–1814. 2017. View Article : Google Scholar : PubMed/NCBI
|
15
|
Shen Y, Zhang J, Fu Z, Zhang B, Chen M,
Ling X and Zou X: Gene microarray analysis of the circular RNAs
expression profile in human gastric cancer. Oncol Lett.
15:9965–9972. 2018.PubMed/NCBI
|
16
|
Huang YS, Jie N, Zou KJ and Weng Y:
Expression profile of circular RNAs in human gastric cancer
tissues. Mol Med Rep. 16:2469–2476. 2017. View Article : Google Scholar : PubMed/NCBI
|
17
|
Bustin SA, Benes V, Garson JA, Hellemans
J, Huggett J, Kubista M, Mueller R, Nolan T, Pfaffl MW, Shipley GL,
et al: The MIQE guidelines: Minimum information for publication of
quantitative real-time PCR experiments. Clin Chem. 55:611–622.
2009. View Article : Google Scholar : PubMed/NCBI
|
18
|
Livak KJ and Schmittgen TD: Analysis of
relative gene expression data using real-time quantitative PCR and
the 2(-Delta Delta C(T)) method. Methods. 25:402–408. 2001.
View Article : Google Scholar : PubMed/NCBI
|
19
|
Liang D and Wilusz JE: Short intronic
repeat sequences facilitate circular RNA production. Genes Dev.
28:2233–2247. 2014. View Article : Google Scholar : PubMed/NCBI
|
20
|
Sellers AH: The clinical classification of
malignant tumours: The TNM system. Can Med Assoc J. 105:836passim.
1971.PubMed/NCBI
|
21
|
Dang CV: MYC on the path to cancer. Cell.
149:22–35. 2012. View Article : Google Scholar : PubMed/NCBI
|
22
|
Mitchell RA, Luwor RB and Burgess AW:
Epidermal growth factor receptor: Structure-function informing the
design of anticancer therapeutics. Exp Cell Res. 371:1–19. 2018.
View Article : Google Scholar : PubMed/NCBI
|
23
|
Juríková M, Danihel Ľ, Polák Š and Varga
I: Ki67, PCNA, and MCM proteins: Markers of proliferation in the
diagnosis of breast cancer. Acta Histochem. 118:544–552. 2016.
View Article : Google Scholar : PubMed/NCBI
|
24
|
Ahn HS, Lee HJ, Hahn S, Kim WH, Lee KU,
Sano T, Edge SB and Yang HK: Evaluation of the seventh American
Joint Committee on Cancer/International Union Against Cancer
Classification of gastric adenocarcinoma in comparison with the
sixth classification. Cancer. 116:5592–5598. 2010. View Article : Google Scholar : PubMed/NCBI
|
25
|
Tulchinsky E, Demidov O, Kriajevska M,
Barlev NA and Imyanitov E: EMT: A mechanism for escape from
EGFR-targeted therapy in lung cancer. Biochim Biophys Acta Rev
Cancer. 1871:29–39. 2019. View Article : Google Scholar : PubMed/NCBI
|
26
|
Nieto MA, Huang RY, Jackson RA and Thiery
JP: Emt: 2016. Cell. 166:21–45. 2016. View Article : Google Scholar : PubMed/NCBI
|
27
|
Christofori G and Semb H: The role of the
cell-adhesion molecule E-cadherin as a tumour-suppressor gene.
Trends Biochem Sci. 24:73–76. 1999. View Article : Google Scholar : PubMed/NCBI
|
28
|
Kim K, Lu Z and Hay ED: Direct evidence
for a role of beta-catenin/LEF-1 signaling pathway in induction of
EMT. Cell Biol Int. 26:463–476. 2002. View Article : Google Scholar : PubMed/NCBI
|
29
|
Mendez MG, Kojima S and Goldman RD:
Vimentin induces changes in cell shape, motility, and adhesion
during the epithelial to mesenchymal transition. FASEB J.
24:1838–1851. 2010. View Article : Google Scholar : PubMed/NCBI
|
30
|
Hazan RB, Phillips GR, Qiao RF, Norton L
and Aaronson SA: Exogenous expression of N-cadherin in breast
cancer cells induces cell migration, invasion, and metastasis. J
Cell Biol. 148:779–790. 2000. View Article : Google Scholar : PubMed/NCBI
|
31
|
Zhang K, Corsa CA, Ponik SM, Prior JL,
Piwnica-Worms D, Eliceiri KW, Keely PJ and Longmore GD: The
collagen receptor discoidin domain receptor 2 stabilizes SNAIL1 to
facilitate breast cancer metastasis. Nat Cell Biol. 15:677–687.
2013. View Article : Google Scholar : PubMed/NCBI
|
32
|
Lin CY, Tsai PH, Kandaswami CC, Lee PP,
Huang CJ, Hwang JJ and Lee MT: Matrix metalloproteinase-9
cooperates with transcription factor Snail to induce
epithelial-mesenchymal transition. Cancer Sci. 102:815–827. 2011.
View Article : Google Scholar : PubMed/NCBI
|
33
|
Yan T, Lin Z, Jiang J, Lu S, Chen M, Que
H, He X, Que G, Mao J, Xiao J, et al: MMP14 regulates cell
migration and invasion through epithelial-mesenchymal transition in
nasopharyngeal carcinoma. Am J Transl Res. 7:950–958.
2015.PubMed/NCBI
|
34
|
Amin MB, Greene FL, Edge SB, Compton CC,
Gershenwald JE, Brookland RK, Meyer L, Gress DM, Byrd DR and
Winchester DP: The Eighth Edition AJCC Cancer Staging Manual:
Continuing to build a bridge from a population-based to a more
‘personalized’ approach to cancer staging. CA Cancer J Clin.
67:93–99. 2017. View Article : Google Scholar : PubMed/NCBI
|
35
|
van Denderen BJ and Thompson EW: Cancer:
The to and fro of tumour spread. Nature. 493:487–488. 2013.
View Article : Google Scholar : PubMed/NCBI
|
36
|
Ghosh P, Beas AO, Bornheimer SJ,
Garcia-Marcos M, Forry EP, Johannson C, Ear J, Jung BH, Cabrera B,
Carethers JM, et al: A G{alpha}i-GIV molecular complex binds
epidermal growth factor receptor and determines whether cells
migrate or proliferate. Mol Biol Cell. 21:2338–2354. 2010.
View Article : Google Scholar : PubMed/NCBI
|
37
|
Ghosh P, Garcia-Marcos M, Bornheimer SJ
and Farquhar MG: Activation of Galphai3 triggers cell migration via
regulation of GIV. J Cell Biol. 182:381–393. 2008. View Article : Google Scholar : PubMed/NCBI
|
38
|
Beas AO, Taupin V, Teodorof C, Nguyen LT,
Garcia-Marcos M and Farquhar MG: Gαs promotes EEA1 endosome
maturation and shuts down proliferative signaling through
interaction with GIV (Girdin). Mol Biol Cell. 23:4623–4634. 2012.
View Article : Google Scholar : PubMed/NCBI
|
39
|
Pece S and Gutkind JS: Signaling from
E-cadherins to the MAPK pathway by the recruitment and activation
of epidermal growth factor receptors upon cell-cell contact
formation. J Biol Chem. 275:41227–41233. 2000. View Article : Google Scholar : PubMed/NCBI
|
40
|
Moiseyenko VM, Procenko SA, Levchenko EV,
Barchuk AS, Moiseyenko FV, Iyevleva AG, Mitiushkina NV, Togo AV,
Semionov II, Ivantsov AO, et al: High efficacy of first-line
gefitinib in non-Asian patients with EGFR-mutated lung
adenocarcinoma. Onkologie. 33:231–238. 2010. View Article : Google Scholar : PubMed/NCBI
|
41
|
Edinger N, Lebendiker M, Klein S, Zigler
M, Langut Y and Levitzki A: Targeting polyIC to EGFR
over-expressing cells using a dsRNA binding protein domain tethered
to EGF. PLoS One. 11:e01623212016. View Article : Google Scholar : PubMed/NCBI
|
42
|
Qian X, Karpova T, Sheppard AM, McNally J
and Lowy DR: E-cadherin-mediated adhesion inhibits ligand-dependent
activation of diverse receptor tyrosine kinases. EMBO J.
23:1739–1748. 2004. View Article : Google Scholar : PubMed/NCBI
|
43
|
Chen D, Wu Z, Luo LJ, Huang X, Qian WQ,
Wang H, Li SH and Liu J: E-cadherin maintains the activity of
neural stem cells and inhibits the migration. Int J Clin Exp
Pathol. 8:14247–14251. 2015.PubMed/NCBI
|
44
|
Boyer B, Vallés AM and Edme N: Induction
and regulation of epithelial-mesenchymal transitions. Biochem
Pharmacol. 60:1091–1099. 2000. View Article : Google Scholar : PubMed/NCBI
|
45
|
Hynes RO: The extracellular matrix: Not
just pretty fibrils. Science. 326:1216–1219. 2009. View Article : Google Scholar : PubMed/NCBI
|
46
|
Venning FA, Wullkopf L and Erler JT:
Targeting ECM Disrupts Cancer Progression. Front Oncol. 5:2242015.
View Article : Google Scholar : PubMed/NCBI
|