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Oncology Letters
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Print ISSN: 1792-1074 Online ISSN: 1792-1082
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May-2021 Volume 21 Issue 5

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Journals

International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

Molecular Medicine Reports

Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

Oncology Reports

Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

Oncology Letters

Oncology Letters

Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

Biomedical Reports

Biomedical Reports

Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

Molecular and Clinical Oncology

Molecular and Clinical Oncology

International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

World Academy of Sciences Journal

World Academy of Sciences Journal

Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

International Journal of Functional Nutrition

International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

International Journal of Epigenetics

International Journal of Epigenetics

Publishes open-access research on using epigenetics to advance understanding and treatment of human disease.

Medicine International

Medicine International

An International Open Access Journal Devoted to General Medicine.

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Correction Open Access

[Corrigendum] Dihydroartemisinin inhibits the Raf/ERK/MEK and PI3K/AKT pathways in glioma cells

  • Authors:
    • Wei Du
    • Changhe Pang
    • Yake Xue
    • Qingjun Zhang
    • Xinting Wei

  • View Affiliations / Copyright

    Affiliations: Department of Neurosurgery, The First Affiliated Hospital, Zhengzhou University, Zhengzhou, Henan 450052, P.R. China, Department of Neurosurgery, Peking University People's Hospital, Beijing 100044, P.R. China
    Copyright: © Du et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
  • Article Number: 375
    |
    Published online on: March 15, 2021
       https://doi.org/10.3892/ol.2021.12636
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Article

Oncol Lett 10: [Related article:] 3266-3270, 2015; DOI: 10.3892/ol.2015.3699

Following the publication of the above article, an interested reader drew to the authors’ attention that lane 1 of the pMEK/BT325 cells panel in Fig. 3A appeared strikingly similar to lane 1 of the BCL-2 / C6 cells panel in Fig. 5B, albeit the protein band was inverted. Furthermore, lanes 1–3 of the MEK / C6 cells panel in Fig. 3B looked strikingly similar to lanes 1–3 of the GAPDH / BT325 cells panel in Fig. 5A.

Figure 3.

DHA inhibits RAF/MEK/ERK and PI3K/AKT signaling pathways in (A) BT325 and (B) C6 cells. Cells were treated with the indicated concentrations of DHA for 24 h. Western blot analysis was performed using specific antibodies against the indicated proteins.

The authors re-examined their original data, and realized that some of the data featured in Fig. 3 had been assembled incorrectly owing to the large number of proteins that had been investigated, and given the fact that not only one person was responsible for generating these data. The corrected version of Fig. 3 is shown opposite. Note that the corrections made to this figure do not affect the overall conclusions reported in the paper. The authors are grateful to the Editor of Oncology Letters for allowing them the opportunity to publish this corrigendum, and apologize to the readership for any inconvenience caused.

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Copy and paste a formatted citation
Spandidos Publications style
Du W, Pang C, Xue Y, Zhang Q and Wei X: [Corrigendum] Dihydroartemisinin inhibits the Raf/ERK/MEK and PI3K/AKT pathways in glioma cells. Oncol Lett 21: 375, 2021.
APA
Du, W., Pang, C., Xue, Y., Zhang, Q., & Wei, X. (2021). [Corrigendum] Dihydroartemisinin inhibits the Raf/ERK/MEK and PI3K/AKT pathways in glioma cells. Oncology Letters, 21, 375. https://doi.org/10.3892/ol.2021.12636
MLA
Du, W., Pang, C., Xue, Y., Zhang, Q., Wei, X."[Corrigendum] Dihydroartemisinin inhibits the Raf/ERK/MEK and PI3K/AKT pathways in glioma cells". Oncology Letters 21.5 (2021): 375.
Chicago
Du, W., Pang, C., Xue, Y., Zhang, Q., Wei, X."[Corrigendum] Dihydroartemisinin inhibits the Raf/ERK/MEK and PI3K/AKT pathways in glioma cells". Oncology Letters 21, no. 5 (2021): 375. https://doi.org/10.3892/ol.2021.12636
Copy and paste a formatted citation
x
Spandidos Publications style
Du W, Pang C, Xue Y, Zhang Q and Wei X: [Corrigendum] Dihydroartemisinin inhibits the Raf/ERK/MEK and PI3K/AKT pathways in glioma cells. Oncol Lett 21: 375, 2021.
APA
Du, W., Pang, C., Xue, Y., Zhang, Q., & Wei, X. (2021). [Corrigendum] Dihydroartemisinin inhibits the Raf/ERK/MEK and PI3K/AKT pathways in glioma cells. Oncology Letters, 21, 375. https://doi.org/10.3892/ol.2021.12636
MLA
Du, W., Pang, C., Xue, Y., Zhang, Q., Wei, X."[Corrigendum] Dihydroartemisinin inhibits the Raf/ERK/MEK and PI3K/AKT pathways in glioma cells". Oncology Letters 21.5 (2021): 375.
Chicago
Du, W., Pang, C., Xue, Y., Zhang, Q., Wei, X."[Corrigendum] Dihydroartemisinin inhibits the Raf/ERK/MEK and PI3K/AKT pathways in glioma cells". Oncology Letters 21, no. 5 (2021): 375. https://doi.org/10.3892/ol.2021.12636
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