|
1
|
Neppl-Huber C, Zappa M, Coebergh JW, et
al: Changes in incidence, survival and mortality of prostate cancer
in Europe and the United States in the PSA era: additional
diagnoses and avoided deaths. Ann Oncol. 23:1325–1334. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Heidenreich A, Bellmunt J, Bolla M, et al:
EAU guidelines on prostate cancer. Part 1: screening, diagnosis,
and treatment of clinically localised disease. Eur Urol. 59:61–71.
2011. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Bonkhoff H: Factors implicated in
radiation therapy failure and radiosensitization of prostate
cancer. Prostate Cancer. 2012:5932412012. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Beckendorf V, Guerif S, Le PE, et al: 70
Gy versus 80 Gy in localized prostate cancer: 5-year results of
GETUG 06 randomized trial. Int J Radiat Oncol Biol Phys.
80:1056–1063. 2011.PubMed/NCBI
|
|
5
|
Merry C, Fu K, Wang J, Yeh IJ and Zhang Y:
Targeting the checkpoint kinase Chk1 in cancer therapy. Cell Cycle.
9:279–283. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Stracker TH, Usui T and Petrini JH: Taking
the time to make important decisions: the checkpoint effector
kinases Chk1 and Chk2 and the DNA damage response. DNA Repair.
8:1047–1054. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Jackson SP and Bartek J: The DNA-damage
response in human biology and disease. Nature. 461:1071–1078. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Diehn M and Clarke MF: Cancer stem cells
and radiotherapy: new insights into tumor radioresistance. J Natl
Cancer Inst. 98:1755–1757. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Baumann M, Krause M and Hill R: Exploring
the role of cancer stem cells in radioresistance. Nat Rev Cancer.
8:545–554. 2008. View
Article : Google Scholar : PubMed/NCBI
|
|
10
|
Hittelman WN, Liao Y, Wang L and Milas L:
Are cancer stem cells radioresistant. Future Oncol. 6:1563–1576.
2010. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Visvader JE and Lindeman GJ: Cancer stem
cells in solid tumours: accumulating evidence and unresolved
questions. Nat Rev Cancer. 8:755–768. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Eyler CE and Rich JN: Survival of the
fittest: cancer stem cells in therapeutic resistance and
angiogenesis. J Clin Oncol. 26:2839–2845. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Wang G, Wang Z, Sarkar FH and Wei W:
Targeting prostate cancer stem cells for cancer therapy. Discov
Med. 13:135–142. 2012.PubMed/NCBI
|
|
14
|
Maitland NJ and Collins AT: Prostate
cancer stem cells: a new target for therapy. J Clin Oncol.
26:2862–2870. 2008. View Article : Google Scholar
|
|
15
|
Wu J, Lai G, Wan F, et al: Knockdown of
checkpoint kinase 1 is associated with the increased
radiosensitivity of glioblastoma stem-like cells. Tohoku J Exp Med.
226:267–274. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Xiao Z, Liu Q, Zhao B, Wu J and Lei T:
Hypoxia induces hemorrhagic transformation in pituitary adenomas
via the HIF-1α signaling pathway. Oncol Rep. 26:1457–1464.
2011.PubMed/NCBI
|
|
17
|
Ma CX, Janetka JW and Piwnica-Worms H:
Death by releasing the breaks: CHK1 inhibitors as cancer
therapeutics. Trends Mol Med. 17:88–96. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Carrassa L and Damia G: Unleashing Chk1 in
cancer therapy. Cell Cycle. 10:2121–2128. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Chen Y and Sanchez Y: Chk1 in the DNA
damage response: conserved roles from yeasts to mammals. DNA
Repair. 3:1025–1032. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Lang SH, Frame FM and Collins AT: Prostate
cancer stem cells. J Pathol. 217:299–306. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Collins AT, Berry PA, Hyde C, Stower MJ
and Maitland NJ: Prospective identification of tumorigenic prostate
cancer stem cells. Cancer Res. 65:10946–10951. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Li C, Heidt DG, Dalerba P, et al:
Identification of pancreatic cancer stem cells. Cancer Res.
67:1030–1037. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Tang DG, Patrawala L, Calhoun T, et al:
Prostate cancer stem/progenitor cells: identification,
characterization, and implications. Mol Carcinog. 46:1–14. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Wei C, Guomin W, Yujun L and Ruizhe Q:
Cancer stem-like cells in human prostate carcinoma cells DU145: the
seeds of the cell line. Cancer Biol Ther. 6:763–768. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Syljuasen RG, Sorensen CS, Hansen LT, et
al: Inhibition of human Chk1 causes increased initiation of DNA
replication, phosphorylation of ATR targets, and DNA breakage. Mol
Cell Biol. 25:3553–3562. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Binder A and Bohm L: Influence of
irradiation and pentoxifylline on histone H3 phosphorylation in
human tumour cell lines. Cell Prolif. 35:37–47. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Sorensen CS, Hansen LT, Dziegielewski J,
et al: The cell-cycle checkpoint kinase Chk1 is required for
mammalian homologous recombination repair. Nat Cell Biol.
7:195–201. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Bahassi EM, Ovesen JL, Riesenberg AL,
Bernstein WZ, Hasty PE and Stambrook PJ: The checkpoint kinases
Chk1 and Chk2 regulate the functional associations between hBRCA2
and Rad51 in response to DNA damage. Oncogene. 27:3977–3985. 2008.
View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Xiao Z, Chen Z, Gunasekera AH, et al: Chk1
mediates S and G2 arrests through Cdc25A degradation in response to
DNA-damaging agents. J Biol Chem. 278:21767–21773. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Sanchez Y, Wong C, Thoma RS, et al:
Conservation of the Chk1 checkpoint pathway in mammals: linkage of
DNA damage to Cdk regulation through Cdc25. Science. 277:1497–1501.
1997. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Sorensen CS, Melixetian M, Klein DK and
Helin K: NEK11: linking CHK1 and CDC25A in DNA damage checkpoint
signaling. Cell Cycle. 9:450–455. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Sidi S, Sanda T, Kennedy RD, et al: Chk1
suppresses a caspase-2 apoptotic response to DNA damage that
bypasses p53, Bcl-2, and caspase-3. Cell. 133:864–877. 2008.
View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Dumont F, Altmeyer A and Bischoff P:
Radiosensitising agents for the radiotherapy of cancer: novel
molecularly targeted approaches. Expert Opin Ther Pat. 19:775–799.
2009. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Yang H, Yoon SJ, Jin J, et al: Inhibition
of checkpoint kinase 1 sensitizes lung cancer brain metastases to
radiotherapy. Biochem Biophys Res Commun. 406:53–58. 2011.
View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Meuth M: Chk1 suppressed cell death. Cell
Div. 5:212010. View Article : Google Scholar
|
|
36
|
Smith J, Tho LM, Xu N and Gillespie DA:
The ATM-Chk2 and ATR-Chk1 pathways in DNA damage signaling and
cancer. Adv Cancer Res. 108:73–112. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Gao Q, Zhou J, Huang X, et al: Selective
targeting of checkpoint kinase 1 in tumor cells with a novel potent
oncolytic adenovirus. Mol Ther. 13:928–937. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Freytag SO, Stricker H, Pegg J, et al:
Phase I study of replication-competent adenovirus-mediated
double-suicide gene therapy in combination with conventional-dose
three-dimensional conformal radiation therapy for the treatment of
newly diagnosed, intermediate- to high-risk prostate cancer. Cancer
Res. 63:7497–7506. 2003.
|
|
39
|
Geoerger B, van Beusechem VW, Opolon P, et
al: Expression of p53, or targeting towards EGFR, enhances the
oncolytic potency of conditionally replicative adenovirus against
neuroblastoma. J Gene Med. 7:584–594. 2005. View Article : Google Scholar
|
|
40
|
Tsuruta Y, Pereboeva L, Breidenbach M, et
al: A fiber-modified mesothelin promoter-based conditionally
replicating adenovirus for treatment of ovarian cancer. Clin Cancer
Res. 14:3582–3588. 2008. View Article : Google Scholar
|
