|
1
|
Siegel R, Ward E, Brawley O and Jemal A:
Cancer statistics, 2011: the impact of eliminating socioeconomic
and racial disparities on premature cancer deaths. CA Cancer J
Clin. 61:212–236. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Damber JE and Aus G: Prostate cancer.
Lancet. 371:1710–1721. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Bartel DP: MicroRNAs: genomics,
biogenesis, mechanism, and function. Cell. 116:281–297. 2004.
View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Meister G, Landthaler M, Patkaniowska A,
Dorsett Y, Teng G and Tuschl T: Human Argonaute2 mediates RNA
cleavage targeted by miRNAs and siRNAs. Mol Cell. 15:185–197. 2004.
View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Feng B, Wang R and Chen LB: Review of
miR-200b and cancer chemosensitivity. Biomed Pharmacother.
66:397–402. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Zhu W, Xu H, Zhu D, et al: miR-200bc/429
cluster modulates multidrug resistance of human cancer cell lines
by targeting BCL2 and XIAP. Cancer Chemother Pharmacol. 69:723–731.
2012. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Wellner U, Schubert J, Burk UC, et al: The
EMT-activator ZEB1 promotes tumorigenicity by repressing
stemness-inhibiting microRNAs. Nat Cell Biol. 11:1487–1495. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Guo BH, Feng Y, Zhang R, et al: Bmi-1
promotes invasion and metastasis, and its elevated expression is
correlated with an advanced stage of breast cancer. Mol Cancer.
10:102011. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Bhattacharya R, Nicoloso M, Arvizo R, et
al: MiR-15a and MiR-16 control Bmi-1 expression in ovarian cancer.
Cancer Res. 69:9090–9095. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Song W, Tao K, Li H, et al: Bmi-1 is
related to proliferation, survival and poor prognosis in pancreatic
cancer. Cancer Sci. 101:1754–1760. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
He X, Dong Y, Wu CW, et al: MicroRNA-218
inhibits cell cycle progression and promotes apoptosis in colon
cancer by downregulating BMI1 polycomb ring finger oncogene. Mol
Med. 18:1491–1498. 2013.PubMed/NCBI
|
|
12
|
Song LB, Li J, Liao WT, et al: The
polycomb group protein Bmi-1 represses the tumor-suppressor PTEN
and induces epithelial-mesenchymal transition in human
nasopharyngeal epithelial cells. J Clin Invest. 119:3626–3636.
2009. View
Article : Google Scholar
|
|
13
|
Molofsky AV, Pardal R, Iwashita T, Park
IK, Clarke MF and Morrison SJ: Bmi-1 dependence distinguishes
neural stem cell self-renewal from progenitor proliferation.
Nature. 425:962–967. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Lukacs RU, Memarzadeh S, Wu H and Witte
ON: Bmi-1 is a crucial regulator of prostate stem cell self-renewal
and malignant transformation. Cell Stem Cell. 7:682–693. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Livak KJ and Schmittgen TD: Analysis of
relative gene expression data using real-time quantitative PCR and
the 2−ΔΔCT method. Methods. 25:402–408. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Onder TT, Kara N, Cherry A, et al:
Chromatin-modifying enzymes as modulators of reprogramming. Nature.
483:598–602. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Korpal M, Lee ES, Hu G and Kang Y: The
miR-200 family inhibits epithelial-mesenchymal transition and
cancer cell migration by direct targeting of E-cadherin
transcriptional repressors ZEB1 and ZEB2. J Biol
Chem. 283:14910–14914. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Fan C, He L, Kapoor A, et al: Bmi1
promotes prostate tumorigenesis via inhibiting p16INK4A
and p14ARF expression. Biochim Biophys Acta.
1782:642–648. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Dong P, Kaneuchi M, Watari H, et al:
MicroRNA-194 inhibits epithelial to mesenchymal transition of
endometrial cancer cells by targeting oncogene BMI-1. Mol Cancer.
10:992011. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Schaefer A, Jung M, Mollenkopf HJ, et al:
Diagnostic and prognostic implications of microRNA profiling in
prostate carcinoma. Int J Cancer. 126:1166–1176. 2010.PubMed/NCBI
|
|
21
|
Kong D, Banerjee S, Ahmad A, et al:
Epithelial to mesenchymal transition is mechanistically linked with
stem cell signatures in prostate cancer cells. PLoS One.
5:e124452010. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Park SM, Gaur AB, Lengyel E and Peter ME:
The miR-200 family determines the epithelial phenotype of cancer
cells by targeting the E-cadherin repressors ZEB1 and ZEB2. Genes
Dev. 22:894–907. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Liu YN, Yin JJ, Abou-Kheir W, et al: MiR-1
and miR-200 inhibit EMT via Slug-dependent and tumorigenesis via
Slug-independent mechanisms. Oncogene. 32:296–306. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Tang H, Kong Y, Guo J, et al: Diallyl
disulfide suppresses proliferation and induces apoptosis in human
gastric cancer through Wnt-1 signaling pathway by up-regulation of
miR-200b and miR-22. Cancer Lett. 340:72–81. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Pacurari M, Addison JB, Bondalapati N, et
al: The microRNA-200 family targets multiple non-small cell lung
cancer prognostic markers in H1299 cells and BEAS-2B cells. Int J
Oncol. 43:548–560. 2013.PubMed/NCBI
|
|
26
|
Yoshino H, Enokida H, Itesako T, et al:
Epithelial-mesenchymal transition-related microRNA-200s regulate
molecular targets and pathways in renal cell carcinoma. J Hum
Genet. 58:508–516. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Köhler CU, Bryk O, Meier S, et al:
Analyses in human urothelial cells identify methylation of miR-152,
miR-200b and miR-10a genes as candidate bladder cancer biomarkers.
Biochem Biophys Res Commun. 438:48–53. 2013.
|
|
28
|
Cao Q, Mani RS, Ateeq B, et al:
Coordinated regulation of polycomb group complexes through
microRNAs in cancer. Cancer Cell. 20:187–199. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Sun L, Yao Y, Liu B, et al: MiR-200b and
miR-15b regulate chemotherapy-induced epithelial-mesenchymal
transition in human tongue cancer cells by targeting BMI1.
Oncogene. 31:432–445. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Kang MK, Kim RH, Kim SJ, et al: Elevated
Bmi-1 expression is associated with dysplastic cell transformation
during oral carcinogenesis and is required for cancer cell
replication and survival. Br J Cancer. 96:126–133. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Siddique HR and Saleem M: Role of BMI1, a
stem cell factor, in cancer recurrence and chemoresistance:
preclinical and clinical evidences. Stem Cells. 30:372–378. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Siddique HR, Parray A, Tarapore RS, et al:
BMI1 polycomb group protein acts as a master switch for growth and
death of tumor cells: regulates TCF4-transcriptional factor-induced
BCL2 signaling. PLoS One. 8:e606642013. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Wolters T, Vissers KJ, Bangma CH, Schröder
FH and van Leenders GJ: The value of EZH2, p27kip1,
BMI-1 and MIB-1 on biopsy specimens with low-risk prostate cancer
in selecting men with significant prostate cancer at prostatectomy.
BJU Int. 106:280–286. 2010.PubMed/NCBI
|
|
34
|
van Leenders GJ, Dukers D, Hessels D, et
al: Polycomb-group oncogenes EZH2, BMI1, and RING1 are
overexpressed in prostate cancer with adverse pathologic and
clinical features. Eur Urol. 52:455–463. 2007.PubMed/NCBI
|
|
35
|
Jacobs JJ, Kieboom K, Marino S, DePinho RA
and van Lohuizen M: The oncogene and Polycomb-group gene
bmi-1 regulates cell proliferation and senescence through
the ink4a locus. Nature. 397:164–168. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Crea F, Duhagon Serrat MA, Hurt EM, Thomas
SB, Danesi R and Farrar WL: BMI1 silencing enhances docetaxel
activity and impairs antioxidant response in prostate cancer. Int J
Cancer. 128:1946–1954. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Thiery JP, Acloque H, Huang RY and Nieto
MA: Epithelial-mesenchymal transitions in development and disease.
Cell. 139:871–890. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Sánchez-Tilló E, Lázaro A, Torrent R, et
al: ZEB1 represses E-cadherin and induces an EMT by recruiting the
SWI/SNF chromatin-remodeling protein BRG1. Oncogene. 29:3490–3500.
2010.PubMed/NCBI
|
|
39
|
Yang MH, Hsu DS, Wang HW, et al: Bmi1 is
essential in Twist1-induced epithelial-mesenchymal transition. Nat
Cell Biol. 12:982–992. 2010. View
Article : Google Scholar : PubMed/NCBI
|
