|
1
|
Tsubura A, Lai YC, Kuwata M, Uehara N and
Yoshizawa K: Anticancer effects of garlic and garlic-derived
compounds for breast cancer control. Anticancer Agents Med Chem.
11:249–253. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Bianchini F and Vainio H: Allium
vegetables and organosulfur compounds: do they help prevent cancer?
Environ Health Perspect. 109:893–902. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Milner JA: Mechanisms by which garlic and
allyl sulfur compounds suppress carcinogen bioactivation. Garlic
and carcinogenesis. Adv Exp Med Biol. 492:69–81. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Kim JY and Kwon O: Garlic intake and
cancer risk: an analysis using the Food and Drug Administration’s
evidence-based review system for the scientific evaluation of
health claims. Am J Clin Nutr. 89:257–264. 2009.
|
|
5
|
Fleischauer AT and Arab L: Garlic and
cancer: a critical review of the epidemiologic literature. J Nutr.
131:S1032–S1040. 2001.PubMed/NCBI
|
|
6
|
Ngo SN, Williams DB, Cobiac L and Head RJ:
Does garlic reduce risk of colorectal cancer? A systematic review.
J Nutr. 137:2264–2269. 2007.PubMed/NCBI
|
|
7
|
Sperka T, Wang J and Rudolph KL: DNA
damage checkpoints in stem cells, ageing and cancer. Nat Rev Mol
Cell Biol. 13:579–590. 2012. View
Article : Google Scholar : PubMed/NCBI
|
|
8
|
Canavese M, Santo L and Raje N: Cyclin
dependent kinases in cancer: potential for therapeutic
intervention. Cancer Biol Ther. 13:451–457. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Modem S, Dicarlo SE and Reddy TR: Fresh
garlic extract induces growth arrest and morphological
differentiation of MCF7 breast cancer cells. Genes Cancer.
3:177–186. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Lund T, Stokke T, Olsen ØE and Fodstad Ø:
Garlic arrests MDA-MB-435 cancer cells in mitosis, phosphorylates
the proapoptotic BH3-only protein BimEL and induces apoptosis. Br J
Cancer. 92:1773–1781. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Frantz DJ, Hughes BG, Nelson DR, Murray BK
and Christensen MJ: Cell cycle arrest and differential gene
expression in HT-29 cells exposed to an aqueous garlic extract.
Nutr Cancer. 38:255–264. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Su CC, Chen GW, Tan TW, Lin JG and Chung
JG: Crude extract of garlic induced caspase-3 gene expression
leading to apoptosis in human colon cancer cells. In Vivo.
20:85–90. 2006.PubMed/NCBI
|
|
13
|
Kim HJ, Han MH, Kim GY, Choi YW and Choi
YH: Hexane extracts of garlic cloves induce apoptosis through the
generation of reactive oxygen species in Hep3B human
hepatocarcinoma cells. Oncol Rep. 28:1757–1763. 2012.
|
|
14
|
Na HK, Kim EH, Choi MA, Park JM, Kim DH
and Surh YJ: Diallyl trisulfide induces apoptosis in human breast
cancer cells through ROS-mediated activation of JNK and AP-1.
Biochem Pharmacol. 84:1241–1250. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Xiao D, Herman-Antosiewicz A, Antosiewicz
J, Xiao H, Brisson M, Lazo JS and Singh SV: Diallyl
trisulfide-induced G(2)-M phase cell cycle arrest in human prostate
cancer cells is caused by reactive oxygen species-dependent
destruction and hyperphosphorylation of Cdc 25C. Oncogene.
24:6256–6268. 2005. View Article : Google Scholar
|
|
16
|
Di X, Andrews DM, Tucker CJ, Yu L, Moore
AB, Zheng X, Castro L, Hermon T, Xiao H and Dixon D: A high
concentration of genistein down-regulates activin A, Smad3 and
other TGF-β pathway genes in human uterine leiomyoma cells. Exp Mol
Med. 44:281–292. 2012.PubMed/NCBI
|
|
17
|
Medema RH and Macůrek L: Checkpoint
control and cancer. Oncogene. 31:2601–2613. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Rozenblat S, Grossman S, Bergman M,
Gottlieb H, Cohen Y and Dovrat S: Induction of G2/M arrest and
apoptosis by sesquiterpene lactones in human melanoma cell lines.
Biochem Pharmacol. 75:369–382. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Lim S and Kaldis P: Cdks, cyclins and
CKIs: roles beyond cell cycle regulation. Development.
140:3079–3093. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Sánchez I and Dynlacht BD: New insights
into cyclins, CDKs, and cell cycle control. Semin Cell Dev Biol.
16:311–321. 2005.PubMed/NCBI
|
|
21
|
Jeong AL and Yang Y: PP2A function toward
mitotic kinases and substrates during the cell cycle. BMB Rep.
46:289–294. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Lindqvist A, Rodríguez-Bravo V and Medema
RH: The decision to enter mitosis: feedback and redundancy in the
mitotic entry network. J Cell Biol. 185:193–202. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Porter LA and Donoghue DJ: Cyclin B1 and
CDK1: nuclear localization and upstream regulators. Prog Cell Cycle
Res. 5:335–347. 2003.PubMed/NCBI
|
|
24
|
Abbas T and Dutta A: p21 in cancer:
intricate networks and multiple activities. Nat Rev Cancer.
9:400–414. 2009. View
Article : Google Scholar : PubMed/NCBI
|
|
25
|
Brown L, Boswell S, Raj L and Lee SW:
Transcriptional targets of p53 that regulate cellular
proliferation. Crit Rev Eukaryot Gene Expr. 17:73–85. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Danova M, Giordano M, Mazzini G and
Riccardi A: Expression of p53 protein during the cell cycle
measured by flow cytometry in human leukemia. Leuk Res. 14:417–422.
1990. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Burz C, Berindan-Neagoe I, Balacescu O and
Irimie A: Apoptosis in cancer: key molecular signaling pathways and
therapy targets. Acta Oncol. 48:811–821. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Fulda S and Debatin KM: Extrinsic versus
intrinsic apoptosis pathways in anticancer chemotherapy. Oncogene.
25:4798–4811. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Liu Y, Bertram CC, Shi Q and Zinkel SS:
Proapoptotic Bid mediates the Atr-directed DNA damage response to
replicative stress. Cell Death Differ. 18:841–852. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Shamas-Din A, Brahmbhatt H, Leber B and
Andrews DW: BH3-only proteins: Orchestrators of apoptosis. Biochim
Biophys Acta. 1813:508–520. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Tewari M, Quan LT, O’Rourke K, Desnoyers
S, Zeng Z, Beidler DR, Poirier GG, Salvesen GS and Dixit VM:
Yama/CPP32 beta, a mammalian homolog of CED-3, is a
CrmA-inhibitable protease that cleaves the death substrate
poly(ADP-ribose) polymerase. Cell. 81:801–809. 1995. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Low IC, Kang J and Pervaiz S: Bcl-2: a
prime regulator of mitochondrial redox metabolism in cancer cells.
Antioxid Redox Signal. 15:2975–2987. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Galluzzi L, Larochette N, Zamzami N and
Kroemer G: Mitochondria as therapeutic targets for cancer
chemotherapy. Oncogene. 25:4812–4830. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
LaCasse EC, Mahoney DJ, Cheung HH,
Plenchette S, Baird S and Korneluk RG: IAP-targeted therapies for
cancer. Oncogene. 27:6252–6275. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Hunter AM, LaCasse EC and Korneluk RG: The
inhibitors of apoptosis (IAPs) as cancer targets. Apoptosis.
12:1543–1568. 2007. View Article : Google Scholar : PubMed/NCBI
|
