|
1
|
Steigman SA, Kunisaki SM, Wilkins-Haug L,
Takoudes TC and Fauza DO: Optical properties of human amniotic
fluid: implications for videofetoscopic surgery. Fetal Diagn Ther.
27:87–90. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Kunisaki C, Makino H, Kimura J, et al:
Impact of lymphovascular invasion in patients with stage I gastric
cancer. Surgery. 147:204–211. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Arunkumar R, Sharmila G, Elumalai P, et
al: Effect of diallyl disulfide on insulin-like growth factor
signaling molecules involved in cell survival and proliferation of
human prostate cancer cells in vitro and in silico approach through
docking analysis. Phytomedicine. 19:912–923. 2012. View Article : Google Scholar
|
|
4
|
Lai KC, Kuo CL, Ho HC, et al: Diallyl
sulfide, diallyl disulfide and diallyl trisulfide affect drug
resistant gene expression in colo 205 human colon cancer cells in
vitro and in vivo. Phytomedicine. 19:625–630. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Yi L, Ji XX, Tan H, et al: Involvement of
Mcl1 in diallyl disulfide-induced G2/M cell cycle arrest in HL-60
cells. Oncol Rep. 27:1911–1917. 2012.PubMed/NCBI
|
|
6
|
Ling H, Zhang LY, Su Q, et al: Erk is
involved in the differentiation induced by diallyl disulfide in the
human gastric cancer cell line MGC803. Cell Mol Biol Lett.
11:408–423. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Yi L and Su Q: Molecular mechanisms for
the anti-cancer effects of diallyl disulfide. Food Chem Toxicol.
57:362–370. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Song JD, Lee SK, Kim KM, et al: Molecular
mechanism of diallyl disulfide in cell cycle arrest and apoptosis
in HCT-116 colon cancer cells. J Biochem Mol Toxicol. 23:71–79.
2009. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Liao QJ, Su J, He J, Song Y, Tang HL and
Su Q: Effect of diallyl disulfide on cell cycle arrest of human
colon cancer SW480 cells. Ai Zheng. 28:138–141. 2009.PubMed/NCBI
|
|
10
|
Dasgupta P and Bandyopadhyay SS: Role of
di-allyl disulfide, a garlic component in NF-κB mediated transient
G2-M phase arrest and apoptosis in human leukemic cell-lines. Nutr
Cancer. 65:611–622. 2013.PubMed/NCBI
|
|
11
|
Arunkumar A, Vijayababu MR, Srinivasan N,
Aruldhas MM and Arunakaran J: Garlic compound, diallyl disulfide
induces cell cycle arrest in prostate cancer cell line PC-3. Mol
Cell Biochem. 288:107–113. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Knowles LM and Milner JA: Diallyl
disulfide inhibits p34cdc2 kinase activity through
changes in complex formation and phosphorylation. Carcinogenesis.
21:1129–1134. 2000.PubMed/NCBI
|
|
13
|
Ashra H and Rao KV: Elevated
phosphorylation of Chk1 and decreased phosphorylation of Chk2 are
associated with abrogation of G2/M checkpoint control during
transformation of Syrian hamster embryo (SHE) cells by Malachite
green. Cancer Lett. 237:188–198. 2006. View Article : Google Scholar
|
|
14
|
Xiang SL, Xiao XL, Ling H, et al:
Antitumor effect of diallyl disulfide on human gastric cancer
MGC803 cells xenograft in nude mice. Ai Zheng. 24:940–944. 2005.(In
Chinese).
|
|
15
|
Yuan JP, Wang GH, Ling H, et al: Diallyl
disulfide-induced G2/M arrest of human gastric cancer MGC803 cells
involves activation of p38 MAP kinase pathways. World J
Gastroenterol. 10:2731–2734. 2004.
|
|
16
|
Ling H, Wen L, Ji XX, et al: Growth
inhibitory effect and Chk1-dependent signaling involved in
G2/M arrest on human gastric cancer cells induced by
diallyl disulfide. Braz J Med Biol Res. 43:271–278. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Shioya K, Michaux C, Kuenne C, Hain T, et
al: Genome-wide identification of small RNAs in the opportunistic
pathogen Enterococcus faecalis V583. PLoS One. 6:e239482011.
View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Dickson MA and Schwartz GK: Development of
cell-cycle inhibitors for cancer therapy. Curr Oncol. 16:36–43.
2009.PubMed/NCBI
|
|
19
|
Call JA, Eckhardt SG and Camidge DR:
Targeted manipulation of apoptosis in cancer treatment. Lancet
Oncol. 9:1002–1011. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Bucher N and Britten CD: G2 checkpoint
abrogation and checkpoint kinase-1 targeting in the treatment of
cancer. Br J Cancer. 98:523–528. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Gong QF, Liu EH, Xin R, Huang X and Gao N:
2ME and 2OHE2 exhibit growth inhibitory effects and cell cycle
arrest at G2/M in RL95-2 human endometrial cancer cells through
activation of p53 and Chk1. Mol Cell Biochem. 352:221–230. 2011.
View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Wu J, Lai G, Wan F, et al: Knockdown of
checkpoint kinase 1 is associated with the increased
radiosensitivity of glioblastoma stem-like cells. Tohoku J Exp Med.
226:267–274. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Hu W, Zong Q, John-Baptiste A and Jessen
B: Transient knock down of checkpoint kinase 1 in hematopoietic
progenitors is linked to bone marrow toxicity. Toxicol Lett.
204:141–147. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Zeng X and Kinsella TJ: BNIP3 is essential
for mediating 6-thioguanine- and 5-fluorouracil-induced autophagy
following DNA mismatch repair processing. Cell Res. 20:665–675.
2010. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Hsu SC, Yu CC, Yang JS, et al: A novel
synthetic 2-(3-methoxyphenyl)- 6,7-methylenedioxoquinolin-4-one
arrests the G2/M phase arrest via Cdc25c and induces apoptosis
through caspase- and mitochondria-dependent pathways in TSGH8301
human bladder cancer cells. Int J Oncol. 40:731–738. 2012.
|
|
26
|
Pabla N, Bhatt K and Dong Z: Checkpoint
kinase 1 (Chk1)-short is a splice variant and endogenous inhibitor
of Chk1 that regulates cell cycle and DNA damage checkpoints. Proc
Natl Acad Sci USA. 109:197–202. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Niida H, Murata K, Shimada M, et al:
Cooperative functions of Chk1 and Chk2 reduce tumour susceptibility
in vivo. EMBO J. 29:3558–3570. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Soule BP, Simone NL, DeGraff WG, Choudhuri
R, Cook JA and Mitchell JB: Loratadine dysregulates cell cycle
progression and enhances the effect of radiation in human tumor
cell lines. Radiat Oncol. 5:82010. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Moon DO, Kim MO, Nam TJ, Kim SK, Choi YH
and Kim GY: Pectenotoxin-2 induces G2/M phase cell cycle
arrest in human breast cancer cells via ATM and Chk1/2-mediated
phosphorylation of cdc25C. Oncol Rep. 24:271–276. 2010.PubMed/NCBI
|
|
30
|
Yan Y, Cao PT, Greer PM, et al: Protein
phosphatase 2A has an essential role in the activation of
γ-irradiation-induced G2/M checkpoint response. Oncogene.
29:4317–4329. 2010.
|
|
31
|
Rong JJ, Hu R, Song XM, et al: Gambogic
acid triggers DNA damage signaling that induces
p53/p21Waf1/CIP1 activation through the ATR-Chk1
pathway. Cancer Lett. 296:55–64. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Bonnet J, Mayonove P and Morris MC:
Differential phosphorylation of Cdc25C phosphatase in mitosis.
Biochem Biophys Res Commun. 370:483–488. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Reinhardt HC and Yaffe MB: Kinases that
control the cell cycle in response to DNA damage: Chk1, Chk2, and
MK2. Curr Opin Cell Biol. 21:245–255. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Zegerman P and Diffley JF: DNA replication
as a target of the DNA damage checkpoint. DNA Repair. 8:1077–1088.
2009. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Shi L, Chen J, Wang YY, et al: Gossypin
induces G2/M arrest in human malignant glioma U251 cells by the
activation of Chk1/Cdc25C pathway. Cell Mol Neurobiol. 32:289–296.
2012. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Aressy B and Ducommun B: Cell cycle
control by the CDC25 phosphatases. Anticancer Agents Med Chem.
8:818–824. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Chaudhary P, Sharma R, Sahu M, Vishwanatha
JK, Awasthi S and Awasthi YC: 4-Hydroxynonenal induces
G2/M phase cell cycle arrest by activation of the ataxia
telangiectasia mutated and Rad3-related protein (ATR)/checkpoint
kinase 1 (Chk1) signaling pathway. J Biol Chem. 288:20532–20546.
2013.PubMed/NCBI
|
|
38
|
Duan J, Yu Y, Li Y, et al: Toxic effect of
silica nanoparticles on endothelial cells through DNA damage
response via Chk1-dependent G2/M checkpoint. PLoS One.
8:e620872013. View Article : Google Scholar : PubMed/NCBI
|
|
39
|
Matsuoka S, Ballif BA, Smogorzewska A, et
al: ATM and ATR substrate analysis reveals extensive protein
networks responsive to DNA damage. Science. 316:1160–1166. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
40
|
Nishida H, Tatewaki N, Nakajima Y, et al:
Inhibition of ATR protein kinase activity by schisandrin B in DNA
damage response. Nucleic Acids Res. 37:5678–5689. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
41
|
Furusawa Y, Iizumi T, Fujiwara Y, et al:
Inhibition of checkpoint kinase 1 abrogates G2/M checkpoint
activation and promotes apoptosis under heat stress. Apoptosis.
17:102–112. 2012. View Article : Google Scholar : PubMed/NCBI
|
