|
1
|
Kweon KH, Lee CR, Jung SJ, Ban EJ, Kang
SW, Jeong JJ, Nam KH, Jo YS, Lee J and Chung WY: Sirt1 induction
confers resistance to etoposide-induced genotoxic apoptosis in
thyroid cancers. Int J Oncol. 45:2065–2075. 2014.PubMed/NCBI
|
|
2
|
Xing Y, Luo DY, Long MY, Zeng SL and Li
HH: High ALDH1A1 expression correlates with poor survival in
papillary thyroid carcinoma. World J Surg Oncol. 12:292014.
View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Tang C, Yang L, Wang N, Li L, Xu M, Chen
GG and Liu ZM: High expression of GPER1, EGFR and CXCR1 is
associated with lymph node metastasis in papillary thyroid
carcinoma. Int J Clin Exp Pathol. 7:3213–3223. 2014.PubMed/NCBI
|
|
4
|
Minna E, Romeo P, De Cecco L, Dugo M,
Cassinelli G, Pilotti S, Degl'Innocenti D, Lanzi C, Casalini P,
Pierotti MA, et al: miR-199a-3p displays tumor suppressor functions
in papillary thyroid carcinoma. Oncotarget. 5:2513–2528.
2014.PubMed/NCBI
|
|
5
|
Polyak K, Xia Y, Zweier JL, Kinzler KW and
Vogelstein B: A model for p53-induced apoptosis. Nature.
389:300–305. 1997. View
Article : Google Scholar : PubMed/NCBI
|
|
6
|
Contente A, Dittmer A, Koch MC, Roth J and
Dobbelstein M: A polymorphic microsatellite that mediates induction
of PIG3 by p53. Nat Genet. 30:315–320. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Porté S, Valencia E, Yakovtseva EA, Borràs
E, Shafqat N, Debreczeny JE, Pike AC, Oppermann U, Farrés J, Fita
I, et al: Three-dimensional structure and enzymatic function of
proapoptotic human p53-inducible quinone oxidoreductase PIG3. J
Biol Chem. 284:17194–17205. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Lee JH, Kang Y, Khare V, Jin ZY, Kang MY,
Yoon Y, Hyun JW, Chung MH, Cho SI, Jun JY, et al: The p53-inducible
gene 3 (PIG3) contributes to early cellular response to DNA damage.
Oncogene. 29:1431–1450. 2010. View Article : Google Scholar
|
|
9
|
Gorgoulis VG, Liloglou T, Sigala F,
Korkolis D, Yannoukakos D, Papalambros E, Asimacopoulos PJ,
Papavassiliou AG and Kotsinas A: Absence of association with cancer
risk and low frequency of alterations at a p53 responsive PIG3 gene
polymorphism in breast and lung carcinomas. Mutat Res. 556:143–150.
2004. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Ito M, Nishiyama H, Watanabe J, Kawanishi
H, Takahashi T, Kamoto T, Habuchi T and Ogawa O: Association of the
PIG3 promoter polymorphism with invasive bladder cancer in a
Japanese population. Jpn J Clin Oncol. 36:116–120. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Guan X, Liu Z, Wang L, Wang LE, Sturgis EM
and Wei Q: Functional repeats (TGYCC)n in the p53-inducible gene 3
(PIG3) promoter and susceptibility to squamous cell carcinoma of
the head and neck. Carcinogenesis. 34:812–817. 2013. View Article : Google Scholar :
|
|
12
|
Kotsinas A, Pateras IS, Galanos PS,
Karamouzis MV, Sfikakis PP and Gorgoulis VG: Why is p53-inducible
gene 3 rarely affected in cancer? Oncogene. 29:52202010. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Li B, Shang ZF, Yin JJ, Xu QZ, Liu XD,
Wang Y, Zhang SM, Guan H and Zhou PK: PIG3 functions in DNA damage
response through regulating DNA-PKcs homeostasis. Int J biol Sci.
9:425–434. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Xie P, Tian C, An L, Nie J, Lu K, Xing G,
Zhang L and He F: Histone methyltransferase protein SETD2 interacts
with p53 and selectively regulates its downstream genes. Cell
Signal. 20:1671–1678. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Kryston TB, Georgiev AB, Pissis P and
Georgakilas AG: Role of oxidative stress and DNA damage in human
carcinogenesis. Mutat Res. 711:193–201. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Moreli JB, Santos JH, Rocha CR, Damasceno
DC, Morceli G, Rudge MV, Bevilacqua E and Calderon IM: DNA damage
and its cellular response in mother and fetus exposed to
hyperglycemic environment. Biomed Res Int. 2014:6767582014.
View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Halazonetis TD, Gorgoulis VG and Bartek J:
An oncogene-induced DNA damage model for cancer development.
Science. 319:1352–1355. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Chambers JT, Carcangiu ML, Voynick IM and
Schwartz PE: Immunohistochemical evaluation of estrogen and
progesterone receptor content in 183 patients with endometrial
carcinoma. Part II: Correlation between biochemical and
immunohistochemical methods and survival. Am J Clin Pathol.
94:255–260. 1990.PubMed/NCBI
|
|
19
|
Flatt PM, Polyak K, Tang LJ, Scatena CD,
Westfall MD, Rubinstein LA, Yu J, Kinzler KW, Vogelstein B, Hill
DE, et al: p53-dependent expression of PIG3 during proliferation,
genotoxic stress, and reversible growth arrest. Cancer Lett.
156:63–72. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Hanahan D and Weinberg RA: Hallmarks of
cancer: The next generation. Cell. 144:646–674. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Ivanova D, Bakalova R, Lazarova D, Gadjeva
V and Zhelev Z: The impact of reactive oxygen species on anticancer
therapeutic strategies. Adv Clin Exp Med. 22:899–908. 2013.
|
|
22
|
Kotsinas A, Aggarwal V, Tan EJ, Levy B and
Gorgoulis VG: PIG3: A novel link between oxidative stress and DNA
damage response in cancer. Cancer Lett. 327:97–102. 2012.
View Article : Google Scholar
|
|
23
|
Liu R, Liu D, Trink E, Bojdani E, Ning G
and Xing M: The Akt-specific inhibitor MK2206 selectively inhibits
thyroid cancer cells harboring mutations that can activate the
PI3K/Akt pathway. J Clin Endocrinol Metab. 96:E577–E585. 2011.
View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Lim HJ, Crowe P and Yang JL: Current
clinical regulation of PI3K/PTEN/Akt/mTOR signalling in treatment
of human cancer. J Cancer Res Clin Oncol. 141:671–689. 2015.
View Article : Google Scholar
|
|
25
|
Korbecki J, Baranowska-Bosiacka I,
Gutowska I and Chlubek D: The effect of reactive oxygen species on
the synthesis of prostanoids from arachidonic acid. J Physiol
Pharmacol. 64:409–421. 2013.PubMed/NCBI
|
|
26
|
Kumar B, Koul S, Khandrika L, Meacham RB
and Koul HK: Oxidative stress is inherent in prostate cancer cells
and is required for aggressive phenotype. Cancer Res. 68:1777–1785.
2008. View Article : Google Scholar : PubMed/NCBI
|
