|
1
|
Torre LA, Bray F, Siegel RL, Ferlay J,
Lortet-Tieulent J and Jemal A: Global cancer statistics, 2012. CA
Cancer J Clin. 65:87–108. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Piazuelo MB and Correa P: Gastric cancer:
Overview. Colomb Med (Cali). 44:192–201. 2013.
|
|
3
|
Steeg PS: Metastasis suppressors alter the
signal transduction of cancer cells. Nat Rev Cancer. 3:55–63. 2003.
View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Kalluri R and Weinberg RA: The basics of
epithelial-mesenchymal transition. J Clin Invest. 119:1420–1428.
2009. View
Article : Google Scholar : PubMed/NCBI
|
|
5
|
Thiery JP, Acloque H, Huang RY and Nieto
MA: Epithelial-mesenchymal transitions in development and disease.
Cell. 139:871–890. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Thiery JP and Sleeman JP: Complex networks
orchestrate epithelial-mesenchymal transitions. Nat Rev Mol Cell
Biol. 7:131–142. 2006. View
Article : Google Scholar : PubMed/NCBI
|
|
7
|
Grego-Bessa J, Díez J, Timmerman L and de
la Pompa JL: Notch and epithelial-mesenchyme transition in
development and tumor progression: Another turn of the screw. Cell
Cycle. 3:718–721. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Kang Y and Massagué J:
Epithelial-mesenchymal transitions: Twist in development and
metastasis. Cell. 118:277–279. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Fuxe J, Vincent T and Garcia de Herreros
A: Transcriptional crosstalk between TGF-β and stem cell pathways
in tumor cell invasion: Role of EMT promoting Smad complexes. Cell
Cycle. 9:2363–2374. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Li HX, Han M, Bernier M, Zheng B, Sun SG,
Su M, Zhang R, Fu JR and Wen JK: Krüppel-like factor 4 promotes
differentiation by transforming growth factor-beta
receptor-mediated Smad and p38 MAPK signaling in vascular smooth
muscle cells. J Biol Chem. 285:17846–17856. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Overdier DG, Porcella A and Costa RH: The
DNA-binding specificity of the hepatocyte nuclear factor 3/forkhead
domain is influenced by amino-acid residues adjacent to the
recognition helix. Mol Cell Biol. 14:2755–2766. 1994. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Hoggatt AM, Kriegel AM, Smith AF and
Herring BP: Hepatocyte nuclear factor-3 homologue 1 (HFH-1)
represses transcription of smooth muscle-specific genes. J Biol
Chem. 275:31162–31170. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Feuerborn A, Srivastava PK, Küffer S,
Grandy WA, Sijmonsma TP, Gretz N, Brors B and Gröne HJ: The
Forkhead factor FoxQ1 influences epithelial differentiation. J Cell
Physiol. 226:710–719. 2011. View Article : Google Scholar
|
|
14
|
Qiao Y, Jiang X, Lee ST, Karuturi RK, Hooi
SC and Yu Q: FOXQ1 regulates epithelial-mesenchymal transition in
human cancers. Cancer Res. 71:3076–3086. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Zhang H, Meng F, Liu G, Zhang B, Zhu J, Wu
F, Ethier SP, Miller F and Wu G: Forkhead transcription factor
foxq1 promotes epithelial-mesenchymal transition and breast cancer
metastasis. Cancer Res. 71:1292–1301. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Sehrawat A, Kim SH, Vogt A and Singh SV:
Suppression of FOXQ1 in benzyl isothiocyanate-mediated inhibition
of epithelial-mesenchymal transition in human breast cancer cells.
Carcinogenesis. 34:864–873. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Kaneda H, Arao T, Tanaka K, Tamura D,
Aomatsu K, Kudo K, Sakai K, De Velasco MA, Matsumoto K, Fujita Y,
et al: FOXQ1 is overexpressed in colorectal cancer and enhances
tumorigenicity and tumor growth. Cancer Res. 70:2053–2063. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Feng J, Zhang X, Zhu H, Wang X, Ni S and
Huang J: FoxQ1 overexpression influences poor prognosis in
non-small cell lung cancer, associates with the phenomenon of EMT.
PLoS One. 7:e399372012. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Feng J, Xu L, Ni S, Gu J, Zhu H, Wang H,
Zhang S, Zhang W and Huang J: Involvement of FoxQ1 in NSCLC through
regulating EMT and increasing chemosensitivity. Oncotarget.
5:9689–9702. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Wang W, He S, Ji J, Huang J, Zhang S and
Zhang Y: The prognostic significance of FOXQ1 oncogene
overexpression in human hepatocellular carcinoma. Pathol Res Pract.
209:353–358. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Xia L, Huang W, Tian D, Zhang L, Qi X,
Chen Z, Shang X, Nie Y and Wu K: Forkhead box Q1 promotes
hepatocellular carcinoma metastasis by transactivating ZEB2 and
VersicanV1 expression. Hepatology. 59:958–973. 2014. View Article : Google Scholar
|
|
22
|
Pei Y, Wang P, Liu H, He F and Ming L:
FOXQ1 promotes esophageal cancer proliferation and metastasis by
negatively modulating CDH1. Biomed Pharmacother. 74:89–94. 2015.
View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Peng X, Luo Z, Kang Q, Deng D, Wang Q,
Peng H, Wang S and Wei Z: FOXQ1 mediates the crosstalk between
TGF-β and Wnt signaling pathways in the progression of colorectal
cancer. Cancer Biol Ther. 16:1099–1109. 2015. View Article : Google Scholar
|
|
24
|
Liang SH, Yan XZ, Wang BL, Jin HF, Yao LP,
Li YN, Chen M, Nie YZ, Wang X, Guo XG, et al: Increased expression
of FOXQ1 is a prognostic marker for patients with gastric cancer.
Tumour Biol. 34:2605–2609. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Xiang XJ, Deng J, Liu YW, Wan LY, Feng M,
Chen J and Xiong JP: MiR-1271 inhibits cell proliferation, invasion
and EMT in gastric cancer by targeting FOXQ1. Cell Physiol Biochem.
36:1382–1394. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Savagner P, Boyer B, Valles AM, Jouanneau
J and Thiery JP: Modulations of the epithelial phenotype during
embryogenesis and cancer progression. Cancer Treat Res. 71:229–249.
1994. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Kudo-Saito C, Shirako H, Takeuchi T and
Kawakami Y: Cancer metastasis is accelerated through
immunosuppression during Snail-induced EMT of cancer cells. Cancer
Cell. 15:195–206. 2009. View Article : Google Scholar : PubMed/NCBI
|
