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Oncology Reports
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Print ISSN: 1021-335X Online ISSN: 1791-2431
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Jan-Feb 2000 Volume 7 Issue 1

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Journals

International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

Molecular Medicine Reports

Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

Oncology Reports

Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

Oncology Letters

Oncology Letters

Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

Biomedical Reports

Biomedical Reports

Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

Molecular and Clinical Oncology

Molecular and Clinical Oncology

International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

World Academy of Sciences Journal

World Academy of Sciences Journal

Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

International Journal of Functional Nutrition

International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

International Journal of Epigenetics

International Journal of Epigenetics

Publishes open-access research on using epigenetics to advance understanding and treatment of human disease.

Medicine International

Medicine International

An International Open Access Journal Devoted to General Medicine.

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Article

Lack of effect by prostaglandin F2alpha on the proliferation of the HCT-8 and HT-29 human adenocarcinoma cell lines.

  • Authors:
    • G Cassano
    • G Gasparre
    • F Susca
    • C Lippe
    • G Guanti

  • View Affiliations / Copyright

    Affiliations: Dipartimento di Fisiologia Generale ed Ambientale, I-70124 Bari, Italy.
  • Pages: 183-189
    |
    Published online on: January 1, 2000
       https://doi.org/10.3892/or.7.1.183
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Abstract

A variety of studies have supported the finding that regular intake of aspirin or non-steroidal anti-inflammatory drugs can affect colorectal cancer carcinogenesis by decreasing the synthesis of prostaglandins (PGs). We report that PG F2alpha, in the presence of indomethacin, did not stimulate the proliferation in HCT-8 and HT-29 human colon adenocarcinoma cells. Moreover, in both cell lines fluprostenol, a specific agonist of FP receptors, did not increase intracellular Ca2+ concentration, monitored with the fluorescent dye fura-2. These results indicate that in HCT-8 and HT-29 cells: i) proliferation is not sensitive to PG F2alpha; ii) functional FP receptors are absent. Therefore, either PG F2alpha is not necessarily involved in the proliferation of colorectal mucosa or cell lines other than HCT-8 and HT-29 should be used to assess the role played by PG F2alpha in promoting cell proliferation in colon cancer.

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Copy and paste a formatted citation
Spandidos Publications style
Cassano G, Gasparre G, Susca F, Lippe C and Guanti G: Lack of effect by prostaglandin F2alpha on the proliferation of the HCT-8 and HT-29 human adenocarcinoma cell lines.. Oncol Rep 7: 183-189, 2000.
APA
Cassano, G., Gasparre, G., Susca, F., Lippe, C., & Guanti, G. (2000). Lack of effect by prostaglandin F2alpha on the proliferation of the HCT-8 and HT-29 human adenocarcinoma cell lines.. Oncology Reports, 7, 183-189. https://doi.org/10.3892/or.7.1.183
MLA
Cassano, G., Gasparre, G., Susca, F., Lippe, C., Guanti, G."Lack of effect by prostaglandin F2alpha on the proliferation of the HCT-8 and HT-29 human adenocarcinoma cell lines.". Oncology Reports 7.1 (2000): 183-189.
Chicago
Cassano, G., Gasparre, G., Susca, F., Lippe, C., Guanti, G."Lack of effect by prostaglandin F2alpha on the proliferation of the HCT-8 and HT-29 human adenocarcinoma cell lines.". Oncology Reports 7, no. 1 (2000): 183-189. https://doi.org/10.3892/or.7.1.183
Copy and paste a formatted citation
x
Spandidos Publications style
Cassano G, Gasparre G, Susca F, Lippe C and Guanti G: Lack of effect by prostaglandin F2alpha on the proliferation of the HCT-8 and HT-29 human adenocarcinoma cell lines.. Oncol Rep 7: 183-189, 2000.
APA
Cassano, G., Gasparre, G., Susca, F., Lippe, C., & Guanti, G. (2000). Lack of effect by prostaglandin F2alpha on the proliferation of the HCT-8 and HT-29 human adenocarcinoma cell lines.. Oncology Reports, 7, 183-189. https://doi.org/10.3892/or.7.1.183
MLA
Cassano, G., Gasparre, G., Susca, F., Lippe, C., Guanti, G."Lack of effect by prostaglandin F2alpha on the proliferation of the HCT-8 and HT-29 human adenocarcinoma cell lines.". Oncology Reports 7.1 (2000): 183-189.
Chicago
Cassano, G., Gasparre, G., Susca, F., Lippe, C., Guanti, G."Lack of effect by prostaglandin F2alpha on the proliferation of the HCT-8 and HT-29 human adenocarcinoma cell lines.". Oncology Reports 7, no. 1 (2000): 183-189. https://doi.org/10.3892/or.7.1.183
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