1
|
Shaw TJ and Martin P: Wound repair at a
glance. J Cell Sci. 122:3209–3213. 2009. View Article : Google Scholar : PubMed/NCBI
|
2
|
Gurtner GC, Werner S, Barrandon Y and
Longaker MT: Wound repair and regeneration. Nature. 453:314–321.
2008. View Article : Google Scholar : PubMed/NCBI
|
3
|
Brem H and Tomic-Canic M: Cellular and
molecular basis of wound healing in diabetes. J Clin Invest.
117:1219–1222. 2007. View
Article : Google Scholar : PubMed/NCBI
|
4
|
Velazquez OC: Angiogenesis and
vasculogenesis: Inducing the growth of new blood vessels and wound
healing by stimulation of bone marrow-derived progenitor cell
mobilization and homing. J Vasc Surg. 45:(Suppl A). A39–A47. 2007.
View Article : Google Scholar : PubMed/NCBI
|
5
|
Rafii S and Lyden D: Therapeutic stem and
progenitor cell transplantation for organ vascularization and
regeneration. Nat Med. 9:702–712. 2003. View Article : Google Scholar : PubMed/NCBI
|
6
|
Amadesi S, Reni C, Katare R, Meloni M,
Oikawa A, Beltrami AP, Avolio E, Cesselli D, Fortunato O, Spinetti
G, et al: Role for substance p-based nociceptive signaling in
progenitor cell activation and angiogenesis during ischemia in mice
and in human subjects. Circulation. 125:1774–1786, S1-S19. 2012.
View Article : Google Scholar : PubMed/NCBI
|
7
|
Fadini GP, Miorin M, Facco M, Bonamico S,
Baesso I, Grego F, Menegolo M, de Kreutzenberg SV, Tiengo A,
Agostini C and Avogaro A: Circulating endothelial progenitor cells
are reduced in peripheral vascular complications of type 2 diabetes
mellitus. J Am Coll Cardiol. 45:1449–1457. 2005. View Article : Google Scholar : PubMed/NCBI
|
8
|
Gallagher KA, Liu ZJ, Xiao M, Chen H,
Goldstein LJ, Buerk DG, Nedeau A, Thom SR and Velazquez OC:
Diabetic impairments in NO-mediated endothelial progenitor cell
mobilization and homing are reversed by hyperoxia and SDF-1 alpha.
J Clin Invest. 117:1249–1259. 2007. View
Article : Google Scholar : PubMed/NCBI
|
9
|
Yu FX and Guan KL: The Hippo pathway:
Regulators and regulations. Genes Dev. 27:355–371. 2013. View Article : Google Scholar : PubMed/NCBI
|
10
|
Gumbiner BM and Kim NG: The Hippo-YAP
signaling pathway and contact inhibition of growth. J Cell Sci.
127:709–717. 2014. View Article : Google Scholar : PubMed/NCBI
|
11
|
Hansen CG, Moroishi T and Guan KL: YAP and
TAZ: A nexus for Hippo signaling and beyond. Trends Cell Biol.
25:499–513. 2015. View Article : Google Scholar : PubMed/NCBI
|
12
|
Lee MJ, Byun Ran M, Furutani-Seiki M, Hong
JH and Jung HS: YAP and TAZ regulate skin wound healing. J Invest
Dermatol. 134:518–525. 2014. View Article : Google Scholar : PubMed/NCBI
|
13
|
Hong HS, Lee J, Lee E, Kwon YS, Lee E, Ahn
W, Jiang MH, Kim JC and Son Y: A new role of substance P as an
injury-inducible messenger for mobilization of CD29(+) stromal-like
cells. Nat Med. 15:425–435. 2009. View
Article : Google Scholar : PubMed/NCBI
|
14
|
Kant V, Kumar D, Kumar D, Prasad R, Gopal
A, Pathak NN, Kumar P and Tandan SK: Topical application of
substance P promotes wound healing in streptozotocin-induced
diabetic rats. Cytokine. 73:144–155. 2015. View Article : Google Scholar : PubMed/NCBI
|
15
|
Leal EC, Carvalho E, Tellechea A, Kafanas
A, Tecilazich F, Kearney C, Kuchibhotla S, Auster ME, Kokkotou E,
Mooney DJ, et al: Substance p promotes wound healing in diabetes by
modulating inflammation and macrophage phenotype. Am J Pathol.
185:1638–1648. 2015. View Article : Google Scholar : PubMed/NCBI
|
16
|
Yang L, Di G, Qi X, Qu M, Wang Y, Duan H,
Danielson P, Xie L and Zhou Q: Substance P promotes diabetic
corneal epithelial wound healing through molecular mechanisms
mediated via the neurokinin-1 receptor. Diabetes. 63:4262–4274.
2014. View Article : Google Scholar : PubMed/NCBI
|
17
|
Um J, Jung N, Chin S, Cho Y, Choi S and
Park KS: Substance P enhances EPC mobilization for accelerated
wound healing. Wound Repair Regen. 24:402–410. 2016. View Article : Google Scholar : PubMed/NCBI
|
18
|
Wang LH, Zhou SX, Li RC, Zheng LR, Zhu JH,
Hu SJ and Sun YL: Serum levels of calcitonin gene-related peptide
and substance P are decreased in patients with diabetes mellitus
and coronary artery disease. J Int Med Res. 40:134–140. 2012.
View Article : Google Scholar : PubMed/NCBI
|
19
|
Antezana M, Sullivan SR, Usui M, Gibran N,
Spenny M, Larsen J, Ansel J, Bunnett N and Olerud J: Neutral
endopeptidase activity is increased in the skin of subjects with
diabetic ulcers. J Invest Dermatol. 119:1400–1404. 2002. View Article : Google Scholar : PubMed/NCBI
|
20
|
Baustian C, Hanley S and Ceredig R:
Isolation, selection and culture methods to enhance clonogenicity
of mouse bone marrow derived mesenchymal stromal cell precursors.
Stem Cell Res Ther. 6:1512015. View Article : Google Scholar : PubMed/NCBI
|
21
|
Hinz B: Formation and function of the
myofibroblast during tissue repair. J Invest Dermatol. 127:526–537.
2007. View Article : Google Scholar : PubMed/NCBI
|
22
|
Darby IA, Bisucci T, Hewitson TD and
MacLellan DG: Apoptosis is increased in a model of
diabetes-impaired wound healing in genetically diabetic mice. Int J
Biochem Cell Biol. 29:191–200. 1997. View Article : Google Scholar : PubMed/NCBI
|
23
|
Harvey KF, Zhang X and Thomas DM: The
Hippo pathway and human cancer. Nat Rev Cancer. 13:246–257. 2013.
View Article : Google Scholar : PubMed/NCBI
|
24
|
Asahara T, Murohara T, Sullivan A, Silver
M, van der Zee R, Li T, Witzenbichler B, Schatteman G and Isner JM:
Isolation of putative progenitor endothelial cells for
angiogenesis. Science. 275:964–967. 1997. View Article : Google Scholar : PubMed/NCBI
|