1
|
Toyomura K, Yamaguchi K, Kawamoto H, et
al: Relation of cigarette smoking and alcohol use to colorectal
adenomas by subsite: the self-defense forces health study. Cancer
Sci. 95:72–76. 2004. View Article : Google Scholar : PubMed/NCBI
|
2
|
Robertson DJ, Sandler RS, Haile R, et al:
Fat, fiber, meat and the risk of colorectal adenomas. Am J
Gastroenterol. 100:2789–2795. 2005. View Article : Google Scholar : PubMed/NCBI
|
3
|
Sharma S, Iwasaki M, Kunieda C, et al:
Development of a quantitative food frequency questionnaire for
assessing food, nutrient, and heterocyclic aromatic amines intake
in Japanese Brazilians for a colorectal adenoma case-control study.
Int J Food Sci Nutr. 60(Suppl 7): 128–139. 2009. View Article : Google Scholar
|
4
|
Mansoor-ul-Haq and Faisal N: Familial
adenomatous polyposis. J Coll Physicians Surg Pak. 21:46–48.
2011.
|
5
|
Wilschut JA, Habbema JD, Ramsey SD, Boer
R, Looman CW and van Ballegooijen M: Increased risk of adenomas in
individuals with a family history of colorectal cancer: results of
a meta-analysis. Cancer Causes Control. 21:2287–2293. 2010.
View Article : Google Scholar : PubMed/NCBI
|
6
|
Zhuo W, Wang Y, Zhuo X, et al: CYP1A1 and
GSTM1 polymorphisms and oral cancer risk: association studies via
evidence-based meta-analyses. Cancer Invest. 27:86–95. 2009.
View Article : Google Scholar : PubMed/NCBI
|
7
|
Hein DW: N-acetyltransferase 2 genetic
polymorphism: effects of carcinogen and haplotype on urinary
bladder cancer risk. Oncogene. 25:1649–1658. 2006. View Article : Google Scholar : PubMed/NCBI
|
8
|
Zhou W, Liu G, Thurston SW, et al: Genetic
polymorphisms in N-acetyltransferase-2 and microsomal epoxide
hydrolase, cumulative cigarette smoking, and lung cancer. Cancer
Epidemiol Biomarkers Prev. 11:15–21. 2002.PubMed/NCBI
|
9
|
Bolt HM, Selinski S, Dannappel D,
Blaszkewicz M and Golka K: Re-investigation of the concordance of
human NAT2 phenotypes and genotypes. Arch Toxicol. 79:196–200.
2005. View Article : Google Scholar : PubMed/NCBI
|
10
|
Hein DW and Doll MA: Accuracy of various
human NAT2 SNP genotyping panels to infer rapid, intermediate and
slow acetylator phenotypes. Pharmacogenomics. 13:31–42. 2012.
View Article : Google Scholar : PubMed/NCBI
|
11
|
Taja-Chayeb L, González-Fierro A,
Miguez-Muñoz C, et al: Acetylator status and N-acetyltransferase 2
gene polymorphisms; phenotype-genotype correlation with the
sulfamethazine test. Pharmacogenet Genomics. 21:894–901. 2011.
View Article : Google Scholar : PubMed/NCBI
|
12
|
Munafò MR, Clark TG and Flint J: Assessing
publication bias in genetic association studies: evidence from a
recent meta-analysis. Psychiatry Res. 129:39–44. 2004.PubMed/NCBI
|
13
|
Egger M, Davey Smith G, Schneider M and
Minder C: Bias in meta-analysis detected by a simple, graphical
test. BMJ. 315:629–634. 1997. View Article : Google Scholar : PubMed/NCBI
|
14
|
Raimondi S, Botteri E, Iodice S, Lowenfels
AB and Maisonneuve P: Gene-smoking interaction on colorectal
adenoma and cancer risk: review and meta-analysis. Mutat Res.
670:6–14. 2009. View Article : Google Scholar : PubMed/NCBI
|
15
|
Potter JD, Bigler J, Fosdick L, et al:
Colorectal adenomatous and hyperplastic polyps: smoking and
N-acetyltransferase 2 polymorphisms. Cancer Epidemiol Biomarkers
Prev. 8:69–75. 1999.PubMed/NCBI
|
16
|
Moonen H, Engels L, Kleinjans J and Kok T:
The CYP1A2-164A→C polymorphism (CYP1A2*1F) is associated with the
risk for colorectal adenomas in humans. Cancer Lett. 229:25–31.
2005.
|
17
|
Bunschoten A, Tiemersma E, Schouls L and
Kampman E: Simultaneous determination of polymorphism in
N-acetyltransferase 1 and 2 genes by reverse line blot
hybridization. Anal Biochem. 285:156–162. 2000. View Article : Google Scholar : PubMed/NCBI
|
18
|
Crabtree MD, Fletcher C, Churchman M, et
al: Analysis of candidate modifier loci for the severity of colonic
familial adenomatous polyposis, with evidence for the importance of
the N-acetyl transferases. Gut. 53:271–276. 2004. View Article : Google Scholar : PubMed/NCBI
|
19
|
Lin HJ, Probst-Hensch NM, Hughes NC, et
al: Variants of N-acetyltransferase NAT1 and a case-control study
of colorectal adenomas. Pharmacogenetics. 8:269–281. 1998.
View Article : Google Scholar : PubMed/NCBI
|
20
|
Probst-Hensch NM, Haile RW, Ingles SA, et
al: Acetylation polymorphism and prevalence of colorectal adenomas.
Cancer Res. 55:2017–2020. 1995.PubMed/NCBI
|
21
|
Voskuil DW, Kampman E, Grubben MJ, et al:
Meat consumption and preparation, and genetic susceptibility in
relation to colorectal adenomas. Cancer Lett. 114:309–311. 1997.
View Article : Google Scholar : PubMed/NCBI
|
22
|
Tiemersma EW, Voskuil DW, Bunschoten A, et
al: Risk of colorectal adenomas in relation to meat consumption,
meat preparation, and genetic susceptibility in a Dutch population.
Cancer Causes Control. 15:225–236. 2004. View Article : Google Scholar : PubMed/NCBI
|
23
|
Ishibe N, Sinha R, Hein DW, et al: Genetic
polymorphisms in heterocyclic amine metabolism and risk of
colorectal adenomas. Pharmacogenetics. 12:145–150. 2002. View Article : Google Scholar : PubMed/NCBI
|
24
|
Lang NP, Butler MA, Massengill J, et al:
Rapid metabolic phenotypes for acetyltransferase and cytochrome
P4501A2 and putative exposure to food-borne heterocyclic amines
increase the risk for colorectal cancer or polyps. Cancer Epidemiol
Biomarkers Prev. 3:675–682. 1994.
|
25
|
Moslehi R, Chatterjee N, Church TR, et al:
Cigarette smoking, N-acetyltransferase genes and the risk of
advanced colorectal adenoma. Pharmacogenomics. 7:819–829. 2006.
View Article : Google Scholar : PubMed/NCBI
|
26
|
Probst-Hensch NM, Haile RW, Li DS, et al:
Lack of association between the polyadenylation polymorphism in the
NAT1 (acetyltransferase 1) gene and colorectal adenomas.
Carcinogenesis. 17:2125–2129. 1996. View Article : Google Scholar : PubMed/NCBI
|
27
|
Shin A, Shrubsole MJ, Rice JM, et al: Meat
intake, heterocyclic amine exposure, and metabolizing enzyme
polymorphisms in relation to colorectal polyp risk. Cancer
Epidemiol Biomarkers Prev. 17:320–329. 2008. View Article : Google Scholar : PubMed/NCBI
|
28
|
Tiemersma EW, Bunschoten A, Kok FJ, Glatt
H, de Boer SY and Kampman E: Effect of SULT1A1 and NAT2 genetic
polymorphism on the association between cigarette smoking and
colorectal adenomas. Int J Cancer. 108:97–103. 2004. View Article : Google Scholar : PubMed/NCBI
|
29
|
Wang H, Yamamoto JF, Caberto C, et al:
Genetic variation in the bioactivation pathway for polycyclic
hydrocarbons and heterocyclic amines in relation to risk of
colorectal neoplasia. Carcinogenesis. 32:203–209. 2011. View Article : Google Scholar : PubMed/NCBI
|
30
|
Tobias A: Assessing the influence of a
single study in the meta-analysis estimate. Stata Techn Bull.
47:15–17. 1999.
|
31
|
Gong C, Hu X, Gao Y, Cao Y, Gao F and Mo
Z: A meta-analysis of the NAT1 and NAT2 polymorphisms and prostate
cancer: a huge review. Med Oncol. 28:365–376. 2011. View Article : Google Scholar : PubMed/NCBI
|
32
|
Ying XJ, Dong P, Shen B, Wang J, Wang S
and Wang G: Possible association of NAT2 polymorphism with
laryngeal cancer risk: an evidence-based meta-analysis. J Cancer
Res Clin Oncol. 137:1661–1667. 2011. View Article : Google Scholar : PubMed/NCBI
|
33
|
Cui D, Wang Z, Zhao E, Ma J and Lu W: NAT2
polymorphism and lung cancer risk: a meta-analysis. Lung Cancer.
73:153–157. 2011. View Article : Google Scholar : PubMed/NCBI
|
34
|
Zhong X, Hui C, Xiao-Ling W, Yan L and Na
L: NAT2 polymorphism and gastric cancer susceptibility: a
meta-analysis. Arch Med Res. 41:275–280. 2010. View Article : Google Scholar : PubMed/NCBI
|
35
|
Ye Z and Parry JM: Meta-analysis of 20
case-control studies on the N-acetyltransferase 2 acetylation
status and colorectal cancer risk. Med Sci Monit. 8:CR558–565.
2002.PubMed/NCBI
|
36
|
Borlak J and Reamon-Buettner SM:
N-acetyltransferase 2 (NAT2) gene polymorphisms in colon and lung
cancer patients. BMC Med Genet. 7:582006. View Article : Google Scholar : PubMed/NCBI
|
37
|
Zhang LQ, Zhou JN, Wang J, et al: Absence
of association between N-acetyltransferase 2 acetylator status and
colorectal cancer susceptibility: based on evidence from 40
studies. PLoS One. 7:e324252012. View Article : Google Scholar : PubMed/NCBI
|
38
|
Botteri E, Iodice S, Raimondi S,
Maisonneuve P and Lowenfels AB: Cigarette smoking and adenomatous
polyps: a meta-analysis. Gastroenterology. 134:388–395. 2008.
View Article : Google Scholar : PubMed/NCBI
|
39
|
Zhou JY, Shi R, Yu HL, Zheng WL and Ma WL:
Association of NQO1 Pro187Ser polymorphism with the risks for
colorectal cancer and colorectal adenoma: a meta-analysis. Int J
Colorectal Dis. 27:1123–1124. 2012. View Article : Google Scholar : PubMed/NCBI
|
40
|
Brockton N, Little J, Sharp L and Cotton
SC: N-acetyltransferase polymorphisms and colorectal cancer: a HuGE
review. Am J Epidemiol. 151:846–861. 2000. View Article : Google Scholar : PubMed/NCBI
|
41
|
Liu L, Wagner CR and Hanna PE:
Isoform-selective inactivation of human arylamine
N-acetyltransferases by reactive metabolites of carcinogenic
arylamines. Chem Res Toxicol. 22:1962–1974. 2009. View Article : Google Scholar : PubMed/NCBI
|
42
|
Walker K, Ginsberg G, Hattis D, Johns DO,
Guyton KZ and Sonawane B: Genetic polymorphism in
N-acetyltransferase (NAT): population distribution of NAT1 and NAT2
activity. J Toxicol Environ Health B Crit Rev. 12:440–472. 2009.
View Article : Google Scholar : PubMed/NCBI
|