1
|
Murry CE, Jennings RB and Reimer KA:
Preconditioning with ischemia: A delay of lethal cell injury in
ischemic myocardium. Circulation. 74:1124–1136. 1986. View Article : Google Scholar : PubMed/NCBI
|
2
|
Das S, Cordis GA, Maulik N and Das DK:
Pharmacological preconditioning with resveratrol: Role of
CREB-dependent Bcl-2 signaling via adenosine A3 receptor
activation. Am J Physiol Heart Circ Physiol. 288:H328–H335. 2005.
View Article : Google Scholar : PubMed/NCBI
|
3
|
Frassdorf J, Weber NC, Obal D, Toma O,
Müllenheim J, Kojda G, Preckel B and Schlack W: Morphine induces
late cardioprotection in rat hearts in vivo: The involvement of
opioid receptors and nuclear transcription factor kappaB. Anesth
Analg. 101:934–941. 2005. View Article : Google Scholar : PubMed/NCBI
|
4
|
Wu SN, Wu AZ and Sung RJ: Identification
of two types of ATP-sensitive K+ channels in rat ventricular
myocytes. Life Sci. 80:378–387. 2007. View Article : Google Scholar : PubMed/NCBI
|
5
|
Kunuthur SP, Mocanu MM, Hemmings BA,
Hausenloy DJ and Yellon DM: The Akt1 isoform is an essential
mediator of ischaemic preconditioning. J Cell Mol Med.
16:1739–1749. 2012. View Article : Google Scholar : PubMed/NCBI
|
6
|
Dragasis S, Bassiakou E, Iacovidou N,
Papadimitriou L, Andreas Steen P, Gulati A and Xanthos T: The role
of opioid receptor agonists in ischemic preconditioning. Eur J
Pharmacol. 720:401–408. 2013. View Article : Google Scholar : PubMed/NCBI
|
7
|
Li G, Whittaker P, Yao M, Kloner RA and
Przyklenk K: The gap junction uncoupler heptanol abrogates infarct
size reduction with preconditioning in mouse hearts. Cardiovasc
Pathol. 11:158–165. 2002. View Article : Google Scholar : PubMed/NCBI
|
8
|
Schulz R, Boengler K, Totzeck A, Luo Y,
Garcia-Dorado D and Heusch G: Connexin 43 in ischemic pre- and
postconditioning. Heart Fail Rev. 12:261–266. 2007. View Article : Google Scholar : PubMed/NCBI
|
9
|
Schulz R, Cohen MV, Behrends M, Downey JM
and Heusch G: Signal transduction of ischemic preconditioning.
Cardiovasc Res. 52:181–198. 2001. View Article : Google Scholar : PubMed/NCBI
|
10
|
Söhl G and Willecke K: Gap junctions and
the connexin protein family. Cardiovasc Res. 62:228–232. 2004.
View Article : Google Scholar : PubMed/NCBI
|
11
|
Smyth JW and Shaw RM: Autoregulation of
connexin43 gap junction formation by internally translated
isoforms. Cell Rep. 5:611–618. 2013. View Article : Google Scholar : PubMed/NCBI
|
12
|
Zhang SS, Hong S, Kléber AG, Lee LP and
Shaw RM: A micropatterning approach for imaging dynamic Cx43
trafficking to cell-cell borders. FEBS Lett. 588:1439–1445. 2014.
View Article : Google Scholar : PubMed/NCBI
|
13
|
Kanter HL, Saffitz JE and Beyer EC:
Molecular cloning of two human cardiac gap junction proteins,
connexin40 and connexin45. J Mol Cell Cardiol. 26:861–868. 1994.
View Article : Google Scholar : PubMed/NCBI
|
14
|
Lau AF, Hatch-Pigott V and Crow DS:
Evidence that heart connexin43 is a phosphoprotein. J Mol Cell
Cardiol. 23:659–663. 1991. View Article : Google Scholar : PubMed/NCBI
|
15
|
Dhein S: Pharmacology of gap junctions in
the cardiovascular system. Cardiovasc Res. 62:287–298. 2004.
View Article : Google Scholar : PubMed/NCBI
|
16
|
Veenstra RD, Wang HZ, Beblo DA, Chilton
MG, Harris AL, Beyer EC and Brink PR: Selectivity of
connexin-specific gap junctions does not correlate with channel
conductance. Circ Res. 77:1156–1165. 1995. View Article : Google Scholar : PubMed/NCBI
|
17
|
Quist AP, Rhee SK, Lin H and Lal R:
Physiological role of gap-junctional hemichannels. Extracellular
calcium-dependent isosmotic volume regulation. J Cell Biol.
148:1063–1074. 2000. View Article : Google Scholar : PubMed/NCBI
|
18
|
Decrock E, Vinken M, De Vuyst E, Krysko
DV, D'Herde K, Vanhaecke T, Vandenabeele P, Rogiers V and Leybaert
L: Connexin-related signaling in cell death: To live or let die?
Cell Death Differ. 16:524–536. 2009. View Article : Google Scholar : PubMed/NCBI
|
19
|
Boengler K, Dodoni G, Rodriguez-Sinovas A,
Cabestrero A, Ruiz-Meana M, Gres P, Konietzka I, Lopez-Iglesias C,
Garcia-Dorado D, Di Lisa F, et al: Connexin 43 in cardiomyocyte
mitochondria and its increase by ischemic preconditioning.
Cardiovasc Res. 67:234–244. 2005. View Article : Google Scholar : PubMed/NCBI
|
20
|
Goubaeva F, Mikami M, Giardina S, Ding B,
Abe J and Yang J: Cardiac mitochondrial connexin 43 regulates
apoptosis. Biochem Biophys Res Commun. 352:97–103. 2007. View Article : Google Scholar : PubMed/NCBI
|
21
|
Ruiz-Meana M, Núñez E, Miro-Casas E,
Martínez-Acedo P, Barba I, Rodriguez-Sinovas A, Inserte J,
Fernandez-Sanz C, Hernando V, Vázquez J and Garcia-Dorado D:
Ischemic preconditioning protects cardiomyocyte mitochondria
through mechanisms independent of cytosol. J Mol Cell Cardiol.
68:79–88. 2014. View Article : Google Scholar : PubMed/NCBI
|
22
|
Garcia-Dorado D, Ruiz-Meana M, Padilla F,
Rodriguez-Sinovas A and Mirabet M: Gap junction-mediated
intercellular communication in ischemic preconditioning. Cardiovasc
Res. 55:456–465. 2002. View Article : Google Scholar : PubMed/NCBI
|
23
|
Rong B, Xie F, Sun T, Hao L, Lin MJ and
Zhong JQ: Nitric oxide, PKC-ε, and connexin43 are crucial for
ischemic preconditioning-induced chemical gap junction uncoupling.
Oncotarget. 7:69243–69255. 2016. View Article : Google Scholar : PubMed/NCBI
|
24
|
Fernández-Jiménez R, García-Prieto J,
Sánchez-González J, Agüero J, López-Martín GJ, Galán-Arriola C,
Molina-Iracheta A, Doohan R, Fuster V and Ibáñez B: Pathophysiology
underlying the bimodal edema phenomenon after myocardial
ischemia/reperfusion. J Am Coll Cardiol. 66:816–828. 2015.
View Article : Google Scholar : PubMed/NCBI
|
25
|
Holmes JW, Borg TK and Covell JW:
Structure and mechanics of healing myocardial infarcts. Annu Rev
Biomed Eng. 7:223–253. 2005. View Article : Google Scholar : PubMed/NCBI
|
26
|
Diaz RJ, Armstrong SC, Batthish M, Backx
PH, Ganote CE and Wilson GJ: Enhanced cell volume regulation: A key
protective mechanism of ischemic preconditioning in rabbit
ventricular myocytes. J Mol Cell Cardiol. 35:45–58. 2003.
View Article : Google Scholar : PubMed/NCBI
|
27
|
Heusch G, Cohen MV and Downey JM: Ischemic
preconditioning through opening of swelling-activated chloride
channels? Circ Res. 89:E482001. View Article : Google Scholar : PubMed/NCBI
|
28
|
Naitoh K, Yano T, Miura T, Itoh T, Miki T,
Tanno M, Sato T, Hotta H, Terashima Y and Shimamoto K: Roles of
Cx43-associated protein kinases in suppression of gap
junction-mediated chemical coupling by ischemic preconditioning. Am
J Physiol Heart Circ Physiol. 296:H396–H403. 2009. View Article : Google Scholar : PubMed/NCBI
|
29
|
Miura T, Ohnuma Y, Kuno A, Tanno M,
Ichikawa Y, Nakamura Y, Yano T, Miki T, Sakamoto J and Shimamoto K:
Protective role of gap junctions in preconditioning against
myocardial infarction. Am J Physiol Heart Circ Physiol.
286:H214–H221. 2004. View Article : Google Scholar : PubMed/NCBI
|
30
|
Luo Y, Fang J, Fan L, Lin C, Chen Z and
Chen L: Octanol preconditioning alleviates mouse cardiomyocyte
swelling induced by simulated ischemia/reperfusion challenge in
vitro. Nan Fang Yi Ke Da Xue Xue Bao. 32:1419–1422. 2012.(In
Chinese). PubMed/NCBI
|
31
|
Azzam EI, de Toledo SM and Little JB:
Direct evidence for the participation of gap junction-mediated
intercellular communication in the transmission of damage signals
from alpha-particle irradiated to nonirradiated cells. Proc Natl
Acad Sci USA. 98:473–478. 2001. View Article : Google Scholar : PubMed/NCBI
|
32
|
Garcia-Dorado D, Inserte J, Ruiz-Meana M,
González MA, Solares J, Juliá M, Barrabés JA and Soler-Soler J: Gap
junction uncoupler heptanol prevents cell-to-cell progression of
hypercontracture and limits necrosis during myocardial reperfusion.
Circulation. 96:3579–3586. 1997. View Article : Google Scholar : PubMed/NCBI
|
33
|
Garcia-Dorado D, Rodriguez-Sinovas A and
Ruiz-Meana M: Gap junction-mediated spread of cell injury and death
during myocardial ischemia-reperfusion. Cardiovasc Res. 61:386–401.
2004. View Article : Google Scholar : PubMed/NCBI
|
34
|
Chen BP, Mao HJ, Fan FY, Bruce IC and Xia
Q: Delayed uncoupling contributes to the protective effect of
heptanol against ischaemia in the rat isolated heart. Clin Exp
Pharmacol Physiol. 32:655–662. 2005. View Article : Google Scholar : PubMed/NCBI
|
35
|
Rodriguez-Sinovas A, Garcia-Dorado D,
Ruiz-Meana M and Soler-Soler J: Protective effect of gap junction
uncouplers given during hypoxia against reoxygenation injury in
isolated rat hearts. Am J Physiol Heart Circ Physiol.
290:H648–H656. 2006. View Article : Google Scholar : PubMed/NCBI
|
36
|
Schwanke U, Konietzka I, Duschin A, Li X,
Schulz R and Heusch G: No ischemic preconditioning in heterozygous
connexin43-deficient mice. Am J Physiol Heart Circ Physiol.
283:H1740–H1742. 2002. View Article : Google Scholar : PubMed/NCBI
|
37
|
Diaz RJ, Harvey K, Boloorchi A, Hossain T,
Hinek A, Backx PH and Wilson GJ: Enhanced cell volume regulation: A
key mechanism in local and remote ischemic preconditioning. Am J
Physiol Cell Physiol. 306:C1191–C1199. 2014. View Article : Google Scholar : PubMed/NCBI
|
38
|
Kloner RA: New observations regarding
post-ischemia/reperfusion myocardial swelling. J Am Coll Cardiol.
65:324–326. 2015. View Article : Google Scholar : PubMed/NCBI
|
39
|
Oldenburg O, Qin Q, Krieg T, Yang XM,
Philipp S, Critz SD, Cohen MV and Downey JM: Bradykinin induces
mitochondrial ROS generation via NO, cGMP, PKG, and mitoKATP
channel opening and leads to cardioprotection. Am J Physiol Heart
Circ Physiol. 286:H468–H476. 2004. View Article : Google Scholar : PubMed/NCBI
|
40
|
Korchev YE, Gorelik J, Lab MJ, Sviderskaya
EV, Johnston CL, Coombes CR, Vodyanoy I and Edwards CR: Cell volume
measurement using scanning ion conductance microscopy. Biophys J.
78:451–457. 2000. View Article : Google Scholar : PubMed/NCBI
|
41
|
Curl CL, Bellair CJ, Harris PJ, Allman BE,
Roberts A, Nugent KA and Delbridge LM: Single cell volume
measurement by quantitative phase microscopy (QPM): A case study of
erythrocyte morphology. Cell Physiol Biochem. 17:193–200. 2006.
View Article : Google Scholar : PubMed/NCBI
|