|
1
|
Aaron CP, Chervona Y, Kawut SM, Roux AV
Diez, Shen M, Bluemke DA, Van Hee VC, Kaufman JD and Barr RG:
Particulate matter exposure and cardiopulmonary differences in the
multi-ethnic study of atherosclerosis. Environ Health Perspect.
124:1166–1173. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Thurston G and Lippmann M: Ambient
particulate matter air pollution and cardiopulmonary diseases.
Semin Respir Crit Care Med. 36:422–432. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Amatullah H, North ML, Akhtar US, Rastogi
N, Urch B, Silverman FS, Chow CW, Evans GJ and Scott JA:
Comparative cardiopulmonary effects of size-fractionated airborne
particulate matter. Inhal Toxicol. 24:161–171. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Cui P, Huang Y, Han J, Song F and Chen K:
Ambient particulate matter and lung cancer incidence and mortality:
A meta-analysis of prospective studies. Eur J Public Health.
25:324–329. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Fu J, Jiang D, Lin G, Liu K and Wang Q: An
ecological analysis of PM2.5 concentrations and lung cancer
mortality rates in China. BMJ Open. 5:e0094522015. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Yue H, Yun Y, Gao R, Li G and Sang N:
Winter polycyclic aromatic hydrocarbon-bound particulate matter
from peri-urban North China promotes lung cancer cell metastasis.
Environ Sci Technol. 49:14484–14493. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Hu H, Dailey AB, Kan H and Xu X: The
effect of atmospheric particulate matter on survival of breast
cancer among US females. Breast Cancer Res Treat. 139:217–226.
2013. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Lin CP, Lin FY, Huang PH, Chen YL, Chen
WC, Chen HY, Huang YC, Liao WL, Huang HC, Liu PL, et al:
Endothelial progenitor cell dysfunction in cardiovascular diseases:
Role of reactive oxygen species and inflammation. Biomed Res Int.
2013:8450372013. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Gimbrone MA Jr and García-Cardeña G:
Endothelial cell dysfunction and the pathobiology of
atherosclerosis. Circ Res. 118:620–636. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Yang GZ, Wang ZJ, Bai F, Qin XJ, Cao J, Lv
JY and Zhang MS: Epigallocatechin-3-gallate protects HUVECs from
PM2.5-induced oxidative stress injury by activating critical
antioxidant pathways. Molecules. 20:6626–6639. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Cui Y, Xie X, Jia F, He J, Li Z, Fu M, Hao
H, Liu Y, Liu JZ, Cowan PJ, et al: Ambient fine particulate matter
induces apoptosis of endothelial progenitor cells through reactive
oxygen species formation. Cell Physiol Biochem. 35:353–363. 2015.
View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Tong GQ, Zhang ZH, Zhao Y, Liu JJ and Han
JB: Traffic-related PM2.5 induces cytosolic [Ca2+]
increase regulated by Orai1, alters the CaN-NFAT signaling pathway,
and affects IL-2 and TNF-α cytoplasmic levels in Jurkat T-cells.
Arch Environ Contam Toxicol. 68:31–37. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Li R, Kou X, Xie L, Cheng F and Geng H:
Effects of ambient PM2.5 on pathological injury, inflammation,
oxidative stress, metabolic enzyme activity, and expression of
c-fos and c-jun in lungs of rats. Environ Sci Pollut Res Int.
22:20167–20176. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Ostro B, Malig B, Broadwin R, Basu R, Gold
EB, Bromberger JT, Derby C, Feinstein S, Greendale GA, Jackson EA,
et al: Chronic PM2.5 exposure and inflammation: Determining
sensitive subgroups in mid-life women. Environ Res. 132:168–175.
2014. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Potera C: Toxicity beyond the lung:
Connecting PM2.5, inflammation, and diabetes. Environ Health
Perspect. 122:A292014. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Wang Y, Lin Z, Huang H, He H, Yang L, Chen
T, Yang T, Ren N, Jiang Y, Xu W, et al: AMPK is required for
PM2.5-induced autophagy in human lung epithelial A549 cells. Int J
Clin Exp Med. 8:58–72. 2015.PubMed/NCBI
|
|
17
|
Deng X, Zhang F, Rui W, Long F, Wang L,
Feng Z, Chen D and Ding W: PM2.5-induced oxidative stress triggers
autophagy in human lung epithelial A549 cells. Toxicol In Vitro.
27:1762–1770. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Wang FF, Geng CM, Hao WD, Zhao YD, Li Q,
Wang HM and Qian Y: The cellular toxicity of PM2.5 emitted from
coal combustion in human umbilical vein endothelial cells. Biomed
Environ Sci. 29:107–116. 2016.PubMed/NCBI
|
|
19
|
Montiel-Dávalos A, Alfaro-Moreno E and
López-Marure R: PM2.5 and PM10 induce the expression of adhesion
molecules and the adhesion of monocytic cells to human umbilical
vein endothelial cells. Inhal Toxicol. 19:(Suppl 1). 91–98. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Rui W, Guan L, Zhang F, Zhang W and Ding
W: PM2.5-induced oxidative stress increases adhesion molecules
expression in human endothelial cells through the
ERK/AKT/NF-κB-dependent pathway. J Appl Toxicol. 36:48–59. 2016.
View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Abdelrahim M, Konduri S, Basha R, Philip
PA and Baker CH: Angiogenesis: An update and potential drug
approaches (Review). Int J Oncol. 36:5–18. 2010.PubMed/NCBI
|
|
22
|
Favreau AJ, Vary CP, Brooks PC and
Sathyanarayana P: Cryptic collagen IV promotes cell migration and
adhesion in myeloid leukemia. Cancer Med. 3:265–272. 2014.
View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Hessel M, Steendijk P, den Adel B, Schutte
C and van der Laarse A: Pressure overload-induced right ventricular
failure is associated with re-expression of myocardial tenascin-C
and elevated plasma tenascin-C levels. Cell Physiol Biochem.
24:201–210. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Ide M, Saito K, Tsutsumi S, Tsuboi K,
Yamaguchi S, Asao T, Kuwano H and Nakajima T: Over-expression of
14-3-3σ in budding colorectal cancer cells modulates cell migration
in the presence of tenascin-C. Oncol Rep. 18:1451–1456.
2007.PubMed/NCBI
|
|
25
|
Madsen CD, Ferraris GM, Andolfo A,
Cunningham O and Sidenius N: uPAR-induced cell adhesion and
migration: Vitronectin provides the key. J Cell Biol. 177:927–939.
2007. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Zhu G, Huang Q, Zheng W, Huang Y, Hua J,
Yang S, Zhuang J, Wang J, Chang J, Xu J and Ye J: LPS upregulated
VEGFR-3 expression promote migration and invasion in colorectal
cancer via a mechanism of increased NF-κB binding to the promoter
of VEGFR-3. Cell Physiol Biochem. 39:1665–1678. 2016. View Article : Google Scholar : PubMed/NCBI
|
