1
|
Thompson CS: Diabetic nephropathy:
Treatment with phosphodiesterase type 5 inhibitors. World J
Diabetes. 4:124–129. 2013. View Article : Google Scholar : PubMed/NCBI
|
2
|
Luyckx VA, Tonelli M and Stanifer JW: The
global burden of kidney disease and the sustainable development
goals. Bull World Health Organ. 96:414–422. 2018. View Article : Google Scholar : PubMed/NCBI
|
3
|
Shields J and Maxwell AP: Managing
diabetic nephropathy. Clin Med. 10:500–504. 2010. View Article : Google Scholar
|
4
|
Tominaga T, Abe H, Ueda O, Goto C,
Nakahara K, Murakami T, Matsubara T, Mima A, Nagai K, Araoka T, et
al: Activation of bone morphogenetic protein 4 signaling leads to
glomerulosclerosis that mimics diabetic nephropathy. J Biol Chem.
286:20109–20116. 2011. View Article : Google Scholar : PubMed/NCBI
|
5
|
Fernandes SM, Cordeiro PM, Watanabe M,
Fonseca CD and Vattimo MF: The role of oxidative stress in
streptozotocin-induced diabetic nephropathy in rats. Arch
Endocrinol Metab. 60:443–449. 2016. View Article : Google Scholar : PubMed/NCBI
|
6
|
Yu JB, Shi J, Gong LR, Dong SA, Xu Y,
Zhang Y, Cao XS and Wu LL: Role of Nrf2/ARE pathway in protective
effect of electroacupuncture against endotoxic shock-induced acute
lung injury in rabbits. PLoS One. 9:e1049242014. View Article : Google Scholar : PubMed/NCBI
|
7
|
Ansari MA: Sinapic acid modulates
Nrf2/HO-1 signaling pathway in cisplatin-induced nephrotoxicity in
rats. Biomed Pharmacother. 93:646–653. 2017. View Article : Google Scholar : PubMed/NCBI
|
8
|
Lv S, Zhou Q, Xia Y, You X, Zhao Z, Li Y
and Zou H: The association between oxidative stress alleviation via
sulforaphane-induced Nrf2-HO-1/NQO-1 signaling pathway activation
and chronic renal allograft dysfunction improvement. Kidney Blood
Press Res. 43:191–205. 2018. View Article : Google Scholar : PubMed/NCBI
|
9
|
Ilkun O and Boudina S: Cardiac dysfunction
and oxidative stress in the metabolic syndrome: An update on
antioxidant therapies. Curr Pharm Des. 19:4806–4817. 2013.
View Article : Google Scholar : PubMed/NCBI
|
10
|
Rochette L, Zeller M, Cottin Y and Vergely
C: Diabetes, oxidative stress and therapeutic strategies. Biochim
Biophys Acta. 1840:2709–2729. 2014. View Article : Google Scholar : PubMed/NCBI
|
11
|
Arora MK and Singh UK: Oxidative stress:
Meeting multiple targets in pathogenesis of diabetic nephropathy.
Curr Drug Targets. 15:531–538. 2014. View Article : Google Scholar : PubMed/NCBI
|
12
|
Liu L, Shi M, Wang Y, Zhang C, Su B, Xiao
Y and Guo B: SnoN upregulation ameliorates renal fibrosis in
diabetic nephropathy. PLoS One. 12:e01744712017. View Article : Google Scholar : PubMed/NCBI
|
13
|
Palanisamy N, Kannappan S and Anuradha CV:
Genistein modulates NF-κB-associated renal inflammation, fibrosis
and podocyte abnormalities in fructose-fed rats. Eur J Pharmacol.
667:355–364. 2011. View Article : Google Scholar : PubMed/NCBI
|
14
|
Meng XM, Tang PM, Li J and Lan HY:
TGF-β/Smad signaling in renal fibrosis. Front Physiol. 6:822015.
View Article : Google Scholar : PubMed/NCBI
|
15
|
Guo TL, Germolec DR, Zheng JF, Kooistra L,
Auttachoat W, Smith MJ, White KL and Elmore SA: Genistein protects
female nonobese diabetic mice from developing type 1 diabetes when
fed a soy- and alfalfa-free diet. Toxicol Pathol. 43:435–448. 2015.
View Article : Google Scholar : PubMed/NCBI
|
16
|
Maulik SK, Prabhakar P, Dinda AK and Seth
S: Genistein prevents isoproterenol-induced cardiac hypertrophy in
rats. Can J Physiol Pharmacol. 90:1117–1125. 2012. View Article : Google Scholar : PubMed/NCBI
|
17
|
Zhou P, Wang C, Hu Z, Chen W, Qi W and Li
A: Genistein induces apoptosis of colon cancer cells by reversal of
epithelial-to-mesenchymal via a Notch1/NF-κB/slug/E-cadherin
pathway. BMC Cancer. 17:8132017. View Article : Google Scholar : PubMed/NCBI
|
18
|
Bhattarai G, Poudel SB, Kook SH and Lee
JC: Anti-inflammatory, anti-osteoclastic and antioxidant activities
of genistein protect against alveolar bone loss and periodontal
tissue degradation in a mouse model of periodontitis. J Biomed
Mater Res A. 105:2510–2521. 2017. View Article : Google Scholar : PubMed/NCBI
|
19
|
Rajput MS and Sarkar PD: Modulation of
neuro-inflammatory condition, acetylcholinesterase and antioxidant
levels by genistein attenuates diabetes associated cognitive
decline in mice. Chem Biol Interact. 268:93–102. 2017. View Article : Google Scholar : PubMed/NCBI
|
20
|
Mirahmadi SM, Shahmohammadi A, Rousta AM,
Azadi MR, Fahanik-Babaei J, Baluchnejadmojarad T and Roghani M: Soy
isoflavone genistein attenuates lipopolysaccharide-induced
cognitive impairments in the rat via exerting anti-oxidative and
anti-inflammatory effects. Cytokine. 104:151–159. 2017. View Article : Google Scholar : PubMed/NCBI
|
21
|
Gupta SK, Dongare S, Mathur R, Mohanty IR,
Srivastava S, Mathur S and Nag TC: Genistein ameliorates cardiac
inflammation and oxidative stress in streptozotocin-induced
diabetic cardiomyopathy in rats. Mol Cell Biochem. 408:63–72. 2015.
View Article : Google Scholar : PubMed/NCBI
|
22
|
Yu JY, Lee JJ, Lim Y, Kim TJ, Jin YR,
Sheen YY and Yun YP: Genistein inhibits rat aortic smooth muscle
cell proliferation through the induction of p27kip1. J
Pharmacol Sci. 107:90–98. 2008. View Article : Google Scholar : PubMed/NCBI
|
23
|
Yang R, Jia Q, Liu XF and Ma SF: Effect of
genistein on myocardial fibrosis in diabetic rats and its
mechanism. Mol Med Rep. 17:2929–2936. 2018.PubMed/NCBI
|
24
|
Li WF, Yang K, Zhu P, Zhao HQ, Song YH,
Liu KC and Huang WF: Genistein ameliorates
ischemia/reperfusion-induced renal injury in a SIRT1-dependent
manner. Nutrients. 9:4032017. View Article : Google Scholar :
|
25
|
Elmarakby AA and Sullivan JC: Relationship
between oxidative stress and inflammatory cytokines in diabetic
nephropathy. Cardiovasc Ther. 30:49–59. 2012. View Article : Google Scholar : PubMed/NCBI
|
26
|
Lan HY: Transforming growth factor-β/Smad
signalling in diabetic nephropathy. Clin Exp Pharmacol Physiol.
39:731–738. 2012. View Article : Google Scholar : PubMed/NCBI
|
27
|
Al Hroob AM, Abukhalil MH, Alghonmeen RD
and Mahmoud AM: Ginger alleviates hyperglycemia-induced oxidative
stress, inflammation and apoptosis and protects rats against
diabetic nephropathy. Biomed Pharmacother. 106:381–389. 2018.
View Article : Google Scholar : PubMed/NCBI
|
28
|
Sifuentes-Franco S, Padilla-Tejeda DE,
Carrillo-Ibarra S and Miranda-Díaz AG: Oxidative stress, apoptosis,
and mitochondrial function in diabetic nephropathy. Int J
Endocrinol. 2018:18758702018. View Article : Google Scholar : PubMed/NCBI
|
29
|
Mahmoodnia L, Aghadavod E, Beigrezaei S
and Rafieian-Kopaei M: An update on diabetic kidney disease,
oxidative stress and antioxidant agents. J Renal Inj Prev.
6:153–157. 2017. View Article : Google Scholar : PubMed/NCBI
|
30
|
Sung MJ, Kim DH, Jung YJ, Kang KP, Lee AS,
Lee S, Kim W, Davaatseren M, Hwang JT, Kim HJ, et al: Genistein
protects the kidney from cisplatin-induced injury. Kidney Int.
74:1538–1547. 2008. View Article : Google Scholar : PubMed/NCBI
|
31
|
Sobhy MM, Mahmoud SS, El-Sayed SH, Rizk
EM, Raafat A and Negm MSI: Impact of treatment with a protein
tyrosine kinase inhibitor (Genistein) on acute and chronic
experimental Schistosoma mansoni infection. Exp Parasitol.
185:115–123. 2018. View Article : Google Scholar : PubMed/NCBI
|
32
|
Shafiee G, Saidijam M, Tavilani H,
Ghasemkhani N and Khodadadi I: Genistein induces apoptosis and
inhibits proliferation of HT29 colon cancer cells. Int J Mol Cell
Med. 5:178–191. 2016.PubMed/NCBI
|
33
|
Qi W, Weber CR, Wasland K and Savkovic SD:
Genistein inhibits proliferation of colon cancer cells by
attenuating a negative effect of epidermal growth factor on tumor
suppressor FOXO3 activity. BMC Cancer. 11:2192011. View Article : Google Scholar : PubMed/NCBI
|
34
|
Pillai MS and Shivakumar K: Genistein
abolishes nucleoside uptake by cardiac fibroblasts. Mol Cell
Biochem. 332:121–125. 2009. View Article : Google Scholar : PubMed/NCBI
|
35
|
Guo Y, Zhang A, Ding Y, Wang Y and Yuan W:
Genistein ameliorates parathyroid hormone-induced
epithelial-to-mesenchymal transition and inhibits expression of
connective tissue growth factor in human renal proximal tubular
cells. Arch Med Sci. 9:724–730. 2013. View Article : Google Scholar : PubMed/NCBI
|
36
|
Nogueira A, Pires MJ and Oliveira PA:
Pathophysiological mechanisms of renal fibrosis: A review of animal
models and therapeutic strategies. In Vivo. 31:1–22. 2017.
View Article : Google Scholar : PubMed/NCBI
|
37
|
Wang Z, Han Z, Tao J, Wang J, Liu X, Zhou
W, Xu Z, Zhao C, Tan R and Gu M: Role of endothelial-to-mesenchymal
transition induced by TGF-β1 in transplant kidney interstitial
fibrosis. J Cell Mol Med. 21:2359–2369. 2017. View Article : Google Scholar : PubMed/NCBI
|
38
|
Diez-Marques L, Ortega-Velazquez R, Langa
C, Rodriguez-Barbero A, Lopez-Novoa JM, Lamas S and Bernabeu C:
Expression of endoglin in human mesangial cells: Modulation of
extracellular matrix synthesis. Biochim Biophys Acta. 1587:36–44.
2002. View Article : Google Scholar : PubMed/NCBI
|
39
|
Sutariya B and Saraf M: Betanin, isolated
from fruits of Opuntia elatior Mill attenuates renal fibrosis in
diabetic rats through regulating oxidative stress and TGF-β
pathway. J Ethnopharmacol. 198:432–443. 2017. View Article : Google Scholar : PubMed/NCBI
|
40
|
Song JH, Cha SH, Lee HJ, Lee SW, Park GH
and Kim MJ: Effect of low-dose dual blockade of renin-angiotensin
system on urinary TGF-β in type 2 diabetic patients with advanced
kidney disease. Nephrol Dial Transplant. 21:683–689. 2006.
View Article : Google Scholar : PubMed/NCBI
|
41
|
Samarakoon R, Overstreet JM, Higgins SP
and Higgins PJ: TGF-β1 → SMAD/p53/USF2 → PAI-1 transcriptional axis
in ureteral obstruction-induced renal fibrosis. Cell Tissue Res.
347:117–128. 2012. View Article : Google Scholar : PubMed/NCBI
|
42
|
Wu CF, Chiang WC, Lai CF, Chang FC, Chen
YT, Chou YH, Wu TH, Linn GR, Ling H, Wu KD, et al: Transforming
growth factor β-1 stimulates profibrotic epithelial signaling to
activate pericyte-myofibroblast transition in obstructive kidney
fibrosis. Am J Pathol. 182:118–131. 2013. View Article : Google Scholar : PubMed/NCBI
|
43
|
López-Hernández FJ and López-Novoa JM:
Role of TGF-β in chronic kidney disease: An integration of tubular,
glomerular and vascular effects. Cell Tissue Res. 347:141–154.
2012. View Article : Google Scholar : PubMed/NCBI
|
44
|
Lan HY: Diverse roles of TGF-β/Smads in
renal fibrosis and inflammation. Int J Biol Sci. 7:1056–1067. 2011.
View Article : Google Scholar : PubMed/NCBI
|
45
|
Hata A and Chen YG: TGF-β signaling from
receptors to smads. Cold Spring Harb Perspect Biol. 8(pii):
a0220612016. View Article : Google Scholar : PubMed/NCBI
|
46
|
Zhao K, He J, Zhang Y, Xu Z, Xiong H, Gong
R, Li S, Chen S and He F: Activation of FXR protects against renal
fibrosis via suppressing Smad3 expression. Sci Rep. 6:372342016.
View Article : Google Scholar : PubMed/NCBI
|
47
|
Fan K, Wu K, Lin L, Ge P, Dai J, He X, Hu
K and Zhang L: Metformin mitigates carbon tetrachloride-induced
TGF-β1/Smad3 signaling and liver fibrosis in mice. Biomed
Pharmacother. 90:421–426. 2017. View Article : Google Scholar : PubMed/NCBI
|
48
|
Li X, Han D, Tian Z, Gao B, Fan M, Li C,
Wang Y, Ma S and Cao F: Activation of cannabinoid receptor type II
by AM1241 ameliorates myocardial fibrosis via Nrf2-mediated
inhibition of TGF-β1/Smad3 pathway in myocardial infarction mice.
Cell Physiol Biochem. 39:1521–1536. 2016. View Article : Google Scholar : PubMed/NCBI
|
49
|
Qu Y, Zhang L, Kang Z, Jiang W and Lv C:
Ponatinib ameliorates pulmonary fibrosis by suppressing
TGF-β1/Smad3 pathway. Pulm Pharmacol Ther. 34:1–7. 2015. View Article : Google Scholar : PubMed/NCBI
|
50
|
Wang W, Zhou PH, Hu W, Xu CG, Zhou XJ,
Liang CZ and Zhang J: Cryptotanshinone hinders renal fibrosis and
epithelial transdifferentiation in obstructive nephropathy by
inhibiting TGF-β1/Smad3/integrin β1 signal. Oncotarget.
9:26625–26637. 2017.PubMed/NCBI
|
51
|
Feng J, Xie L, Kong R, Zhang Y, Shi K, Lu
W and Jiang H: RACK1 silencing attenuates renal fibrosis by
inhibiting TGF-β signaling. Int J Mol Med. 40:1965–1970.
2017.PubMed/NCBI
|
52
|
Liu R, Das B, Xiao W, Li Z, Li H, Lee K
and He JC: A novel inhibitor of homeodomain interacting protein
kinase 2 mitigates kidney fibrosis through inhibition of the
TGF-β1/Smad3 pathway. J Am Soc Nephrol. 28:2133–2143. 2017.
View Article : Google Scholar : PubMed/NCBI
|