|
1
|
Taeb AM, Hooper MH and Marik PE: Sepsis:
Current definition, pathophysiology, diagnosis, and management.
Nutr Clin Pract. 32:296–308. 2017. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Dellinger RP, Levy MM, Rhodes A, Annane D,
Gerlach H, Opal SM, Sevransky JE, Sprung CL, Douglas IS, Jaeschke
R, et al: Surviving sepsis campaign: International guidelines for
management of severe sepsis and septic shock, 2012. Crit Care Med.
41:580–637. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Salomao R, Brunialti MK, Rapozo MM,
Baggio-Zappia GL, Galanos C and Freudenberg M: Bacterial sensing,
cell signaling, and modulation of the immune response during
sepsis. Shock. 38:227–242. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Bai Y, Lu B and Sun Q: Pre-exposure to
fine particulate matters may induce endotoxin tolerance in a mouse
model. Austin J Environ Toxicol. 1:10042015.PubMed/NCBI
|
|
5
|
López-Collazo E and Del Fresno C:
Pathophysiology of endotoxin tolerance: Mechanisms and clinical
consequences. Crit Care. 17:2422013. View
Article : Google Scholar : PubMed/NCBI
|
|
6
|
Biswas SK and Lopez-Collazo E: Endotoxin
tolerance: New mechanisms, molecules and clinical significance.
Trends Immunol. 30:475–487. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Lacatus M: Innate immunity in surgical
patients. Chirurgia (Bucur). 108:18–25. 2013.PubMed/NCBI
|
|
8
|
Draisma A, Pickkers P, Bouw MP and van der
Hoeven JG: Development of endotoxin tolerance in humans in vivo.
Crit Care Med. 37:1261–1267. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Pena OM, Pistolic J, Raj D, Fjell CD and
Hancock RE: Endotoxin tolerance represents a distinctive state of
alternative polarization (M2) in human mononuclear cells. J
Immunol. 186:7243–7254. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Milot E, Fotouhi-Ardakani N and Filep JG:
Myeloid nuclear differentiation antigen, neutrophil apoptosis and
sepsis. Front Immunol. 3:3972012. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Xiong Y and Medvedev AE: Induction of
endotoxin tolerance in vivo inhibits activation of IRAK4 and
increases negative regulato rs IRAK-M, SHIP-1, and A20. J Leukoc
Biol. 90:1141–1148. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Piao W, Song C, Chen H, Diaz MA, Wahl LM,
Fitzgerald KA, Li L and Medvedev AE: Endotoxin tolerance
dysregulates MyD88- and Toll/IL-1R domain-containing adapter
inducing IFN-beta-de pendent pathways and increases expression of
negative regulators of TLR signaling. J Leukoc Biol. 86:863–875.
2009. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Cohen P: The search for physiological
substrates of MAP and SAP kinases in mammalian cells. Trends Cell
Biol. 7:353–361. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Nahid MA, Satoh M and Chan EK: MicroRNA in
TLR signaling and endotoxin tolerance. Cell Mol Immunol. 8:388–403.
2011. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Nahid MA, Satoh M and Chan EK: Interleukin
1β-responsive MicroRNA-146a is critical for the cytokine-induced
tolerance and cross-tole rance to toll-like receptor ligands. J
Innate Immun. 7:428–440. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Curtale G, Mirolo M, Renzi TA, Rossato M,
Bazzoni F and Locati M: Negative regulation of Toll-like receptor 4
signaling by IL-10-dependent microRNA-146b. Proc Natl Acad Sci U S
A. 110:11499–11504. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Liu M, John CM and Jarvis GA: Induction of
endotoxin tolerance by pathogenic neisseria is correlated with the
inflammatory potentia l of lipooligosaccharides and regulated by
microRNA-146a. J Immunol. 192:1768–1777. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Pena OM, Hancock DG, Lyle NH, Linder A,
Russell JA, Xia J, Fjell CD, Boyd JH and Hancock RE: An endotoxin
tolerance signature predicts sepsis and organ dysfunction at
initial clinical presentati on. EBioMedicine. 1:64–71. 2014.
View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Beeson PB: Development of tolerance to
typhoid bacterial pyrogen and its abolition by reticulo-endothelial
blockade. Proc Soc Exp Biol Med. 61:248–250. 1946. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Morris MC, Gilliam EA and Li L: Innate
immune programing by endotoxin and its pathological consequences.
Front Immunol. 5:6802015. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Rauniyar N and Yates JR 3rd: Isobaric
labeling-based relative quantification in shotgun proteomics. J
Proteome Res. 13:5293–5309. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Cao Z, Yende S, Kellum JA, Angus DC and
Robinson RA: Proteomics reveals age-related differences in the host
immune response to sepsis. J Proteome Res. 13:422–432. 2014.
View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Wu Z, Pan D, Guo Y, Zeng X and Sun Y:
iTRAQ proteomic analysis of N-acetylmuramic acid mediated
anti-inflammatory capacity in LPS-induced R AW 264.7 cells.
Proteomics. 15:2211–2219. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Yang YX and Li L: Identification of
potential biomarkers of sepsis using bioinformatics analysis. Exp
Ther Med. 13:1689–1696. 2017. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Li S, Luo C, Yin C, Peng C, Han R and Zhou
J, He Q and Zhou J: Endogenous HMGB1 is required in endotoxin
tolerance. J Surg Res. 185:319–328. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Yang NB, Ni SL, Li SS, Zhang SN, Hu DP and
Lu MQ: Endotoxin tolerance alleviates experimental acute liver
failure via inhibition of high mobility group box 1. Int J Clin Exp
Pathol. 8:9062–9071. 2015.PubMed/NCBI
|
|
27
|
Wang H, Liao H, Ochani M, Justiniani M,
Lin X, Yang L, Al-Abed Y, Wang H, Metz C, Miller EJ, et al:
Cholinergic agonists inhibit HMGB1 release and improve survival in
experimental sepsis. Nat Med. 10:1216–1221. 2004. View Article : Google Scholar : PubMed/NCBI
|
