|
1
|
Massano J, Regateiro FS, Januario G and
Ferreira A: Oral squamous cell carcinoma: review of prognostic and
predictive factors. Oral Surg Oral Med Oral Pathol Oral Radiol
Endod. 102:67–76. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Parkin DM, Bray F, Ferlay J and Pisani P:
Global cancer statistics, 2002. CA Cancer J Clin. 55:74–108. 2005.
View Article : Google Scholar
|
|
3
|
Lippman SM and Hong WK: Molecular markers
of the risk of oral cancer. N Engl J Med. 344:1323–1326. 2001.
View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Vokes EE, Weichselbaum RR, Lippman SM and
Hong WK: Head and neck cancer. N Engl J Med. 328:184–194. 1993.
View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Zhou W, Feng X, Li H, et al: Inactivation
of LARS2, located at the commonly deleted region 3p21.3, by both
epigenetic and genetic mechanisms in nasopharyngeal carcinoma. Acta
Biochim Biophys Sin. 41:54–62. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Friend SH, Bernards R, Rogelj S, et al: A
human DNA segment with properties of the gene that predisposes to
retinoblastoma and osteosarcoma. Nature. 323:643–646. 1986.
View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Chiba I, Shindoh M, Yasuda M, et al:
Mutations in the p53 gene and human papillomavirus infection as
significant prognostic factors in squamous cell carcinomas of the
oral cavity. Oncogene. 12:1663–1668. 1996.PubMed/NCBI
|
|
8
|
Call KM, Glaser T, Ito CY, et al:
Isolation and characterization of a zinc finger polypeptide gene at
the human chromosome 11 Wilms’ tumor locus. Cell. 60:509–520.
1990.
|
|
9
|
Kamb A, Shattuck-Eidens D, Eeles R, et al:
Analysis of the p16 gene (CDKN2) as a candidate for the chromosome
9p melanoma susceptibility locus. Nat Genet. 8:23–26. 1994.
View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Aoki K and Taketo MM: Adenomatous
polyposis coli (APC): a multi-functional tumor suppressor gene. J
Cell Sci. 120:3327–3335. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Hahn SA, Schutte M, Hoque AT, et al: DPC4,
a candidate tumor suppressor gene at human chromosome 18q21.1.
Science. 271:350–353. 1996. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Ohta M, Inoue H, Cotticelli MG, et al: The
FHIT gene, spanning the chromosome 3p14.2 fragile site and renal
carcinoma-associated t(3;8) breakpoint, is abnormal in digestive
tract cancers. Cell. 84:587–597. 1996. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Li J, Yen C, Liaw D, et al: PTEN, a
putative protein tyrosine phosphatase gene mutated in human brain,
breast, and prostate cancer. Science. 275:1943–1947. 1997.
View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Steck PA, Pershouse MA, Jasser SA, et al:
Identification of a candidate tumour suppressor gene, MMAC1, at
chromosome 10q23.3 that is mutated in multiple advanced cancers.
Nat Genet. 15:356–362. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Imai FL, Uzawa K, Miyakawa A, Shiiba M and
Tanzawa H: A detailed deletion map of chromosome 20 in human oral
squamous cell carcinoma. Int J Mol Med. 7:43–47. 2001.PubMed/NCBI
|
|
16
|
Miyakawa A, Wang XL, Nakanishi H, et al:
Allelic loss on chromosome 22 in oral cancer: Possibility of the
existence of a tumor suppressor gene on 22q13. Int J Oncol.
13:705–709. 1998.PubMed/NCBI
|
|
17
|
Sparano A, Quesnelle KM, Kumar MS, et al:
Genome-wide profiling of oral squamous cell carcinoma by
array-based comparative genomic hybridization. Laryngoscope.
116:735–741. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Yamamoto N, Mizoe J, Numasawa H, Tsujii H,
Shibahara T and Noma H: Allelic loss on chromosomes 2q, 3p and 21q:
possibly a poor prognostic factor in oral squamous cell carcinoma.
Oral Oncol. 39:796–805. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Roz L, Wu CL, Porter S, et al: Allelic
imbalance on chromosome 3p in oral dysplastic lesions: an early
event in oral carcinogenesis. Cancer Res. 56:1228–1231.
1996.PubMed/NCBI
|
|
20
|
Yamamoto N, Kuroiwa T, Katakura A,
Shibahara T and Choudhury C: Loss of heterozygosity (LOH) on
chromosomes 2q, 3p and 21q in Indian oral squamous cell carcinoma.
Bull Tokyo Dent Coll. 48:109–117. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
van den Berg A, Dijkhuizen T, Draaijers
TG, et al: Analysis of multiple renal cell adenomas and carcinomas
suggests allelic loss at 3p21 to be a prerequisite for malignant
development. Genes Chromosomes Cancer. 19:228–232. 1997.PubMed/NCBI
|
|
22
|
Hung J, Kishimoto Y, Sugio K, et al:
Allele-specific chromosome 3p deletions occur at an early stage in
the pathogenesis of lung carcinoma. JAMA. 273:19081995. View Article : Google Scholar
|
|
23
|
Zhu Y, Spitz MR, Zheng YL, Hong WK and Wu
X: BPDE-induced lymphocytic 3p21.3 aberrations may predict head and
neck carcinoma risk. Cancer. 95:563–568. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Senchenko V, Liu J, Braga E, et al:
Deletion mapping using quantitative real-time PCR identifies two
distinct 3p21.3 regions affected in most cervical carcinomas.
Oncogene. 22:2984–2992. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Senchenko VN, Liu J, Loginov W, et al:
Discovery of frequent homozygous deletions in chromosome 3p21.3
LUCA and AP20 regions in renal, lung and breast carcinomas.
Oncogene. 23:5719–5728. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Baylin SB, Esteller M, Rountree MR,
Bachman KE, Schuebel K and Herman JG: Aberrant patterns of DNA
methylation, chromatin formation and gene expression in cancer. Hum
Mol Genet. 10:687–692. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Bird A: DNA methylation patterns and
epigenetic memory. Genes Dev. 16:6–21. 2002. View Article : Google Scholar
|
|
28
|
Bird AP: CpG-rich islands and the function
of DNA methylation. Nature. 321:209–213. 1986. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Merlo A, Herman JG, Mao L, et al: 5′ CpG
island methylation is associated with transcriptional silencing of
the tumour suppressor p16/CDKN2/MTS1 in human cancers. Nat Med.
1:686–692. 1995.
|
|
30
|
Steinmann K, Sandner A, Schagdarsurengin U
and Dammann RH: Frequent promoter hypermethylation of tumor-related
genes in head and neck squamous cell carcinoma. Oncol Rep.
22:1519–1526. 2009.PubMed/NCBI
|
|
31
|
Zhang H, Feng X, Liu W, et al: Underlying
mechanisms for LTF inactivation and its functional analysis in
nasopharyngeal carcinoma cell lines. J Cell Biochem. 112:1832–1843.
2011. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Zhou W, Feng X, Li H, et al: Functional
evidence for a nasopharyngeal carcinoma-related gene BCAT1 located
at 12p12. Oncol Res. 16:405–413. 2007.PubMed/NCBI
|
|
33
|
Peng D, Ren CP, Yi HM, et al: Genetic and
epigenetic alterations of DLC-1, a candidate tumor suppressor gene,
in nasopharyngeal carcinoma. Acta Biochim Biophys Sin. 38:349–355.
2006. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Zhou L, Feng X, Shan W, et al: Epigenetic
and genetic alterations of the EDNRB gene in nasopharyngeal
carcinoma. Oncology. 72:357–363. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Tuhkanen H, Anttila M, Kosma VM, et al:
Genetic alterations in the peritumoral stromal cells of malignant
and borderline epithelial ovarian tumors as indicated by allelic
imbalance on chromosome 3p. Int J Cancer. 109:247–252. 2004.
View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Liao YC and Lo SH: Deleted in liver
cancer-1 (DLC-1): a tumor suppressor not just for liver. Int J
Biochem Cell Biol. 40:843–847. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Kang JU, Koo SH, Kwon KC and Park JW:
Frequent silence of chromosome 9p, homozygous DOCK8, DMRT1 and
DMRT3 deletion at 9p24.3 in squamous cell carcinoma of the lung.
Int J Oncol. 37:327–335. 2010.PubMed/NCBI
|
|
38
|
Ishiguro H, Tsunoda T, Tanaka T, Fujii Y,
Nakamura Y and Furukawa Y: Identification of AXUD1, a novel human
gene induced by AXIN1 and its reduced expression in human
carcinomas of the lung, liver, colon and kidney. Oncogene.
20:5062–5066. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
39
|
Glavic A, Molnar C, Cotoras D and de Celis
JF: Drosophila Axud1 is involved in the control of
proliferation and displays pro-apoptotic activity. Mech Dev.
126:184–197. 2009. View Article : Google Scholar
|
|
40
|
Ngo JT, Bateman JB, Klisak I, Mohandas T,
Van Dop C and Sparkes RS: Regional mapping of a human rod
alpha-transducin (GNAT1) gene to chromosome 3p22. Genomics.
18:724–725. 1993. View Article : Google Scholar : PubMed/NCBI
|
|
41
|
Yi HM, Ren CP, Peng D, Zhou L, Li H and
Yao KT: Expression, loss of heterozygosity, and methylation of
GNAT1 gene in nasopharyngeal carcinoma. Ai Zheng. 26:9–14. 2007.(In
Chinese).
|
|
42
|
Tomizawa Y, Sekido Y, Kondo M, et al:
Inhibition of lung cancer cell growth and induction of apoptosis
after reexpression of 3p21.3 candidate tumor suppressor gene
SEMA3B. Proc Natl Acad Sci USA. 98:13954–13959. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
43
|
Tse C, Xiang RH, Bracht T and Naylor SL:
Human semaphorin 3B (SEMA3B) located at chromosome 3p21.3
suppresses tumor formation in an adenocarcinoma cell line. Cancer
Res. 62:542–546. 2002.PubMed/NCBI
|
|
44
|
Ochi K, Mori T, Toyama Y, Nakamura Y and
Arakawa H: Identification of semaphorin3B as a direct target of
p53. Neoplasia. 4:82–87. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
45
|
Sekido Y, Bader S, Latif F, et al: Human
semaphorins A(V) and IV reside in the 3p21.3 small cell lung cancer
deletion region and demonstrate distinct expression patterns. Proc
Natl Acad Sci USA. 93:4120–4125. 1996. View Article : Google Scholar : PubMed/NCBI
|
|
46
|
Lerman MI and Minna JD: The 630-kb lung
cancer homozygous deletion region on human chromosome 3p21.3:
identification and evaluation of the resident candidate tumor
suppressor genes. The International Lung Cancer Chromosome 3p213
Tumor Suppressor Gene Consortium. Cancer Res. 60:6116–6133.
2000.
|
|
47
|
Li W, Li X, Wang W, et al: NOR1 is an
HSF1- and NRF1-regulated putative tumor suppressor inactivated by
promoter hypermethylation in nasopharyngeal carcinoma.
Carcinogenesis. 32:1305–1314. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
48
|
Mao WM, Li P, Zheng QQ, et al:
Hypermethylation-modulated downregulation of RASSF1A expression is
associated with the progression of esophageal cancer. Arch Med Res.
42:182–188. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
49
|
Liu WB, Ao L, Zhou ZY, et al: CpG island
hypermethylation of multiple tumor suppressor genes associated with
loss of their protein expression during rat lung carcinogenesis
induced by 3-methylcholanthrene and diethylnitrosamine. Biochem
Biophys Res Commun. 402:507–514. 2010. View Article : Google Scholar
|
|
50
|
An JH, Choi A, Shin KJ, Yang WI and Lee
HY: DNA methylation-specific multiplex assays for body fluid
identification. Int J Legal Med. 402:507–514. 2012.
|
|
51
|
Riquelme E, Tang M, Baez S, et al:
Frequent epigenetic inactivation of chromosome 3p candidate tumor
suppressor genes in gallbladder carcinoma. Cancer Lett.
250:100–106. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
52
|
Kuroki T, Trapasso F, Yendamuri S, et al:
Allelic loss on chromosome 3p21.3 and promoter hypermethylation of
semaphorin 3B in non-small cell lung cancer. Cancer Res.
63:3352–3355. 2003.PubMed/NCBI
|
|
53
|
Ito M, Ito G, Kondo M, et al: Frequent
inactivation of RASSF1A, BLU, and SEMA3B on 3p21.3 by promoter
hypermethylation and allele loss in non-small cell lung cancer.
Cancer Lett. 225:131–139. 2005. View Article : Google Scholar : PubMed/NCBI
|
