1
|
Howes MJ and Houghton PJ: Plants used in
Chinese and Indian traditional medicine for improvement of memory
and cognitive function. Pharmacol Biochem Behav. 75:513–527. 2003.
View Article : Google Scholar : PubMed/NCBI
|
2
|
Hausen BM: Centella asiatica
(Indian pennywort), an effective therapeutic but a weak sensitizer.
Contact Dermatitis. 29:175–179. 1993. View Article : Google Scholar
|
3
|
Rush WR, Murray GR and Graham DJ: The
comparative steadystate bioavailability of the active ingredients
of Madecassol. Eur J Drug Metab Pharmacokinet. 18:323–326. 1993.
View Article : Google Scholar : PubMed/NCBI
|
4
|
Xiong Y, Ding H, Xu M and Gao J:
Protective effects of asiatic acid on rotenone- or
H2O2-induced injury in SH-SY5Y cells.
Neurochem Res. 34:746–754. 2009. View Article : Google Scholar : PubMed/NCBI
|
5
|
Ma K, Zhang Y, Zhu D and Lou Y: Protective
effects of asiatic acid against
D-galactosamine/lipopolysaccharide-induced hepatotoxicity in
hepatocytes and kupffer cells co-cultured system via
redox-regulated leukotriene C4 synthase expression pathway. Eur J
Pharmacol. 603:98–107. 2009. View Article : Google Scholar
|
6
|
Lee YS, Jin DQ, Kwon EJ, et al: Asiatic
acid, a triterpene, induces apoptosis through intracellular
Ca2+ release and enhanced expression of p53 in HepG2
human hepatoma cells. Cancer Lett. 186:83–91. 2002. View Article : Google Scholar : PubMed/NCBI
|
7
|
Bunpo P, Kataoka K, Arimochi H, et al:
Inhibitory effects of asiatic acid and CPT-11 on growth of HT-29
cells. J Med Invest. 52:65–73. 2005. View Article : Google Scholar : PubMed/NCBI
|
8
|
Hsu YL, Kuo PL, Lin LT and Lin CC: Asiatic
acid, a triterpene, induces apoptosis and cell cycle arrest through
activation of extracellular signal-regulated kinase and p38
mitogen-activated protein kinase pathways in human breast cancer
cells. J Pharmacol Exp Ther. 313:333–344. 2005. View Article : Google Scholar
|
9
|
Sastry PS and Rao KS: Apoptosis and the
nervous system. J Neurochem. 74:1–20. 2000. View Article : Google Scholar
|
10
|
Nakano R: Apoptosis: gene-directed cell
death. An overview Horm Res. 48:2–4. 1997. View Article : Google Scholar
|
11
|
Moldoveanu T, Follis AV, Kriwacki RW and
Green DR: Many players in BCL-2 family affairs. Trends Biochem Sci.
39:101–111. 2014. View Article : Google Scholar : PubMed/NCBI
|
12
|
Cheng AM, Byrom MW, Shelton J and Ford LP:
Antisense inhibition of human miRNAs and indications for an
involvement of miRNA in cell growth and apoptosis. Nucleic Acids
Res. 33:1290–1297. 2005. View Article : Google Scholar : PubMed/NCBI
|
13
|
He L, He X, Lim LP, et al: A microRNA
component of the p53 tumour suppressor network. Nature.
447:1130–1134. 2007. View Article : Google Scholar : PubMed/NCBI
|
14
|
Bommer GT, Gerin I, Feng Y, et al:
p53-mediated activation of miRNA34 candidate tumor-suppressor
genes. Curr Biol. 17:1298–1307. 2007. View Article : Google Scholar : PubMed/NCBI
|
15
|
Christoffersen NR, Shalgi R, Frankel LB,
et al: p53-independent upregulation of miR-34a during
oncogene-induced senescence represses MYC. Cell Death Differ.
17:236–245. 2010. View Article : Google Scholar : PubMed/NCBI
|
16
|
Liu C, Yu J, Yu S, et al: MicroRNA-21 acts
as an oncomir through multiple targets in human hepatocellular
carcinoma. J Hepatol. 53:98–107. 2010. View Article : Google Scholar : PubMed/NCBI
|
17
|
Ma X, Kumar M, Choudhury SN, et al: Loss
of the miR-21 allele elevates the expression of its target genes
and reduces tumorigenesis. Proc Natl Acad Sci USA. 108:10144–10149.
2011. View Article : Google Scholar : PubMed/NCBI
|
18
|
Papagiannakopoulos T, Shapiro A and Kosik
KS: MicroRNA-21 targets a network of key tumor-suppressive pathways
in glioblastoma cells. Cancer Res. 68:8164–8172. 2008. View Article : Google Scholar : PubMed/NCBI
|
19
|
Yao Q, Cao S, Li C, Mengesha A, Kong B and
Wei M: Micro-RNA-21 regulates TGF-β-induced myofibroblast
differentiation by targeting PDCD4 in tumor-stroma interaction. Int
J Cancer. 128:1783–1792. 2011.PubMed/NCBI
|
20
|
Medina PP, Nolde M and Slack FJ: OncomiR
addiction in an in vivo model of microRNA-21-induced pre-B-cell
lymphoma. Nature. 467:86–90. 2010. View Article : Google Scholar : PubMed/NCBI
|
21
|
An IS, An S, Kang SM, et al: Titrated
extract of Centella asiatica provides a UVB protective
effect by altering microRNA expression profiles in human dermal
fibroblasts. Int J Mol Med. 30:1194–1202. 2012.
|
22
|
An IS, An S, Choe TB, et al: Centella
asiatica protects against UVB-induced HaCaT keratinocyte damage
through microRNA expression changes. Int J Mol Med. 30:1349–1356.
2012.
|
23
|
Bail S, Swerdel M, Liu H, et al:
Differential regulation of microRNA stability. RNA. 16:1032–1039.
2010. View Article : Google Scholar : PubMed/NCBI
|
24
|
Wu J, Ji X, Zhu L, et al: Up-regulation of
microRNA-1290 impairs cytokinesis and affects the reprogramming of
colon cancer cells. Cancer Lett. 329:155–163. 2013. View Article : Google Scholar : PubMed/NCBI
|
25
|
Li A, Yu J, Kim H, et al: MicroRNA array
analysis finds elevated serum miR-1290 accurately distinguishes
patients with low-stage pancreatic cancer from healthy and disease
controls. Clin Cancer Res. 19:3600–3610. 2013. View Article : Google Scholar
|
26
|
White NM, Bao TT, Grigull J, et al: miRNA
profiling for clear cell renal cell carcinoma: biomarker discovery
and identification of potential controls and consequences of miRNA
dysregulation. J Urol. 186:1077–1083. 2011. View Article : Google Scholar : PubMed/NCBI
|
27
|
Yao T, Rao Q, Liu L, et al: Exploration of
tumor-suppressive microRNAs silenced by DNA hypermethylation in
cervical cancer. Virol J. 10:1752013. View Article : Google Scholar : PubMed/NCBI
|
28
|
An IS, An S, Kwon KJ, Kim YJ and Bae S:
Ginsenoside Rh2 mediates changes in the microRNA expression profile
of human non-small cell lung cancer A549 cells. Oncol Rep.
29:523–528. 2013.PubMed/NCBI
|
29
|
Endo Y, Toyama T, Takahashi S, et al:
miR-1290 and its potential targets are associated with
characteristics of estrogen receptor α-positive breast cancer.
Endocr Relat Cancer. 20:91–102. 2013.
|
30
|
Yelamanchili SV, Morsey B, Harrison EB, et
al: The evolutionary young miR-1290 favors mitotic exit and
differentiation of human neural progenitors through altering the
cell cycle proteins. Cell Death Dis. 5:e9822014. View Article : Google Scholar : PubMed/NCBI
|
31
|
Kluck RM, Bossy-Wetzel E, Green DR and
Newmeyer DD: The release of cytochrome c from mitochondria:
a primary site for Bcl-2 regulation of apoptosis. Science.
275:1132–1136. 1997.PubMed/NCBI
|
32
|
Yang J, Liu X, Bhalla K, et al: Prevention
of apoptosis by Bcl-2: release of cytochrome c from mitochondria
blocked. Science. 275:1129–1132. 1997. View Article : Google Scholar : PubMed/NCBI
|
33
|
Gazitt Y, Rothenberg ML, Hilsenbeck SG,
Fey V, Thomas C and Montegomrey W: Bcl-2 overexpression is
associated with resistance to paclitaxel, but not gemcitabine, in
multiple myeloma cells. Int J Oncol. 13:839–848. 1998.PubMed/NCBI
|
34
|
Cho HJ, Kim JK, Kim KD, et al:
Upregulation of Bcl-2 is associated with cisplatin-resistance via
inhibition of Bax translocation in human bladder cancer cells.
Cancer Lett. 237:56–66. 2006. View Article : Google Scholar : PubMed/NCBI
|
35
|
Cory S and Adams JM: The Bcl2 family:
regulators of the cellular life-or-death switch. Nat Rev Cancer.
2:647–656. 2002. View
Article : Google Scholar : PubMed/NCBI
|
36
|
Choi YM, An S, Lee EM, et al: CYP1A1 is a
target of miR-892a-mediated post-transcriptional repression. Int J
Oncol. 41:331–336. 2012.PubMed/NCBI
|