|
1
|
Stewart BW and Wild CP: World Cancer
Report 2014. IARC Publications; Lyon: 2014
|
|
2
|
Wong A and Ma BBY: Personalizing therapy
for colorectal cancer. Clin Gastroenterol Hepatol. 12:139–144.
2014. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Van Cutsem E, Cervantes A, Nordlinger B
and Arnold D: ESMO Guidelines Working Group: Metastatic colorectal
cancer: ESMO Clinical Practice Guidelines for diagnosis, treatment
and follow-up. Ann Oncol. 25:(Suppl 3). iii1–iii9. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Brand TM, Iida M and Wheeler DL: Molecular
mechanisms of resistance to the EGFR monoclonal antibody cetuximab.
Cancer Biol Ther. 11:777–792. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Prior IA, Lewis PD and Mattos C: A
comprehensive survey of Ras mutations in cancer. Cancer Res.
72:2457–2467. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Schubbert S, Shannon K and Bollag G:
Hyperactive Ras in developmental disorders and cancer. Nat Rev
Cancer. 7:295–308. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Dócs O, Fazakas F, Horváth NL, Tóth L,
András C, Horváth Z and Méhes G: Changes of KRAS exon 2 codon 12/13
mutation status in recurrent colorectal cancer. Pathol Oncol Res.
21:399–404. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Roda D, Castillo J, Telechea-Fernández M,
Gil A, López-Rodas G, Franco L, González-Rodríguez P, Roselló S,
Pérez-Fidalgo JA, García-Trevijano ER, et al: EGF-induced
acetylation of heterogeneous nuclear ribonucleoproteins is
dependent on KRAS mutational status in colorectal cancer cells.
PLoS One. 10:e01305432015. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Soh J, Okumura N, Lockwood WW, Yamamoto H,
Shigematsu H, Zhang W, Chari R, Shames DS, Tang X, MacAulay C, et
al: Oncogene mutations, copy number gains and mutant allele
specific imbalance (MASI) frequently occur together in tumor cells.
PLoS One. 4:e74642009. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Hartman DJ, Davison JM, Foxwell TJ,
Nikiforova MN and Chiosea SI: Mutant allele-specific imbalance
modulates prognostic impact of KRAS mutations in colorectal
adenocarcinoma and is associated with worse overall survival. Int J
Cancer. 131:1810–1817. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Malapelle U, Sgariglia R, De Stefano A,
Bellevicine C, Vigliar E, de Biase D, Sepe R, Pallante P,
Carlomagno C, Tallini G, et al: KRAS mutant allele-specific
imbalance (MASI) assessment in routine samples of patients with
metastatic colorectal cancer. J Clin Pathol. 68:265–269. 2015.
View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Costa V, Aprile M, Esposito R and
Ciccodicola A: RNA-Seq and human complex diseases: Recent
accomplishments and future perspectives. Eur J Hum Genet.
21:134–142. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Du J and Zhang L: Integrated analysis of
DNA methylation and microRNA regulation of the lung adenocarcinoma
transcriptome. Oncol Rep. 34:585–594. 2015.PubMed/NCBI
|
|
14
|
Panagopoulos I, Gorunova L, Zeller B,
Tierens A and Heim S: Cryptic FUS-ERG fusion identified by
RNA-sequencing in childhood acute myeloid leukemia. Oncol Rep.
30:2587–2592. 2013.PubMed/NCBI
|
|
15
|
Wang W, Qin Z, Feng Z, Wang X and Zhang X:
Identifying differentially spliced genes from two groups of RNA-seq
samples. Gene. 518:164–170. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Wang ET, Sandberg R, Luo S, Khrebtukova I,
Zhang L, Mayr C, Kingsmore SF, Schroth GP and Burge CB: Alternative
isoform regulation in human tissue transcriptomes. Nature.
456:470–476. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Matera AG and Wang Z: A day in the life of
the spliceosome. Nat Rev Mol Cell Biol. 15:108–121. 2014.
View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Ladomery M: Aberrant alternative splicing
is another hallmark of cancer. Int J Cell Biol. 2013:4637862013.
View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Li H, Handsaker B, Wysoker A, Fennell T,
Ruan J, Homer N, Marth G, Abecasis G and Durbin R: 1000 Genome
Project Data Processing Subgroup: The Sequence Alignment/Map format
and SAMtools. Bioinformatics. 25:2078–2079. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Trapnell C, Williams BA, Pertea G,
Mortazavi A, Kwan G, van Baren MJ, Salzberg SL, Wold BJ and Pachter
L: Transcript assembly and quantification by RNA-Seq reveals
unannotated transcripts and isoform switching during cell
differentiation. Nat Biotechnol. 28:511–515. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Vitting-Seerup K, Porse BT, Sandelin A and
Waage J: spliceR: An R package for classification of alternative
splicing and prediction of coding potential from RNA-seq data. BMC
Bioinformatics. 15:812014. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Simms D, Cizdziel P and Chomczynski P:
TRIzol: A new reagent for optimal single-step isolation of RNA.
Focus. 15:99–102. 1993.
|
|
23
|
Livak KJ and Schmittgen TD: Analysis of
relative gene expression data using real-time quantitative PCR and
the 2ΔΔCT method. Methods. 25:402–408. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Slattery ML, Pellatt DF, Mullany LE, Wolff
RK and Herrick JS: Gene expression in colon cancer: A focus on
tumor site and molecular phenotype. Genes Chromosomes Cancer.
54:527–541. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Shashoua VE: The role of brain
extracellular proteins in neuroplasticity and learning. Cell Mol
Neurobiol. 5:183–207. 1985. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Nimmrich I, Erdmann S, Melchers U,
Chtarbova S, Finke U, Hentsch S, Hoffmann I, Oertel M, Hoffmann W
and Müller O: The novel ependymin related gene UCC1 is highly
expressed in colorectal tumor cells. Cancer Lett. 165:71–79. 2001.
View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Pradel G, Schachner M and Schmidt R:
Inhibition of memory consolidation by antibodies against cell
adhesion molecules after active avoidance conditioning in
zebrafish. J Neurobiol. 39:197–206. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Hoffmann W and Schwarz H: Ependymins:
Meningeal-derived extracellular matrix proteins at the blood-brain
barrier. Int Rev Cytol. 165:121–158. 1996. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Shashoua VE, Adams D and Boyer-Boiteau A:
CMX-8933, a peptide fragment of the glycoprotein ependymin,
promotes activation of AP-1 transcription factor in mouse
neuroblastoma and rat cortical cell cultures. Neurosci Lett.
312:103–107. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Jin TB, Ren Y, Shi X, Jiri M, He N, Feng
T, Yuan D and Kang L: Genetic variations in the CLNK gene and
ZNF518B gene are associated with gout in case-control sample sets.
Rheumatol Int. 35:1141–1147. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Zhang XY, Geng TT, Liu LJ, Yuan DY, Feng
T, Kang LL, Jin TB and Chen C: SLC2A9 and ZNF518B polymorphisms
correlate with gout-related metabolic indices in Chinese Tibetan
populations. Genet Mol Res. 14:9915–9921. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Maier VK, Feeney CM, Taylor JE, Creech AL,
Qiao JW, Szanto A, Das PP, Chevrier N, Cifuentes-Rojas C, Orkin SH,
et al: Functional proteomic analysis of repressive histone
methyltransferase complexes PRC2 and G9A reveals ZNF518B as a G9A
regulator. Mol Cell Proteomics. 14:1435–1446. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Shankar SR, Bahirvani AG, Rao VK, Bharathy
N, Ow JR and Taneja R: G9a, a multipotent regulator of gene
expression. Epigenetics. 8:16–22. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Zhao Z, Wang D, Zhu C, Shao H, Sun C, Qiu
H, Xue L, Xu J, Guo M and Li W: Aberrant alternative splicing of
human zinc finger gene ZNF268 in human hematological malignancy.
Oncol Rep. 20:1243–1248. 2008.PubMed/NCBI
|
