1
|
ACS: Key statistics for melanoma skin
cancer. http://www.cancer.org/cancer/skincancer-melanoma/detailedguide/melanoma-skin-cancer-key-statisticsLast
revised: 05/20/2016. 12 /12–/16
|
2
|
Yurchenco PD and Schittny JC: Molecular
architecture of basement membranes. FASEB J. 4:1577–1590.
1990.PubMed/NCBI
|
3
|
Barsky SH, Siegal GP, Jannotta F and
Liotta LA: Loss of basement membrane components by invasive tumors
but not by their benign counterparts. Lab Invest. 49:140–147.
1983.PubMed/NCBI
|
4
|
Liotta LA, Tryggvason K, Garbisa S, Hart
I, Foltz CM and Shafie S: Metastatic potential correlates with
enzymatic degradation of basement membrane collagen. Nature.
284:67–68. 1980. View
Article : Google Scholar : PubMed/NCBI
|
5
|
Nikkola J, Vihinen P, Vuoristo MS,
Kellokumpu-Lehtinen P, Kähäri VM and Pyrhönen S: High serum levels
of matrix metalloproteinase-9 and matrix metalloproteinase-1 are
associated with rapid progression in patients with metastatic
melanoma. Clin Cancer Res. 11:5158–5166. 2005. View Article : Google Scholar : PubMed/NCBI
|
6
|
Malaponte G, Zacchia A, Bevelacqua Y,
Marconi A, Perrotta R, Mazzarino MC, Cardile V and Stivala F:
Co-regulated expression of matrix metalloproteinase-2 and
transforming growth factor-β in melanoma development and
progression. Oncol Rep. 24:81–87. 2010. View Article : Google Scholar : PubMed/NCBI
|
7
|
de Visser KE and Coussens LM: The
inflammatory tumor microenvironment and its impact on cancer
development. Contrib Microbiol. 13:118–137. 2006. View Article : Google Scholar : PubMed/NCBI
|
8
|
Coussens LM and Werb Z: Inflammation and
cancer. Nature. 420:860–867. 2002. View Article : Google Scholar : PubMed/NCBI
|
9
|
Mantovani A, Allavena P, Sica A and
Balkwill F: Cancer-related inflammation. Nature. 454:436–444. 2008.
View Article : Google Scholar : PubMed/NCBI
|
10
|
Qin Y, Ekmekcioglu S, Liu P, Duncan LM,
Lizée G, Poindexter N and Grimm EA: Constitutive aberrant
endogenous interleukin-1 facilitates inflammation and growth in
human melanoma. Mol Cancer Res. 9:1537–1550. 2011. View Article : Google Scholar : PubMed/NCBI
|
11
|
Niedzwiecki A, Roomi MW, Kalinovsky T and
Rath M: Micronutrient synergy: a new tool in effective control of
metastasis and other key mechanisms of cancer. Cancer Metastasis
Rev. 29:529–542. 2010. View Article : Google Scholar : PubMed/NCBI
|
12
|
Rath M and Pauling L: Plasmin-induced
proteolysis and the role of apoprotein(a), lysine and synthetic
analogs. Orthomolecular Med. 7:17–23. 1992.
|
13
|
Sun Z, Chen YH, Wang P, Zhang J, Gurewich
V, Zhang P and Liu JN: The blockage of the high-affinity lysine
binding sites of plasminogen by EACA significantly inhibits
prourokinase-induced plasminogen activation. Biochim Biophys Acta.
1596:182–192. 2002. View Article : Google Scholar : PubMed/NCBI
|
14
|
Mussini E, Hutton JJ Jr and Udenfriend S:
Collagen proline hydroxylase in wound healing, granuloma formation,
scurvy, and growth. Science. 157:927–929. 1967. View Article : Google Scholar : PubMed/NCBI
|
15
|
Kivirikko KI and Myllylä R: Collagen
glycosyltransferases. Int Rev Connect Tissue Res. 8:23–72. 1979.
View Article : Google Scholar : PubMed/NCBI
|
16
|
Valcic S, Timmermann BN, Alberts DS,
Wächter GA, Krutzsch M, Wymer J and Guillén JM: Inhibitory effect
of six green tea catechins and caffeine on the growth of four
selected human tumor cell lines. Anticancer Drugs. 7:461–468. 1996.
View Article : Google Scholar : PubMed/NCBI
|
17
|
Mukhtar H and Ahmad N: Tea polyphenols:
Prevention of cancer and optimizing health. Am J Clin Nutr. 71
Suppl:1698S–1702S, discussion 1703S-1704S. 2000.PubMed/NCBI
|
18
|
Yang GY, Liao J, Kim K, Yurkow EJ and Yang
CS: Inhibition of growth and induction of apoptosis in human cancer
cell lines by tea polyphenols. Carcinogenesis. 19:611–616. 1998.
View Article : Google Scholar : PubMed/NCBI
|
19
|
Taniguchi S, Fujiki H, Kobayashi H, Go H,
Miyado K, Sadano H and Shimokawa R: Effect of (−)-epigallocatechin
gallate, the main constituent of green tea, on lung metastasis with
mouse B16 melanoma cell lines. Cancer Lett. 65:51–54. 1992.
View Article : Google Scholar : PubMed/NCBI
|
20
|
Hara Y: Green tea: Health Benefits and
Applications. Marcel Dekker; New York, Basel: 2001, https://doi.org/10.1201/9780203907993 View Article : Google Scholar
|
21
|
Gupta S, Hastak K, Afaq F, Ahmad N and
Mukhtar H: Essential role of caspases in
epigallocatechin-3-gallate-mediated inhibition of nuclear factor
kappa B and induction of apoptosis. Oncogene. 23:2507–2522. 2004.
View Article : Google Scholar : PubMed/NCBI
|
22
|
Fujiki H, Suganuma M, Okabe S, Sueoka E,
Suga K, Imai K, Nakachi K and Kimura S: Mechanistic findings of
green tea as cancer preventive for humans. Proc Soc Exp Biol Med.
220:pp. 225–228. 1999; View Article : Google Scholar : PubMed/NCBI
|
23
|
Kawakami S, Kageyama Y, Fujii Y, Kihara K
and Oshima H: Inhibitory effect of N-acetylcysteine on invasion and
MMP-9 production of T24 human bladder cancer cells. Anticancer Res.
21:213–219. 2001.PubMed/NCBI
|
24
|
Morini M, Cai T, Aluigi MG, Noonan DM,
Masiello L, De Flora S, DAgostini F, Albini A and Fassina G: The
role of the thiol N-acetylcysteine in the prevention of tumor
invasion and angiogenesis. Int J Biol Markers. 14:268–271.
1999.PubMed/NCBI
|
25
|
Yoon SO, Kim MM and Chung AS: Inhibitory
effect of selenite on invasion of HT1080 tumor cells. J Biol Chem.
276:20085–20092. 2001. View Article : Google Scholar : PubMed/NCBI
|
26
|
Cha J, Roomi MW, Ivanov V, Kalinovsky T,
Niedzwiecki A and Rath M: Ascorbate supplementation inhibits growth
and metastasis of B16FO melanoma and 4T1 breast cancer cells in
vitamin C-deficient mice. Int J Oncol. 42:55–64. 2013.PubMed/NCBI
|
27
|
Naidu KA, Karl RC, Naidu KA and Coppola D:
Antiproliferative and proapoptotic effect of ascorbyl stearate in
human pancreatic cancer cells: Association with decreased
expression of insulin-like growth factor 1 receptor. Dig Dis Sci.
48:230–237. 2003. View Article : Google Scholar : PubMed/NCBI
|
28
|
Anthony HM and Schorah CJ: Severe
hypovitaminosis C in lung-cancer patients: The utilization of
vitamin C in surgical repair and lymphocyte-related host
resistance. Br J Cancer. 46:354–367. 1982. View Article : Google Scholar : PubMed/NCBI
|
29
|
Maramag C, Menon M, Balaji KC, Reddy PG
and Laxmanan S: Effect of vitamin C on prostate cancer cells in
vitro: Effect on cell number, viability, and DNA synthesis.
Prostate. 32:188–195. 1997. View Article : Google Scholar : PubMed/NCBI
|
30
|
Koh WS, Lee SJ, Lee H, Park C, Park MH,
Kim WS, Yoon SS, Park K, Hong SI, Chung MH, et al: Differential
effects and transport kinetics of ascorbate derivatives in leukemic
cell lines. Anticancer Res. 18:2487–2493. 1998.PubMed/NCBI
|
31
|
Chen Q, Espey MG, Krishna MC, Mitchell JB,
Corpe CP, Buettner GR, Shacter E and Levine M: Pharmacologic
ascorbic acid concentrations selectively kill cancer cells: Action
as a pro-drug to deliver hydrogen peroxide to tissues. Proc Natl
Acad Sci USA. 102:pp. 13604–13609. 2005; View Article : Google Scholar : PubMed/NCBI
|
32
|
Núñez Martín C, de Apodaca y Ruiz A Ortiz
and Ruiz A: Ascorbic acid in the plasma and blood cells of women
with breast cancer. The effect of the consumption of food with an
elevated content of this vitamin. Nutr Hosp. 10:368–372. 1995.(In
Spanish).
|
33
|
Kurbacher CM, Wagner U, Kolster B,
Andreotti PE, Krebs D and Bruckner HW: Ascorbic acid (vitamin C)
improves the antineoplastic activity of doxorubicin, cisplatin, and
paclitaxel in human breast carcinoma cells in vitro. Cancer Lett.
103:183–189. 1996. View Article : Google Scholar : PubMed/NCBI
|
34
|
Cooke JP and Dzau VJ: Nitric oxide
synthase: Role in the genesis of vascular disease. Annu Rev Med.
48:489–509. 1997. View Article : Google Scholar : PubMed/NCBI
|